Interactive Transcript
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So as far as complications,
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we've talked about some of these already.
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We talked about graft impingement,
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and we'll show you more examples of that.
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Certainly we wanna look for graft disruption, uh,
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in patients who have re injuries
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of their knee following reconstruction, our th fibrosis,
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whether it's global or, uh, focal.
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Um, any hardware failures, hardware migration,
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and I'm not gonna talk about double bundle reconstructions,
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um, as that's kind of a whole another topic in of itself.
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But, uh, be cognizant that, uh, you may encounter double,
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uh, bundle reconstructions
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and the complications overall are similar.
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So with regards to acute graft tears similar
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to the native ACL,
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we can subdivide things into primary MRI signs
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that means problems intrinsic
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to the anterior cruciate ligament or secondary signs.
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Now you have to be careful with the secondary
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signs these have to redo.
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These have to do with malalignment of the knee.
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So anterior tibial translation,
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while they're good in the native knee
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and the native injured ACL, they're not
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as good in the reconstructed knee.
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And I'll show you why. But as far as primary signs go,
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similar to the native ACL, we wanna look for signal
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or morphologic changes, changes in orientation, areas
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of fiber discontinuity, and changes in graft thickness.
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Unfortunately, a lot
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of these findings can be seen in both full
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and partial thickness tears.
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And depending on the age of the graft, as I alluded earlier,
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if you image earlier, you might see some areas
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of increased signal with intact grafts.
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So here's an example of a patient with an acute graft tear.
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We can see focal high signal on our fluid sensitive sequence
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through the mid portion of the graft,
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and this patient had clinical laxity on exam.
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Here's another, uh, example of a complete graft tear,
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which is more chronic.
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Now in this stage, and we can say it's chronic
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because we no longer see any visible graft fibers,
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so similar to the native anterior cru ligament,
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it can either undergo scarring
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or completely resorb over time.
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So this is a complete tear with graft resorption.
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Here's why. Secondary signs not as helpful in the
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reconstructed ligament.
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So this is a paper, um, roughly 10 years ago, uh,
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looking at the secondary signs.
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And they found that if you had slid anterior translation,
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the tibia relative the femur, this had a kind of a low
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positive predictive value for predicting patients
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to have laxity on clinical exam.
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So if you see this finding, it won't,
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it doesn't really have a lot of meaning.
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And why is that? It's probably has to do
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with anterior cruciate ligament reconstructions.
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While we strive to attain perfect anatomic
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reconstructions, sometimes these, uh,
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ligaments are not quite placed in a perfect anatomic
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reconstruction, particularly for single, um,
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bundle reconstructions as opposed to, uh,
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double bundle reconstructions where they're trying
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to create both the antal
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and posterolateral bundles in a more anatomic fashion.
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Conversely, if you see normal alignment,
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so say this tibia was lined up perfectly
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with the posterior aspect of the lateral femoral condyle,
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then this had a high native predictive value, meaning
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that you were unlikely
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to have any translational laxity on exam.
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We've talked about roof impingement,
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particularly in the setting of tibial tunnels
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that are too far anterior.
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And sometimes you'll read in the operative notes
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that the arthroscopy will pull, will perform a notch plasty,
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meaning they'll debride a little bit of bone to make room
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for the graft, but obviously they won't want to over debride
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and create a, uh, um, a, a situation
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where you have knee laxity.
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So again, just look for any kinds of posterior bowing.
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If you see attritional changes
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or thinning of the reconstructed ligament,
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then you are likely dealing with roof impingement.
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Also, these patients may present with, um, loss
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of terminal knee extension.
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Here's just another example of a patient
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with roof impingement.
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Their tibial tunnels a little bit anterior location,
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but even even more importantly, you can notice
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that their femoral tunnels way too anterior was probably
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placed in front of that lateral intercondylar ridge
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or residence ridge.
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So similar to cystic degeneration
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of the native anterior cruciate ligament.
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This can also happen in the reconstructed ligament.
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This can in, uh, affect the intra, um, the joint portion
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of the reconstructed ligament,
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but can also affect the bone tunnels as well.
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So here you can see almost that celery stock appearance of
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that, uh, reconstructive ligament heading into bone
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and resulting in tunnel widening.
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And in of itself, if the patient has a clinical stable exam,
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probably not that important,
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but further down the road, should they tear their ligament
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and require an additional surgery
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or reconstruction, this can result in, um, problems.
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So tunnel cysts, when the largest more than 14 millimeters
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and the patients require a redo surgery, this is, uh,
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where the patients may require a two stage revision.
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So here's an example of a patient
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with enlarging tunnel cyst.
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This is, uh, by eyeball more than 14 millimeters.
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And so here's another example of a patient
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with a large tunnel cyst who had
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to undergo grafting of the tunnel.
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Wait several weeks for the bone graft to mature
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before undergoing a second ACL reconstruction.
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Here's an example of other tunnel complications.
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This was a, uh, former, uh, professional
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Football player of the Chargers organization underwent a
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bone patella tendon bone autograph.
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You can see the changes of the inferior pole, the patella,
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and you can see that the interference screw should be in
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line with the femoral tunnel
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and the bone plug that's also supposed to be in line
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with this, uh, interference screw has dropped into the joint
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and, uh, was actually astutely picked up on the MRI
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and then subsequently confirmed on CT.
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STR fractures are relatively uncommon.
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They tend to occur more on the femoral side.
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Uh, they tend to occur more, um, with larger tunnels
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and that tends to make sense.
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The lower bone you take away, the more likely you're going
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to result in a stress riser.
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So here's an example of a pediatric patient
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who underwent an ACL reconstruction
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with a distal femur stress fracture.
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Whether this is truly a causal relationship to the, um,
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anterior cruciate ligament reconstruction,
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I can't really prove or whether this is just a simple matter
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of fact stress fracturing a patient who, uh,
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with overactivity, certainly we wanna look for,
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uh, hardware complications including hardware migration.
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Here's an example, uh,
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from the literature showing a uh, button.
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Now remember, these are cortical buttons that they're placed
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through the femoral tunnel and flipped,
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and that's how they engage on the, uh,
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of the supracondylar ridge, of the distal femur.
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Much like when you button a shirt
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that keeps it secure in place.
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Here's, uh, one in the literature
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that was placed in the soft tissues,
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and these can be, uh, pretty dramatic
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because as you can imagine, the tension
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of the graph can result in, um, tension on the button and
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therefore tissue necrosis.
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One thing I didn't talk about is a different type
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of graph fixation that you may encounter are cross pins.
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These are another form of dispensary fixations,
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and these are placed along the, um, distal femur.
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And ideally these cross pins should be
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placed entirely within bone.
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But on occasion, these are an inadvertently placed within
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the soft tissue soft tissues outside the femur.
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And here's a couple of cases of broken cross pins
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that were placed outside the femur.
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Here's a case, uh, from our, um, case files
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where a cross pin was placed outside the femur
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and have actually fractured
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and migrated into the gastroc anus muscles.
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You can see here actually dissecting through, uh,
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the intramuscular vein of the gastroc neis muscle back here.
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Certainly, uh, when we have fixation screws,
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these interference screws can migrate as well.
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This one, uh, I can't tell, we don't have pre, um,
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prior imaging demonstrating whether this has extruded
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superiorly or this is how it was natively placed.
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But you can see that there's local localized arthrofibrosis
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raising the possibility of a cyclops lesion
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as we look in the second case.
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Here's another example of screw protrusion at
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the inferior aspect of the tunnel.
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And this has to be correlated with clinical findings.
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Whether this is a symptomatic finding
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or not, here's a more dramatic case of a screw
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that extruded into the subcutaneous adipose tissues.
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So this patient actually had it
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to undergo screw removal given that it was, uh, completely
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underneath the skin
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and mobile I alluded to.
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Um, when placing the femoral tunnel too far posterior,
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you can, uh, encounter this, uh, entity
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of posterior wall blowout,
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so the posterior cortical margin is violated.
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And you can see that the femoral button now is floating in
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space, uh, detached, uh, from the graft.
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And here it is on ct, not engage
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with the lateral cortical margin of the distal femur.
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I.