Interactive Transcript
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So this was a,
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a patient that had an encephalocele involving the right anterior skull base.
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And what we see here, uh, especially seen on the T twos,
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is that we see a defect here involving the anterior cranial fossa and there's
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herniation of, uh, intracranial elements,
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which includes dysplastic brain and also C S F extending into the right nasal
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cavity.
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So this was an example of a frontal nasal or frontal eth mortal ence foal.
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I think, I think either one of these terms are fine. Um,
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if you think it's extending into the ethmoid,
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then it's a frontal eth mortal ence foal.
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If it's extending into the nasal cavity, it's frontal nasal ence,
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probably frontal, um, ethmoidal phae, uh,
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encephalocele is probably the most accurate on this because it looks like it's
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just lateral to the olfactory sulcus. Uh,
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the majority of it is lateral and it's involving the right, um,
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ethmoid air cells. Any questions on this one?
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For this kind of ence, would that cost, uh,
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in intracranial hypotension?
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Uh, that's a really good question. Um, it's possible, yes. Yeah.
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The reason why I hesitate Han is that, um, yes, if, if this is,
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uh, constant leakage of C S F, then yes,
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it could result in intracranial hypotension.
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But the reason why I hesitated is that you can have ence foals involving other
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parts of the brain that are actually due to intracranial hypertension.
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So for instance, you can have meles cave encephalocele,
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you can have ence foals involving the teman
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and the teman mast sodium that are due to intracranial hypertension.
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So in this particular case, if you do see this, then yes,
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it can result in intracranial H Y P O hypotension,
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but just realize some encephalocele can actually be due to intercranial
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hypertension because, um, it's essentially a,
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a release valve if the intercranial pressure goes up too high.
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Okay. They answered my question. Challenging. Yeah. Okay.
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Have to see a lot other signs for hyper or hyper.
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Well, say that again so I understand what you're saying.
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Oh, so me,
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meaning we have to see for other imaging features of hyper
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or hypo, like yes, any sinus stenosis or validation,
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and also maybe need to correlate with lumper puncture, opening pressure.
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Right. And also the pituitary gland. I have to admit,
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like the first thing that I look at when I'm looking at intracranial
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hypertension p e r, is I look at the pituitary gland.
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If the pituitary gland is flattened,
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then I start thinking of idiopathic intracranial hypertension.
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My biggest challenge is that I see,
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I see so many I a c studies, um,
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I see a large variability of variability enlargement of the transverse and the
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sigmoid sinuses. So, um,
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sometimes I don't think that's a super reliable sign. When hypo,
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when idiopathic intracranial hypotension was first,
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um, described,
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you had to have about four or five findings associated with hypotension,
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um, and hypertension. H Y P E R, like I said,
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I kind of look primarily at the flattening of the pituitary gland and dilatation
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and redundancy of the optic nerve sheets.
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Then I'll start looking for dilatation of meles caves and then maybe, yeah,
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narrowing the transverse sinus. Although I have to admit,
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I don't think that's as,
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as a reliable as sign as some of the other signs that I, that I, that I use.
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Okay. So the pituitary green is the first point We look
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Pituitary. Yeah. That's,
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I always start looking off of those midline structures because of the pituitary
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is enlarged in a older patient, if it's really sort of enlarged and,
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and they're sagging of the tonsils,
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and I start thinking about intracranial hypotension.
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But if a pituitary's flattened, especially in an overweight patient,
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and then I start seeing the dilatation of the optic nerve sheet,
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then I start thinking about hypertension.