Interactive Transcript
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Next case is a 58 year old woman who presented
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to the emergency department with chest pain.
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She didn't have any past medical history,
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but her ECG findings were
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concerning enough to take her to cath.
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I'm gonna not tell you what the cath showed
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until we see the case here a little bit.
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So I'll show you first the cene as usual.
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So our two chamber view, I think
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hopefully at this point you're getting
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comfortable looking at these.
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And I think if I freeze it around this frame, one
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of the things that's pretty clear here in the anterior
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and apical inferior segments, there's definitely thinning
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and hypokinesis relative to these basal segments.
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So already on this two chamber view, we're sort
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of suspicious that there's something going on here.
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And then we can also see on this four chamber view here in
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kind of the apical
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and mid for septal segments here we can see similar
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findings, some of this thinning
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and wall motion hypokinesis here,
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and then three chamber, kind of an incidental finding.
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If you look at the aortic valve right here,
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this person has aortic insufficiency, which you can see kind
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of coming back across ATUs.
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But then focusing now again on the myocardium,
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you can see again sort of this relative thinning involving
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septum apex and kind of infra lateral segments of the heart.
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So kind of this apical region is looking not super happy.
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We now move down the short axis images from base to apex
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where we see some myocardium here, basal
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function here, everything's really
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contracting vigorously thickening well looking okay,
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but then one
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or two slices later, as we start to kind of get into the mid
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region, you start to see thinning
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and hypokinesis here of the anterior segment.
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A little bit of anterior septal,
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really this anterior lateral segment as well.
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And then as we start to get down to the true apex,
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it's very thin circumferentially, so
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certainly thinning
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and pretty much kinesis of these apical segments,
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which is similar to what we saw
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on the long axis use.
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So now if we go
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and we look at our findings on LGE imaging, again,
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this is a person with chest pain
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and we've just had a calf going from base to apex,
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not a ton of LGE here, even in areas where we saw a lot
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of thinning and wall motion abnormalities in the
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mid to apical segments.
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But what you do start to see as you kind
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of get down here towards the true apex here is
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where we really start to see LGE
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and a kind of vascular distribution.
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So maybe the first site here that we see is kind of lateral
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aspect of the apex.
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There's sub, sub endocardial, transmural, LGE,
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similarly here in the more inferior aspect of the true apex.
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And then if we get out here to the far apex, it looks like
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probably transmural.
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So we'll take a look at our long axis views.
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I think the three chamber may be a good view
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to take a look at here.
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This is, I'm clicking through the three chamber.
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You start to see again as we get into the apex here,
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this is gonna be kind of lateral aspect of the apex,
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our inferior and infra lateral aspect of the apex.
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You see transmural, LGE here, probably some transmural,
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LGE kind of more circumferentially at the apex.
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As I click through these three chamber slices,
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look at a similar view here on the four chamber.
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And on some of these views you can see the whole apex has
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completely got LGE, kind of transmural, LGE.
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And then a few areas maybe that have a little bit
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of less transmural, LGE, so not the entirety of
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The segments that were Hypokinetic.
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You know, again, just as a summary, you know,
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we really see hypokinesis involving a lot of
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what looks like LED territory, these anterior segments.
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But the LGE is primarily focused down
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And the apex itself, you know,
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from a cardiac MR standpoint, I think what we would say is,
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you know, there appears to be apical myocardial infarction.
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That is, you know, depending on the chronicity
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of the finding and the presentation, you know, is transmural
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with low likelihood of functional recovery.
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Now this was more of an acute finding
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or you know, kind of when we're doing the MR Subacute.
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So it's a little viability,
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still a little bit challenging when they're
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still kind of subacute.
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What this actually turned out to be on cath,
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this was the case of spontaneous coronary artery dissection.
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So the patient came in with chest pain.
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50 year old women are tend to be predisposed
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to spontaneous coronary artery dissection or scad.
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That's kind of the vernacular.
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And so it was a pretty long segment distal LAD
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or mid to distal LAD.
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And so it's probably that's what's causing a lot
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of this wall motion abnormality is maybe more of a ischemia
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or even sort of a stunning, uh,
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appearance of the myocardium.
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And then unfortunately,
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probably did have true myocardial infarction of the apex.
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So this is a case of SCAD and this woman.