Interactive Transcript
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Other smaller ligaments such
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as the faveo fibular ligament can be incredibly hard to see.
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And in some papers they've suggested
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that this can only be identified in as little as 8%
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of patients, and you'll more commonly see it when patients
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have a well-formed faa.
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And it can be seen as this, uh, thick, uh, structure
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of low signal intensity similar to other ligaments.
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And again, you'll notice here,
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this is your lateral inferior geniculate vessel.
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So this is a nice landmark for trying
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to identify the faveo fibular ligament,
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but again, not routinely seen.
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What about the arcuate ligament?
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The arcuate is even a smaller structure.
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So again, if you find the, um,
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inferior lateral Gena vessels behind it,
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you should see the falo fibular ligament.
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In this case, we don't see it,
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but in front of it, in front of this fat plane is
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where typically the arcuate ligament at lives.
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And it's been suggested the literature
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that these have an inverse relationship to each other.
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So if you have a thicker faveo fibular ligament,
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the arcuate ligament will be more di definitive
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and vice versa.
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It's important to also notice, uh, note that the fabelo,
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you may still have a faveo fibular ligament in the absence
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of an ossified favela.
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So let's talk about arcuate, uh, ligament avulsion injuries
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and more specifically fractures.
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Here you can see two cases of where the tip
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of the ulnar sil is a vols.
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So now we know what attaches
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to there in front the poppie fibular ligament
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and behind it the fabelo fibular ligament.
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And if we were lucky, maybe we could hallucinate an arcu
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ligament right in front of these ICT vessels.
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So the, here comes the diagnostic dilemma when we're trying
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to consider whether we were doing with a poster,
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lateral coronary injury.
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So here's a patient who had an ACL tear.
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The popliteal fibrillary ligament was torn
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and is the in arcuate ligament tear?
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Well, I already said that isolated, uh,
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or excuse me, edema in the poster lateral corner may not be,
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uh, as clinically relevant in terms
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of a patient's having a significant poster
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lateral corner injury.
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So this has born, been born out in both the radiology
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and orthopedic literature.
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Again, those big three, um, of the, uh, biceps femorals,
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the pope tendon, and the fibular collateral ligament.
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These are significant predictors
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of post later corona instability.
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And most importantly, with the fibular collateral ligament
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assessment of these smaller poster later corner structures
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here, the popal fibular ligament
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and those smaller ones that are hard
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to find really did not improve diagnostic performance
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of current MRI to help predict clinically significant
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poster later instability.
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And these are patients who require, uh,
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post lateral reconstruction.
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And this is also investigated even earlier
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In the orthopedic literature, same big three structures.
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They did found that in the majority
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of patients they also have abnormal
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popliteal fibular ligaments.
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But identification of the arcu
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and favell fibular ligaments was very hard
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and not really necessary
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to report a clinically unstable post later coronary injury.
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So in the end, don't really worry about
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those smaller structures.
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Uh, you'll probably be okay if you can't find 'em.
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So with that, I'd like to conclude, uh,
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this is probably a more appropriate, uh,
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title for this lecture.
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That the lateral ligaments are complex
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and they're comprised of two major structures.
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The antola complex subdivided into the AOL L
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and the iliotibial band and the poster lateral corner.
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In my mind, the big three we talk about is the biceps
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femoris, the poppie tendon,
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and the fibular collateral ligament.
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And you can add plus or minus the popliteal fbri ligament,
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which is included in the original description
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of the poster lateral corners.
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So with that, I'd like to conclude
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and thank you for your attention.