Interactive Transcript
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Now this is tendinosis
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and it looks like tendinosis anywhere in the body.
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The tendon is thickened signal becomes more heterogeneous.
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There may be some surface fibrillation or fraying,
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and it's not particularly bright on your
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T two weighted images.
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Now, we could argue about this little piece here.
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Maybe there's a micro tear developing.
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Uh, but we can, uh, recognize basically we have a spectrum
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between, uh, tendinosis and, and tears.
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So perhaps there's a little bit of a tear here,
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but the rest of this is all what you expect to see
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with tendinosis.
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He also has some changes related to arthrosis, uh, at the,
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uh, patello, uh, femoral joint.
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So this is very, very common.
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And when you look at tendinosis in the quadriceps,
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unlike tendinosis in some other areas,
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it's often can be hypervascular.
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Um, so if you do ultrasound, uh, you may see some, uh, micro
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angiogenesis, uh, in this area.
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If you give contrast, you may see some enhancement, uh,
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in the, in the quadriceps.
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And that's just a feature of tendinosis in this area
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and why it's often painful.
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Now, this is a patient, uh, with an acute tear.
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Now we don't normally see tears in patients
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unless the tendon is already abnormal.
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It's a very, very strong tendon.
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So the majority of patients
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who develop frank tendon tearing have a background
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of tendonosis of that tendon in the first place,
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only about 10% of quadriceps tears are considered to be due
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to just an acute deceleration trauma.
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About 90% are in patients who have abnormal tendons,
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either due to overuse aging
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and a host of systemic diseases such as diabetes, obesity,
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hyperparathyroidism, et cetera, that are known
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to weaken the tendon and its attachment, uh, to the bone.
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Here are some of the diseases that have been associated
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with quadriceps tendon tears.
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Uh, and this is particularly important when you see tearing
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in women, because in women,
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especially when you have bilateral tearing in women, uh,
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these systemic, uh,
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disorders are found in a significant number of patients.
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So consider these,
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and I especially like to think about hyperparathyroidism.
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We have a large renal failure population,
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and it's certainly very strongly associated with development
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of, uh, quadriceps tears, uh, which which can be bilateral.
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You also wanna think about these more when you are looking
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at a young patient who doesn't have tendonosis, who comes in
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with a quadriceps tendon tear,
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and you can search to see if they have any history
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of steroid use or, or diabetes
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or, uh, one of these other, uh, disorders.
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Now, in terms of description, what we wanna do is to try
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to describe these as well as we can.
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And it's difficult
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because you're dealing with a multi-layered structure.
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So in terms of thickness, thickness goes front to back.
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So this is a partial thickness tear
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because we have intact posterior fibers.
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So again, when you look at the patella,
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the patella tendon is gonna insert along the anterior half.
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The posterior half is covered by this triangular, uh,
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pre femoral, uh,
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by this triangular supra patella, uh, fat pad.
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So as I look at this case, I see
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that the tendon fibers just in front
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of the fat pad are intact.
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So I know that's vastus intermedius.
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And then I go here and I see a thinner bundle
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retracted more approximately.
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That's going to be the rectus,
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and then a thicker bundle located in the middle,
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and that's gonna be the medias and lateral alis.
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The patients may have surprisingly good function, uh,
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with just the intermedius, uh, intact.
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And we like to open up our field
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of view if we're monitoring the exam, uh,
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which unfortunately doesn't happen much of the time,
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but if we do happen to be monitoring it,
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you could open up the field of view, uh, to make sure
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that you got really captured the extent of retraction
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that you have and to be able to assess the muscle quality,
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uh, in these, uh, patients.
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So those are the partial thickness tears involving
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the anterior part.
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And these are common
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because if you look at partial thickness tears,
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the most common component to be torn is the rectus femoris.
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The partial thickness tears
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that are posterior are much more
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challenging for the clinician.
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They're not difficult for us.
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We can see here that the anterior fibers look pretty good,
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but then we have a clump of fibers that are torn
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and retracted,
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and those are the fibers that should be right in front
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of the effect pad.
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That area is irregular, so I know that's vast
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as intermediates, but this doesn't lead to the degree
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of swelling and hemorrhage at the anterior knee,
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that tearing of the anterior fibers does.
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And the patient, again, may have fairly good function.
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So this diagnosis may not be suspected, uh, clinically.
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So Mr is very, very helpful
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and it gives them a really good handle on the size
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of the gap and the quality of the underlying tendon,
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which are important factors
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for deciding whether this can be fixed.
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This is an unfortunate patient
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who had had a patellar tendon tear in the past
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that had been, uh, reconstructed,
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and he presented with repeat pain.
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You can see now we have a full thickness tear.
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All of the layers of the quadriceps are disrupted
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with fluid extravasated from the joint.
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There's a stump of disease degenerated tendon at the upper
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Patella.
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The stump approximately doesn't look too bad.
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We measure this gap,
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tell them this tissue is severely degenerated.
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It's gonna need to be debrided.
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Uh, in this, uh, in this example
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and in this tear, you can see that the change in the patella
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or position, the more extensive the tearing,
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the more changes we'll see in patella position.
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And the typical position changes that'll occur is going
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to be patella baja.
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Not very striking in this case,
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and really difficult to interpret given the previous surgery
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here, that could have led to contracture of that tendon.
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But Baja is the downward migration.
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And the other finding we'll see is that the patella
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will flex so that the space at the top widens
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and the space at the bottom remains the same
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or narrows that flexion.
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Again. It requires a high grade tear for the patella
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to assume this kind of a position.
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Now, in order to call a tear complete, you want
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to be sure it goes all the way from medial to lateral
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as we have in this case.
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So you can chase your sagittal images side to side.
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You can use the other planes.
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And here you can see on the axial images,
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there's nothing left.
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The medial ret inoculum has also been
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disrupted in this example.
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And on our coronals also, we can see
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that the tear is going all the way from medial to lateral,
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and that would be a, uh, complete tear, uh, which is, uh,
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got a big gap in this example and a tendon otic, uh, tendon.
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And this patient required a, uh, reconstruction.