Interactive Transcript
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Our next case is a 64 year old man with a history
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of LAD stent who was found prior to this study
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to have inducible, non-sustained ventricular tachycardia
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during a recent stress test.
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So this exam was to evaluate for any scar in the myocardium
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and viability of the myocardium.
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So again, starting in our usual approach,
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we are looking first at the two chamber
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and not a ton to say on this one.
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It looks like to me there may be a little bit of thinning
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relative, at least to the rest of the myocardium here
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towards the apex, kind of the anterior segment of the apex
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and all the way around.
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I mean, we know that he is had an LED
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territory infarction at some point,
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so perhaps this is gonna end up being residual from
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his LED infarction.
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The other thing, and I think this will show a little bit
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more nicely on the short XS images,
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I think he's overall has a little bit of
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left ventricular hypertrophy,
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which sometimes can make our lives easier
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'cause there's just more, at least in terms
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of viability imaging, there's more
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myocardium to sort of look at.
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So there's the two chamber view,
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four chamber view here.
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Again, not much to add, but always important to look at.
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And same on three chamber.
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Overall, this person's function looks pretty good
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and I, I do think that there's gonna be some left
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ventricular hypertrophy when we look on the short axis.
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So now I'm gonna move our way down from base to apex
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and there is gonna be circumferential hypertrophy.
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I won't measure here since this is a
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schemic cardiomyopathy talk,
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but it's important to at least recognize this
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and comment on it when appropriate.
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You know, it can be related to many things,
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including chronic hypertension,
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hypertrophic cardiomyopathy, amyloidosis.
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So all things to at least note here,
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even on patients where the exam is not indicated,
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a little arrhythmia in this patient.
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So the image quality is not as pristine
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as maybe we would hope,
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but this is kind of like what happens in real life.
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So starting to get into
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some interesting findings.
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I think here I'm gonna start to draw your attention to sort
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of this region, the basal to mid
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anterolateral segment here.
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And as I scroll through the cynic lip,
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you'll probably see it looks a little bit relative
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to the rest of the myocardium,
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maybe a little bit thin there,
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or at least something is different about the wall motion,
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maybe a little hypokinesis in that region.
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And scrolling through, again, a little bit
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of gating artifact here in this patient
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Who, who may have had some arrhythmia,
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this is gonna be a little bit better quality example.
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And I think you can see if you contrast that kind
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of anterolateral segment relative to
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the other segments of the myocardium,
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which are contracting quite vigorously, that looks
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a little bit hypokinetic
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or maybe even a kinetic in that segment.
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More of the same here.
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And now we're down into the apex, which relative to the rest
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of the myocardium may look a little bit than like we
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saw in the long axis.
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So now go moving to LGE imaging.
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So we're gonna start with the short axis here.
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And almost immediately as we get into the first kind
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of true basal slice here, we see that there's gonna be some
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late gata limb enhancement involving
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that anterolateral segment.
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And here it gets quite intense relative to maybe
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what we would see in more
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of an non-ischemic cardiomyopathy or something like that.
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There is a lot of contrast that's been taken up here
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and it's definitely in a sub endocardial distribution.
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I cannot identify any endocardium below this area
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of Lake Catalan enhancement.
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And as we keep clicking down, clicking through kind of in
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that area where we saw, you know,
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questionable wall motion abnormality
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and thinning, we're definitely making out some late
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Catalan enhancement in this region.
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And more of the same as we move down.
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Notice we kind of don't have any as we get
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to the true apex in that particular region.
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So here I'm kind of scrolling up
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and down through that region.
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And then as we get down into the apex though, we start
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to see some inferior sub endocardial,
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late gado limb enhancement
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that becomes nearly circumferential at the apex.
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So this is probably related
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to his old LAD territory infarction with sort
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of like a wraparound LAD anatomy
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that you know is supplying kind
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of the inferior aspect of the apex.
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But this is gonna be actually most likely a circ territory
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myocardial infarction here.
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And I, I really want to use the long axis in this case
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to try to understand the morality of this
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because it's a little challenging I think in the setting
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of hypertrophy here to know kind of like
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what the true myocardial thickness is.
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So let's see if the four chamber is
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gonna cut right through here.
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So I'm gonna pull the four chamber next
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and see if we can get a better idea of what
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the mural extent is here.
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And I think we probably can, this is probably the best view
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that's kind of cutting right through it.
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And you can see this is probably normal myocardial thickness
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here, and this is maybe normal myocardial thickness here.
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And so here is kind of our infarcted region
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and it's actually less extensive than I think it looks
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Like on the short axis.
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So this is again, why a long axis view can be very helpful.
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This I would probably put at 50%,
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or probably in my kind of world, I just use that 50% cutoff
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and say, you know, less than 50%.
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And I would say this has a high likelihood
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of functional recovery.
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So again, here's what it looks like on short axis
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when we get thinned out a little bit.
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Sometimes, especially in a hyper sort of area of LVH,
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you can get a little bit of where's the blood pull stop
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and the enhancement start.
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And that's why I think those long axis views can be quite
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helpful and kind of give you a little bit more myocardium
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to look at to see, okay, what's actual normal thickness
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in this patient versus areas of enhancement.
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So circumflex territory, myocardial infarction
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with viability.