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Case: Asbestosis

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So here's a nice case where the pulmonary fibrosis is

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really not that floored.

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So clearly peripheral and basal predominant.

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There wasn't much in the upper lobes,

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especially in the A lung AEs.

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But here we start to see some mild reticulation

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and some mild traction bronchiectasis as well.

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Maybe a little hard to see.

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They pointed out there's, there's always,

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sometimes there's motion

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here, but you just gotta read through it.

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But these, so these small cysts here, like for example,

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right in there, really in the outer, say two centimeters of,

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of lung, you really shouldn't see any airways.

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So this is a small little cystic thing

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superimposed reticulation.

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That's an area of traction bronchiectasis.

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And so that tells us that clearly there is some mild

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fibrosis in this patient with this reticular abnormality.

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But why am I showing this, this very boring case?

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Um, I'm showing to you

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because as a radiologist you gotta look at everything.

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And so one of the things

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that we gonna look at are gonna be the soft tissue windows.

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And so if we look carefully, we're gonna see a lot

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of these plural plaques.

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And so if I was just looking at the CT in terms

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of lung parenchyma, I might've called this an

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indeterminant for UIP pattern.

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It's just, it's just not that severe enough for me

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to be confident what's going on.

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But now that we see clearly these asbestos related pleural

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plaques bilaterally, which, so just to remember, just

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as a reminder, uh, these

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almost always will spare the lung AEs.

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How there, see how there are no plaques up here.

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And if we go down the Coran angle,

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they all always will spare the claran angle as well

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to go down the Coran engles here.

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There's none down here, no plaques.

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There are no plaques in the Coran angles.

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I don't know why that happens. But yeah,

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the pleural plaques, they don't involve those areas.

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So if you see pleural abnormality,

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even if it's calcified extending into the cos, Frank Angles

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or lung AEs, you start

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to think about other alternative diagnosis too.

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Asbestos related pleural disease.

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But in this case we have classic asbestos related pleural

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disease, these beautiful calcified plaques bilaterally

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symmetric with mild fibrosis, this case of asbestosis.

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The tiny wrinkle here is

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that this patient also has some mild

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dendri formm pulmonary ossification.

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I don't know if you guys noticed that

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initially, but it's bilateral.

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So these areas, so these are not plaques, right?

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These are actually within the lung parenchyma.

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So how that hyperdense

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and they are branching in terms of the morphology,

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if we put it into our lung window here.

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Harder to see the hyper density,

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but I think it's easier to see the branching morphology

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of these areas of dendri formm pulmonary ossification.

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And so what is dendri formm pulmonary ossification?

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So it's probably, so no one really knows for sure,

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but it's probably a response to chronic lung inflammation.

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We see it very commonly in pulmonary fibrosis.

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So we see it in UIP idiopathic pulmonary fibrosis.

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We can even see in a connective tissue disease.

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It's I think most strongly described the setting

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of systemic sclerosis.

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We tend not to see in a fibrotic hp,

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there's some sparse data on that, but classic

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Fibrotic HP cases, I don't know why,

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but we don't see it as much.

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And then the other thing that sometimes we'll see is even

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outside of patients with pulmonary fibrosis,

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just sometimes you'll see isolated

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and driven form pulmonary ossification in patients

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who have chronic recurrent aspiration.

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And there's some modest data which supports that

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as a theory.

Report

Faculty

Jonathan H. Chung, MD

Professor of Radiology and Division Chief of Cardiothoracic Imaging

UCSD - University of California San Diego

Tags

Non-infectious Inflammatory

Lungs

Chest CT

Chest

CT