Interactive Transcript
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So next case, this is a 20-year-old skateboarder, uh,
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with injury two months earlier
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and he had chronic recurrent instability,
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although on further interrogation,
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I mean this is a skateboarder, right?
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So he's fallen multiple times.
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So he is had multiple injuries over his years
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of skateboarding and I believe he was professional
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or maybe semi-professional.
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He had some, uh, uh, on physical exam, some vari laxity
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and a positive quad active test.
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And basically what that is is when you have a PCL tear,
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your knee rests with slight sag, meaning
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that tibia translates posteriorly.
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And so when the patient's on the exam table
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with their knee flexed,
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once they activate their quadriceps tendon,
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that pulls the tibia forward.
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And hence that is a positive quad activation test.
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Now the reason why I pull up this case is
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'cause this patient is a chronic injury.
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So if you've reviewed the images ahead of time,
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this is his left knee, this is the symptomatic knee
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and he doesn't have a lot of edema
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or he doesn't have any edema frankly,
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to really draw your attention to where the pathology is.
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So what I've actually done is he had bilateral, uh,
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knee MRIs done with in within close proximity to each other,
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but it was advantageous for us
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because we could compare his, uh, contralateral knee,
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which was actually relatively more stable compared
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to his injured knee.
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And so I wanna focus on this fibular collateral ligament.
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Again, find this apex of this femoral epicondyle
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and you'll find the fibular collateral ligament.
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And as we followed inferiorly,
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here's the biceps semus muscle
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and tendon, there's the fib collateral.
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We can see that it just sort of dies and disappears
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before we even get to the fibula.
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Again, biceps fems tendon.
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If I show you the contralateral side,
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maybe that'll convince you
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that there's something wrong about this knee.
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Here's fibular collateral ligament,
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we're gonna follow it inferiorly.
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And here it is again, interdigitating with the biceps fems.
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And one more time, we follow this guy down inferiorly,
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the collateral ligament right here
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and it just sort of disappears there.
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So because this is not acute injury, again, you may be,
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you may easily miss this.
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And in fact, this was missed.
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This was that these were outside MRIs
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and I was asked to review it at the request
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of the orthopedic surgeon.
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Alright, so let's look at the coronal images.
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Here's the normal knee and then here's the injured knee.
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And you'll notice on the injured side,
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there's a little slight dip
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or redundancy of that fibular collateral ligament.
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Lemme go ahead and blow that up.
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Here's that slight redundancy.
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And you notice you can't really quite follow
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it down to the fibula.
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And here's the other knee, the uninjured knee.
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And here at least on this one slice, you can
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for the most part, follow
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that fibular collateral ligament all the way down.
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So this was the basis of this patient's
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chronic varus instability. But,
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Uh, from the history, the patient also had some degree
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of posterior instability or posterior laxity.
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So if you interrogate both knees,
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this is the uninjured injured side.
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You can see that there's a little bit of the thickening
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of the posterior crucet ligament.
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There are some fibers that are intact,
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but here, kind of this posterior, uh, bundle
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or poster medial bundle, if you will, is thickened,
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uh, with increased signal.
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So this tells us that there's been some sort of injury
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to the posterior cruciate ligament
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before, whereas on the normal side we can see it's nice
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uniform and thickness and signal intensity.
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And I think Dr. Chang showed you a beautiful case
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of a patient who had a posterior cruciate injury,
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posterior cruciate ligament injury
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with subsequent follow-up, how it can fibrosis
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and look almost for all intents
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and purposes, normal on follow-up exam.
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So just a good example of more of a chronic injury
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to keep, uh, a lookout for.
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So this patient I follow-up, was not,
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hadn't undergone surgery, was leaning more towards surgery
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to include a poster lateral corner reconstruction with
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or without a posterior cruciate ligament reconstruction.
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Oh, one more, uh, cherry on the top.
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Uh, I put that slide in to remind me and I think Dr.
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Pather will discuss this in her popal fossa talk.
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Um, shortly is in this injured knee, you notice that the,
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um, anterior tibial artery has a high origin from
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the popal artery.
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Usually this takes off more inferiorly below the joint line.
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So here it takes off, uh, just at
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or above, above the joint line.
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And this is an aberrant, anterior tibial artery.
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You can see how it's closely opposed to the posterior aspect
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of the proximal tibia.
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If you go to his other knee, you'll notice
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that he doesn't have this configuration.
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So it's not always bilateral.
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And if you look at the literature, the incidences
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of this aberrant anterior tibial artery, it's 2%.
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So you should be seeing it around every 50 knee cases.
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And if you aren't seeing this at all,
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you're not looking hard enough.
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And this definitely has implications in this case
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because remember,
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this patient may undergo a posterior cruise ship
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ligament reconstruction.
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So often they'll have to put trans osseous tibial tunnels
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to get to the posterior aspect of the tibia.
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And when you have this vascular structure right behind it,
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well that might result in an intraoperative
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consult to vascular surgery.
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So you definitely wanna alert your orthopedic surgeon
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to this anatomic variant.