Interactive Transcript
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As we consider the mechanism of injury
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for ulnar collateral ligament abnormalities at the thumb.
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Historically this was referred to as the gamekeeper's thumb,
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and as we think about the times when we had hunters trapping
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small animals, sacrificing them on site, they would do so
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by breaking the neck of the animal
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between the thumb and index finger.
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And we can imagine that that could be a chronic, repetitive
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injury or it could be an acute injury
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or even acute on chronic, of course.
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Now the more common mechanism would be something like this,
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the thumb being, uh, exposed
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to this abduction force through falling in this case
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with the ski pole in the hand that that the, uh,
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more classic again, contemporary injury.
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And again, we could imagine this being a chronic repetitive
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microtrauma perhaps for the beginning skier
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and acute on chronic or specifically an acute lesion.
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So as we think about the injuries that can occur here,
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we see the hyper abduction force at the level
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of the metacarpal phal joint.
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And you can imagine here again, a stretching phenomenon,
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a plastic bowing type deformity
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with chronic repetitive microtrauma
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and acute post traumatic event again, or acute on chronic.
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As we consider how this diagnosis with historically made,
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it was through stress radiographs of the thumb.
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Here you see the fingers
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of the examiner placing stress at the MCP
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to determine whether there's widening at the ulnar
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aspect of the joint.
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As we look at the radiograph lines placed along the long
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axis of the metacarpal long axis of the proximal phx,
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that angle is what we're looking for.
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If it's greater than 35 to 45 degrees considered
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to be abnormal.
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If you have the other side for comparison,
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if there's a greater than 10 degree difference
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between the normal slash asymptomatic or uninjured side
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and the injured side, that would be considered abnormal.
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With the advent of more advanced cross-sectional imaging
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techniques, such as mr, there is no need now
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to add an additional hyper abduction force
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to the stress radiograph.
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Rather, we can non-invasively
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and directly visualize those structures.
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As we think about a classification system
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for ulnar collateral ligament injury, we can have
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that elongation, that plastic bow deformity.
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In this setting, we would either have an injury
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that didn't result in a complete failure,
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or again, perhaps chronic repetitive microtrauma.
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Other areas in the body you may, uh,
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encounter such an injury.
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Think about the medial patella reac where it becomes
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attenuated and lax, where you'll see subluxation at
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that patella femoral articulation.
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Something similar can happen with the UCL, again,
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considering that hyper abduction force
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with failure at the distal attachment.
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This consider the type one lesion.
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Remember that if there's no osseous component,
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the plain fill may look completely normal In these cases,
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because we're dealing with the ulnar aspect
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of the articulation, soft tissue prominence
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and those findings are sometimes obscured by
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that first web space.
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Here's a variation of that type one.
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We're seeing failure at the UCL
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but not at its distal attachment,
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rather at its proximal attachment.
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As you look at the fluid sensitive sequence,
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the secondary finding of seeing
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that high signal intensity extend proximally down the
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metacarpal shaft telling us, alerting us to the fact
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that we've got capsular insufficiency.
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In this case, we can clearly see the torn and detached end.
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However, you can imagine if this person presented
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to us more sub acutely, perhaps the presence
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of granulation tissue
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and synovial hypertrophy, soft tissue changes may obscure
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that distal, or excuse me,
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proximal discontinuous attachment site arrow here again
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pointing to that discontinuity.
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Much more high resolution study here acquired
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with a focal microscopy coil.
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The advantage you can clearly understand,
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very high resolution imaging, the disadvantage,
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very limited field of view in this case.
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As you look at the ulnar collateral ligament,
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we can clearly see the discontinuous distal end from the
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base of the proximal vein links.
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We can see the intact relationship
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of the abuc diap neurosis lying superficial to that
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inside two UCL.
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By inside two, I mean it has not retracted proximally.
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And we can even see detail with respect
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to articular cartilage at the articular surfaces
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and easily identify things like bone marrow edema,
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subcutaneous edema as well.
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When we think about the type two lesion,
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this probably least encountered failure in the mid substance
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of that ulnar collateral ligament
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and the type three lesion, O osteos AULs.
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And now we move into the territory
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where these lesions are identified on plain film.
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When we look at the plain film,
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we should not only identify the irregularity
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of the ulnar base of the proximal pH length,
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but really get every piece
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of inflammation we can out of that plain film.
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In this case, the displaced fragment coming
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to lie proximal at the level of the joint line.
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That may give us a clue with respect
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to a potential complication with the
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Ligament, the torn component with the osseous fragment.
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And again, its relationship with the overlying a neurosis.
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As you look at the T one weighted mr, we know
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that we're gonna closely look at the subc chondral bone
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plate, look for irregularity, in this case,
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a small osseous fragment.
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As you look at the soft tissues distorted, in this case,
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thickened, but difficult to really assess again,
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the ligament proper in the subacute setting,
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this can be the case
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because you've got this granulation tissue
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that ultimately develops.