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Wk 6, Case 4, Hip MR - Review

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So next case that we had a 30 year old male complaining of hip pain since

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falling down a slope, while carrying a refrigerator,

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at which time he heard a pop. So the uh,

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main findings in this case was, uh, subc, chondral, uh,

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signal abnormality in the intra superior femoral head.

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We can see there is like this scent and then there's flattening of the

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articular surface. This is AGRE image.

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I'm waiting for the, the T two fat set images to come.

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The coronal T two fat set images.

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We can go back to this axial proton density.

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And what we can see there is a lot of reactive marrow edema,

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reactive joint effusion.

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And the main abnormality seems to be this subc chondral

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geographic area. We have this ous, uh, linear, uh,

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high point and signal and with, um,

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a bright signal in the subc chondral region. So this will be your, uh,

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subc chondral fracture. And this, uh, ous line suggests avascular necrosis.

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There is because of, uh, like a prominent, uh,

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subc chondral fracture, there is articular surface his collapse.

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So these are changes of avascular necrosis.

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The changes are acute and that's why we have reactive mar edema and then a

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reactive joint effusion. So, uh,

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until the images are loading completely. Let's, um,

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review some points about avascular necrosis.

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So early detection of avascular necrosis is important.

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All treatments are geared towards preservation of the femoral head.

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And this is more successful when we do this early in the course of the disease

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and of all imaging tests,

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MR is the most sensitive means to diagnose av and because, uh,

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it picks up the earliest mar edema, like it,

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it takes time for it to show up on CT x-ray, and mr,

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and probably bone scan 'cause the two things.

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But bone scan will have a lower resolution.

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These are the two imaging modalities, but Mr would be the most sensitive of all.

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So what are your classic imaging features? That on T one weighted images,

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we see a crescentic, uh,

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band of low signal intensity in the superior portion of the femoral head

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bone marrow.

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And this band is thought to represent the reactive interface between the

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necrotic bone and the reparative, uh,

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zones that typically extend to the subc chondral plate.

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And on T two weighted images,

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you get this classic double line sign where you have this high signal,

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um, uh,

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linear high signal along the inner side and a low signal on the outer side.

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So that's your double line sign.

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So the high signal is from granulation tissue and the low signal is from the

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necrotic bone. So, uh,

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currently the value of MR is more for, uh,

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diagnosis and also the helps and prognosis by giving the size and location of

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the lesions. So like small size, uh,

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lesions that are more in the medial superior portion, they, uh,

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tend not to collapse.

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So they can be amenable to conservative treatment and avian that does not extend

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to the subc chondral margin also has good prognosis regardless of the size of

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the lesion.

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But if you have bigger lesion that ex with articular of his involvement,

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like that extent all the way to the subc chondral bone, um,

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they are more likely to collapse.

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And we need to do core decompression at an earlier stage for these patients. So,

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and then there was, um,

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one study that showed that 74% of femoral hit collapse was seen in

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the region of avian and MR involved poor than two thirds of the weight bearing.

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So if more the weight bearing surface areas involved higher chances of

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femoral head collapse,

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there's also a classification on MR that's based on the signal intensity of this

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dead ma like the, the, the infarcted bone,

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how it looks on T one and T two weight images. It can have fat,

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it can have blood, it can have fluid, or it can have fibrosis.

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It actually shows the evolution of changes in avascular necrosis and just

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suggests the timeline of, or tells you about the acuity of the, the problem.

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And there is this more detailed classification and we know that, look, you know,

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we do this classification too where like, uh, stage zero is, uh,

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everything is normal. Stage one is when your MR is showing mar edema.

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Stage two is you have changes of AV N but there is no crescent sign.

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Stage three is when you have a crescent sign.

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Late three is when you have collapse, and four is when you have osteoarthritis.

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So that's the basic grading. So when you have an MR of avascular necrosis,

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let's see if all our images are here. Not yet. So when you have an mr, uh,

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first of all,

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we need to make the diagnosis and that's pretty easy when you have this

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geographic area of, uh, yeah,

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I was saying when you have the subc chondral geographic area of altered mirow

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signal intensity, you know, it's avascular necrosis. So once we have that,

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the other things that we need to do on MR imaging to give the approximate, um,

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area of how much of the femoral head articular surface it's involving, it, it's,

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it's, uh, nothing objective. It's more subjective where you tell, okay,

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I think it's, it's 30 to 40% of the femoral head that's involved.

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And then you'll do the same thing on coronal images. Then, uh, we tell,

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uh, if there is a subc chondral fracture,

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like it's there in this case we see this nice fluid signal in the subc chondral,

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uh, right underneath the articular cortex.

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And then there is associated articular surface collapse.

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So these are your pertinent positive findings that we need to mention with this

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case. And if there is, uh, any secondary osteoarthritis in the joint or not.

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So here the articular cartilage looks pretty decent, so, uh,

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it's probably your stage three. Yeah. So, uh,

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first to make a diagnosis of avascular necrosis when we have these, um,

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geographic area of subc chondral marrow signal abnormality,

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and the other things that we need to tell how much area of the articular surface

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of the femoral head is involved,

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if there is any articular surface collapse and if there are secondary

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osteoarthritic changes.

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So that completes the report on cases of avascular necrosis. Okay.

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Any questions on this one?

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Um, you said just a subjective assessment of the percentage of the mm-Hmm.

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White brain surface involved. So are you,

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are you just looking at the sort of upper half of the

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sphere of the federal head? No, no. You're looking at that.

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What whatcha you looking at?

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No, you're looking at the entire article surface, like starting from here.

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I'll just trace it all the way till here. So from here to here,

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if you think it's a hundred percent how much of this area is in what?

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So I would say it's, it's predominant pro, probably around 40%.

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But you do that assessment,

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like when you're scrolling that entire stack from anterior to posterior,

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and then you'll also do it on sagittal.

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We cannot like ignore the sagittal because in fact maybe,

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maybe smaller on coronal, but it is more wider on the sagittal images.

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So that increases the area. And then, then the main thing is, again,

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don't have to be very accurate.

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All they wanna know is if it's less than 50% or more than 50%.

Report

Patient History

30-year-old male complaining of pain since falling down a slope while carrying a refrigerator, at which time he heard a pop.

Findings

SKELETAL: Mildly depressed osteochondral fracture representing avascular necrosis involving the superior to anterosuperior left femoral head weight-bearing surface measuring 1.6cm in depth, 3.3cm in anteroposterior dimension and 3.2cm in width. The lesion involves at least 75 percent of the weight-bearing surface of the femoral head.

Moderate to severe reactive confluent osteoedema extending down to the intertrochanteric region.

PELVIS: Left sacroiliac joint, iliac blade, iliopectineal and ilioischial lines are intact.

JOINTS: Loss of the normal femoral head sphericity of the bilateral femoral heads with widening of the femoral head-neck junction. The left hip alpha angle is of 71.7 degrees.

Synovial pitting seen at the lateral aspect of the femoral head-neck junction more conspicuous in the right.

Moderate left hip joint osteoarthrosis. Diffuse chondral plate delamination with moderate reactive stress osteoedema involving the superior to anterosuperior acetabulum.

Moderate reactive joint effusion without internal debris or free bodies. Reactive synovitis.

ACETABULUM/LABRUM: Chronic-appearing anterosuperior labral tear with a subcentimeter dystrophic ossification. No displacement. No paralabral cysts.

MUSCLES/TENDONS/LIGAMENTS: Adductor and abductor muscular compartments are intact.

Ligamentum teres, ischiofemoral and iliofemoral ligaments are intact.

OTHER/SOFT TISSUE: Unremarkable pelvic basin contents.

Normal neurovascular bundle.

Impressions

1. Mildly depressed avascular necrosis of the left femoral head measuring 1.6cm in depth, 3.3cm in AP and 3.2cm in width.

2. Background of combined femoroacetabular impingement (FAI) due to cam-type and secondary pincer-type deformity. These last findings are secondary to a chronic repetitive microtraumatic anterosuperior labral tear with a subcentimeter reactive dystrophic ossification.

3. Moderate left hip joint osteoarthrosis with diffuse chondral plate delamination and moderate reactive stress osteoedema involving the superior to anterosuperior acetabulum.

4. Moderate reactive joint effusion with capsulosynovitis.

Case Discussion

Faculty

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Gitanjali Bajaj, MD

Assistant Professor

University of Arkansas for Medical Sciences

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Brian Y. Chan, MD

Assistant Professor of Musculoskeletal Radiology

University of Utah

Todd D. Greenberg, MD

Radiologist

ProScan

Tags

Musculoskeletal (MSK)

MRI

Hip & Thigh