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Infarct Cx

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Our next case is a 64 year old man with a history

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of LAD stent who was found prior to this study

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to have inducible, non-sustained ventricular tachycardia

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during a recent stress test.

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So this exam was to evaluate for any scar in the myocardium

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and viability of the myocardium.

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So again, starting in our usual approach,

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we are looking first at the two chamber

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and not a ton to say on this one.

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It looks like to me there may be a little bit of thinning

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relative, at least to the rest of the myocardium here

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towards the apex, kind of the anterior segment of the apex

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and all the way around.

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I mean, we know that he is had an LED

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territory infarction at some point,

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so perhaps this is gonna end up being residual from

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his LED infarction.

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The other thing, and I think this will show a little bit

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more nicely on the short XS images,

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I think he's overall has a little bit of

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left ventricular hypertrophy,

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which sometimes can make our lives easier

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'cause there's just more, at least in terms

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of viability imaging, there's more

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myocardium to sort of look at.

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So there's the two chamber view,

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four chamber view here.

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Again, not much to add, but always important to look at.

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And same on three chamber.

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Overall, this person's function looks pretty good

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and I, I do think that there's gonna be some left

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ventricular hypertrophy when we look on the short axis.

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So now I'm gonna move our way down from base to apex

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and there is gonna be circumferential hypertrophy.

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I won't measure here since this is a

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schemic cardiomyopathy talk,

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but it's important to at least recognize this

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and comment on it when appropriate.

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You know, it can be related to many things,

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including chronic hypertension,

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hypertrophic cardiomyopathy, amyloidosis.

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So all things to at least note here,

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even on patients where the exam is not indicated,

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a little arrhythmia in this patient.

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So the image quality is not as pristine

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as maybe we would hope,

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but this is kind of like what happens in real life.

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So starting to get into

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some interesting findings.

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I think here I'm gonna start to draw your attention to sort

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of this region, the basal to mid

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anterolateral segment here.

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And as I scroll through the cynic lip,

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you'll probably see it looks a little bit relative

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to the rest of the myocardium,

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maybe a little bit thin there,

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or at least something is different about the wall motion,

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maybe a little hypokinesis in that region.

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And scrolling through, again, a little bit

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of gating artifact here in this patient

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Who, who may have had some arrhythmia,

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this is gonna be a little bit better quality example.

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And I think you can see if you contrast that kind

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of anterolateral segment relative to

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the other segments of the myocardium,

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which are contracting quite vigorously, that looks

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a little bit hypokinetic

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or maybe even a kinetic in that segment.

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More of the same here.

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And now we're down into the apex, which relative to the rest

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of the myocardium may look a little bit than like we

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saw in the long axis.

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So now go moving to LGE imaging.

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So we're gonna start with the short axis here.

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And almost immediately as we get into the first kind

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of true basal slice here, we see that there's gonna be some

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late gata limb enhancement involving

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that anterolateral segment.

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And here it gets quite intense relative to maybe

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what we would see in more

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of an non-ischemic cardiomyopathy or something like that.

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There is a lot of contrast that's been taken up here

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and it's definitely in a sub endocardial distribution.

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I cannot identify any endocardium below this area

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of Lake Catalan enhancement.

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And as we keep clicking down, clicking through kind of in

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that area where we saw, you know,

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questionable wall motion abnormality

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and thinning, we're definitely making out some late

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Catalan enhancement in this region.

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And more of the same as we move down.

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Notice we kind of don't have any as we get

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to the true apex in that particular region.

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So here I'm kind of scrolling up

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and down through that region.

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And then as we get down into the apex though, we start

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to see some inferior sub endocardial,

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late gado limb enhancement

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that becomes nearly circumferential at the apex.

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So this is probably related

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to his old LAD territory infarction with sort

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of like a wraparound LAD anatomy

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that you know is supplying kind

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of the inferior aspect of the apex.

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But this is gonna be actually most likely a circ territory

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myocardial infarction here.

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And I, I really want to use the long axis in this case

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to try to understand the morality of this

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because it's a little challenging I think in the setting

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of hypertrophy here to know kind of like

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what the true myocardial thickness is.

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So let's see if the four chamber is

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gonna cut right through here.

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So I'm gonna pull the four chamber next

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and see if we can get a better idea of what

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the mural extent is here.

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And I think we probably can, this is probably the best view

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that's kind of cutting right through it.

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And you can see this is probably normal myocardial thickness

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here, and this is maybe normal myocardial thickness here.

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And so here is kind of our infarcted region

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and it's actually less extensive than I think it looks

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Like on the short axis.

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So this is again, why a long axis view can be very helpful.

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This I would probably put at 50%,

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or probably in my kind of world, I just use that 50% cutoff

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and say, you know, less than 50%.

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And I would say this has a high likelihood

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of functional recovery.

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So again, here's what it looks like on short axis

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when we get thinned out a little bit.

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Sometimes, especially in a hyper sort of area of LVH,

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you can get a little bit of where's the blood pull stop

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and the enhancement start.

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And that's why I think those long axis views can be quite

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helpful and kind of give you a little bit more myocardium

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to look at to see, okay, what's actual normal thickness

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in this patient versus areas of enhancement.

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So circumflex territory, myocardial infarction

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with viability.

Report

Faculty

Bradley D. Allen, MD, MS

Assistant Professor; Chief, Cardiovascular and Thoracic Imaging

Northwestern University Feinberg School of Medicine

Tags

Vascular

Myocardium

MRI

Coronary arteries

Cardiac Chambers

Cardiac