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ACL Reconstruction Complications

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So as far as complications,

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we've talked about some of these already.

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We talked about graft impingement,

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and we'll show you more examples of that.

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Certainly we wanna look for graft disruption, uh,

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in patients who have re injuries

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of their knee following reconstruction, our th fibrosis,

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whether it's global or, uh, focal.

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Um, any hardware failures, hardware migration,

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and I'm not gonna talk about double bundle reconstructions,

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um, as that's kind of a whole another topic in of itself.

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But, uh, be cognizant that, uh, you may encounter double,

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uh, bundle reconstructions

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and the complications overall are similar.

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So with regards to acute graft tears similar

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to the native ACL,

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we can subdivide things into primary MRI signs

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that means problems intrinsic

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to the anterior cruciate ligament or secondary signs.

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Now you have to be careful with the secondary

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signs these have to redo.

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These have to do with malalignment of the knee.

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So anterior tibial translation,

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while they're good in the native knee

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and the native injured ACL, they're not

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as good in the reconstructed knee.

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And I'll show you why. But as far as primary signs go,

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similar to the native ACL, we wanna look for signal

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or morphologic changes, changes in orientation, areas

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of fiber discontinuity, and changes in graft thickness.

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Unfortunately, a lot

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of these findings can be seen in both full

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and partial thickness tears.

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And depending on the age of the graft, as I alluded earlier,

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if you image earlier, you might see some areas

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of increased signal with intact grafts.

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So here's an example of a patient with an acute graft tear.

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We can see focal high signal on our fluid sensitive sequence

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through the mid portion of the graft,

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and this patient had clinical laxity on exam.

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Here's another, uh, example of a complete graft tear,

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which is more chronic.

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Now in this stage, and we can say it's chronic

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because we no longer see any visible graft fibers,

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so similar to the native anterior cru ligament,

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it can either undergo scarring

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or completely resorb over time.

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So this is a complete tear with graft resorption.

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Here's why. Secondary signs not as helpful in the

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reconstructed ligament.

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So this is a paper, um, roughly 10 years ago, uh,

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looking at the secondary signs.

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And they found that if you had slid anterior translation,

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the tibia relative the femur, this had a kind of a low

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positive predictive value for predicting patients

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to have laxity on clinical exam.

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So if you see this finding, it won't,

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it doesn't really have a lot of meaning.

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And why is that? It's probably has to do

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with anterior cruciate ligament reconstructions.

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While we strive to attain perfect anatomic

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reconstructions, sometimes these, uh,

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ligaments are not quite placed in a perfect anatomic

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reconstruction, particularly for single, um,

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bundle reconstructions as opposed to, uh,

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double bundle reconstructions where they're trying

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to create both the antal

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and posterolateral bundles in a more anatomic fashion.

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Conversely, if you see normal alignment,

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so say this tibia was lined up perfectly

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with the posterior aspect of the lateral femoral condyle,

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then this had a high native predictive value, meaning

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that you were unlikely

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to have any translational laxity on exam.

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We've talked about roof impingement,

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particularly in the setting of tibial tunnels

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that are too far anterior.

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And sometimes you'll read in the operative notes

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that the arthroscopy will pull, will perform a notch plasty,

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meaning they'll debride a little bit of bone to make room

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for the graft, but obviously they won't want to over debride

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and create a, uh, um, a, a situation

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where you have knee laxity.

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So again, just look for any kinds of posterior bowing.

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If you see attritional changes

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or thinning of the reconstructed ligament,

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then you are likely dealing with roof impingement.

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Also, these patients may present with, um, loss

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of terminal knee extension.

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Here's just another example of a patient

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with roof impingement.

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Their tibial tunnels a little bit anterior location,

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but even even more importantly, you can notice

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that their femoral tunnels way too anterior was probably

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placed in front of that lateral intercondylar ridge

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or residence ridge.

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So similar to cystic degeneration

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of the native anterior cruciate ligament.

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This can also happen in the reconstructed ligament.

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This can in, uh, affect the intra, um, the joint portion

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of the reconstructed ligament,

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but can also affect the bone tunnels as well.

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So here you can see almost that celery stock appearance of

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that, uh, reconstructive ligament heading into bone

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and resulting in tunnel widening.

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And in of itself, if the patient has a clinical stable exam,

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probably not that important,

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but further down the road, should they tear their ligament

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and require an additional surgery

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or reconstruction, this can result in, um, problems.

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So tunnel cysts, when the largest more than 14 millimeters

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and the patients require a redo surgery, this is, uh,

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where the patients may require a two stage revision.

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So here's an example of a patient

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with enlarging tunnel cyst.

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This is, uh, by eyeball more than 14 millimeters.

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And so here's another example of a patient

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with a large tunnel cyst who had

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to undergo grafting of the tunnel.

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Wait several weeks for the bone graft to mature

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before undergoing a second ACL reconstruction.

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Here's an example of other tunnel complications.

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This was a, uh, former, uh, professional

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Football player of the Chargers organization underwent a

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bone patella tendon bone autograph.

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You can see the changes of the inferior pole, the patella,

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and you can see that the interference screw should be in

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line with the femoral tunnel

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and the bone plug that's also supposed to be in line

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with this, uh, interference screw has dropped into the joint

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and, uh, was actually astutely picked up on the MRI

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and then subsequently confirmed on CT.

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STR fractures are relatively uncommon.

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They tend to occur more on the femoral side.

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Uh, they tend to occur more, um, with larger tunnels

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and that tends to make sense.

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The lower bone you take away, the more likely you're going

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to result in a stress riser.

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So here's an example of a pediatric patient

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who underwent an ACL reconstruction

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with a distal femur stress fracture.

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Whether this is truly a causal relationship to the, um,

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anterior cruciate ligament reconstruction,

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I can't really prove or whether this is just a simple matter

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of fact stress fracturing a patient who, uh,

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with overactivity, certainly we wanna look for,

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uh, hardware complications including hardware migration.

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Here's an example, uh,

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from the literature showing a uh, button.

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Now remember, these are cortical buttons that they're placed

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through the femoral tunnel and flipped,

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and that's how they engage on the, uh,

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of the supracondylar ridge, of the distal femur.

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Much like when you button a shirt

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that keeps it secure in place.

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Here's, uh, one in the literature

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that was placed in the soft tissues,

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and these can be, uh, pretty dramatic

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because as you can imagine, the tension

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of the graph can result in, um, tension on the button and

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therefore tissue necrosis.

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One thing I didn't talk about is a different type

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of graph fixation that you may encounter are cross pins.

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These are another form of dispensary fixations,

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and these are placed along the, um, distal femur.

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And ideally these cross pins should be

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placed entirely within bone.

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But on occasion, these are an inadvertently placed within

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the soft tissue soft tissues outside the femur.

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And here's a couple of cases of broken cross pins

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that were placed outside the femur.

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Here's a case, uh, from our, um, case files

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where a cross pin was placed outside the femur

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and have actually fractured

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and migrated into the gastroc anus muscles.

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You can see here actually dissecting through, uh,

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the intramuscular vein of the gastroc neis muscle back here.

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Certainly, uh, when we have fixation screws,

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these interference screws can migrate as well.

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This one, uh, I can't tell, we don't have pre, um,

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prior imaging demonstrating whether this has extruded

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superiorly or this is how it was natively placed.

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But you can see that there's local localized arthrofibrosis

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raising the possibility of a cyclops lesion

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as we look in the second case.

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Here's another example of screw protrusion at

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the inferior aspect of the tunnel.

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And this has to be correlated with clinical findings.

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Whether this is a symptomatic finding

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or not, here's a more dramatic case of a screw

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that extruded into the subcutaneous adipose tissues.

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So this patient actually had it

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to undergo screw removal given that it was, uh, completely

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underneath the skin

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and mobile I alluded to.

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Um, when placing the femoral tunnel too far posterior,

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you can, uh, encounter this, uh, entity

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of posterior wall blowout,

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so the posterior cortical margin is violated.

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And you can see that the femoral button now is floating in

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space, uh, detached, uh, from the graft.

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And here it is on ct, not engage

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with the lateral cortical margin of the distal femur.

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I.

Report

Faculty

Donald Resnick, MD

Professor Emeritus, Department of Radiology

University of California, San Diego

Mini N. Pathria, MD, FRCP(C)

Division Chief, Musculoskeletal Imaging

University of California San Diego

Eric Y. Chang, MD

Adjunct Professor, Radiology

University of California, San Diego

Brady K. Huang, MD

Clinical Professor of Radiology

UC San Diego Medical Center

Tags

Musculoskeletal (MSK)

MRI

Knee