Interactive Transcript
0:00
Let's move now to the patellar tendon.
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So the patellar tendon technically is not really a tendon.
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It could also be considered a ligament
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because it's going from bone, uh, bone to bone.
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But I'll use the term patellar tendon.
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Um, it can be lax in the extended position.
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So it's quite commonly you'll see these little magic angle,
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uh, artifacts, uh, related, uh, to the, uh, tendon.
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So just be aware of those. They're normal finding.
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They will disappear as you go to a more long T two, uh,
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longer t uh, sequence.
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And they're quite common here
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because the knee is extended during the mr.
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And so the patella tendon, uh, tends to be, uh, lax
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like the quadriceps.
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We will frequently see tendinopathy, uh, in this area.
0:49
This patient has partial tearing with the high grade POF
0:53
of the quadriceps from the upper, uh, patella, uh,
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and a lot of tendinosis, uh, in the, uh, patellar tendon,
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showing you the typical findings of thickening.
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And as I mentioned, uh, earlier, uh, this again,
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like the quadriceps does have a tendency to be angioplastic
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and can be painful even when you're not seeing a lot
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of fluid on your T two weighted, uh, images.
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So you, you wanna mention, uh, the, uh, the tendinosis.
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Now I want to just show you this
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'cause it looks kind of like tendinosis.
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It's more extensive in this example, but this is gout
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and this is gouty deposition within the patellar tendon.
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And it can be very small and maybe focal.
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I don't really see great bone erosion here.
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Probably some erosion right in this area has developed
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'cause there's some bone marrow edema, uh, in that area.
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Um, but you can certainly see, uh, well-defined
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or better erosions than, than in this case.
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But it's a nice example showing you
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how the diffuse involvement,
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certainly some erosive disease at the tibial tuberosity
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where the cortex looks irregular
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and there's more intense, uh, bone marrow edema.
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So gout loves the extensive mechanism,
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and this is from the same patient where he has
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aranon bursitis with hyperdense tophaceous material,
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a little bit of which is calcified.
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And again, that sort of surface blistering para articular
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erosion, that's very characteristic of gout.
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Now, in terms of partial tears, uh, they are most common
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on the articular side
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of the tendon near its patellar insertion.
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They can certainly occur distally.
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We'll often see that in patients
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with Osgood slaughter disease, for example.
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Uh, this patient has some findings suggesting sending Larsen
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with bone proliferation.
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But this is the place
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that you wanna focus when you're looking for a partial tear,
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is the undersurface fibers near the patellar tendon.
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So this type of tearing has been referred to
2:58
as a jumper's knee.
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Uh, it may be quite focal
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as we see in this example over here.
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And if you're going quickly, the tear
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may be just on one slice on your sagittal images
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because the tears are often sag oriented
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and they're quite easy to overlook.
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So really focus in on that area.
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And I like to look on my axial images as well,
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because the sagittal nature of the tear,
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you can often calculate its depth better on your images,
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uh, that are axial.
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Um, this is thought to be due to
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tensile stresses at the undersurface of the patella.
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And because of the fiber lengths being different,
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the shorter fibers in the back,
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the tensile stresses are actually higher in the
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posterior fibers.
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It's also been suggested that a prominent peaked lower pole,
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as we see in this example, can lead
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to direct impingement on the posterior fibers leading
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to this characteristic, uh, pattern of a jumper's knee.
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Now in a, in a complete tear, you can see in this patient
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that we have the opposite of patella Baja.
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We have patella alta with superior migration.
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And as a rough rule, um, again,
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you can use those measurements, none of which I really like.
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It's a rough rule you can use as measurement
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of seven centimeters from the tibial tubercle
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and, uh, on mr, we can see the tear, uh, directly, uh,
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in this example with the background of tendinosis.
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Now what we see in children is quite different.
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In children, you can get a number
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of different, uh, injuries.
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This is a, what's known as a patella sleeve injury,
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which is an avulsion of bone
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and periosteum from the patella.
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Uh, it's a, it's a equivalent to a complete tear
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of the patella tendon,
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but it occurs at the periosteum and bone.
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I do not have an mr example of this
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because we do not do pediatrics, uh, at our, uh, facility.
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I will show you Mrs.
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On some of the more common abnormalities.
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This is syn Larsen, which is a more chronic condition
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with ossification, irregularities
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and marrow edema thought to be due
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to repetitive excess traction at the patella.
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We see this usually in our young athletes, uh,
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who are involved, uh, in, in sports,
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and it's really the pediatric equivalent of, uh, the adult,
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uh, jumpers knee.
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And at the other end we have Osgood Slaughter disease.
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In this case, it's really mild.
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We have marrow edema without boning changes. This is common.
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You don't usually need to get an MR for this.
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It's diagnosed clinically, uh,
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with the focal tibial tuberosity pain
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and, uh, during the period of rapid growth.
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But it can go on to form obstacles
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and, uh, that don't unite.
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It can then be associated with chronic problems
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Of the patellar tendon, which can be torn.
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This one looks pretty intact,
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but the patient does have bursitis.
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But you do run into all kinds of issues here.
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Patient still has edema
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and they can be symptomatic well into adulthood once you
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start getting these displaced, uh, ossicles.
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So this can persist, um, as a symptomatic, uh,
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abnormality into into adulthood.