Interactive Transcript
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So the first case, it was a 72 year old male with, uh,
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anterior pain with squatting. Okay? And first off,
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I actually probably spend the most time on localizer sequences. Uh,
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I love anatomy, but as we all know,
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the localizer sequences are typically larger field of view. And,
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uh, I, you know, I catch some, some things sometimes, uh,
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incidentals that may or may not be pertinent to, uh, the patient's presentation.
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But, uh, sometimes we do catch a rare cancer. So I do like to, uh,
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look at the localizers. And funny thing, I,
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I tend to spend a little bit more time, uh, sometimes on that, especially on,
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um, pelvises and what have you, or spines. Okay. But, um,
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moving right along. Uh, I am a structural sort of person. Okay?
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I, I did not grow up, uh, unfortunately, uh,
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back in the day when we still hung films.
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So the older staff I've noticed that I've worked with, they tend to, um,
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look at the images by planes, and they, they, so,
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so they look at all the structures on one plane.
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So they'll work through from far posterior, far anterior on, on a coronal,
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let's say, starting with the patella, and then work their ways all the way back.
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Okay? But the way I do things, and I've been asked this before, and just,
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just to get that, uh,
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get this outta the way right from the start and for future, um,
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discussions and, uh, office hours or my office hours, um,
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I tend to divide all the joints into a few broad categories.
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Okay? I do ligaments, tendons, joints,
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ancillary stuff to the joint, okay? Uh, soft tissues, muscles,
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and, uh, bone morphology and marrow signal. So that's my checklist.
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So you can sort of get a sense that I'm a structural sort of person.
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I like to look at each structure in each plane, be it, you know, uh, you know,
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so, so for instance, to extrapolate that checklist out to a knee,
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if I were dictating a stone cold, normal knee, my ligaments,
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for lack of a better term, sorry, I, I start with the menisci, though,
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obviously the medial and lateral menisci.
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And then along with the ligaments I do next, the cruciate ligaments.
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My next on the checklist is the medial and lateral supporting structures. Okay?
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Those, those, uh, obviously are capsular ligaments. And as, as,
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as some of you may know, medial, medial medially and laterally, the capsular,
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uh, the, the ligaments about the knee are divided into about three layers. Okay?
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Um, I noticed in some of your checklists, you use tibial collateral,
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ligament fibular, collateral ligament, what have you. That's fine.
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Lateral collateral, ligament, just stay.
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I just ask that you stay consistent between your terminology, especially when,
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when talking to your clinicians, you know, talk to your clinicians,
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see what terms they use so that you're, you guys are communicating effectively.
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Okay? Next on the, the, the checklist for the knee, I,
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I go from media lateral supporting structures. I go to the, um,
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extensor mechanism and fat pads, then sliding right into the joints.
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I look at joint alignment, the amount of fusion, whether they're synovitis,
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pase, particularly media superior pika, which are the most common. Okay?
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Then I look at the cartilage, starting at the patella femoral compartment,
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and then the median lateral femoral tubal compartments.
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Then I check for popal cysts. Okay?
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And then finally I wrap up with soft tissues, muscles, bone, morphology,
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and marrow signal. Okay? So just before we get into the case,
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just to fly through that real quick, okay? Um,
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so if I were looking at this case, I'd look at the menisci, okay?
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Just scrolling through real quick. Um, you know,
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on the sagittals and coronals, that's gonna be key for me.
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Then I look at the cruciate, okay? While I'm on my sagittal,
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I will actually jump back to the coronals, okay?
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And I also like the axials for the cruise ships as well, especially for the ACL,
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because the ACL, uh, depending on who you read, okay,
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is divided into two bundles, right? Uh, AMPL is the pneumonic.
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I teach my trainees and your medium posterior lateral, uh, bundles, okay?
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So I find that the axials are better to, uh, better, um,
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show a partial terror, particularly of one, one or, or other of the bundles.
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But mind you, mind you, okay, groups out of Pittsburgh,
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they're actually doing fancy computer aided, um,
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studies where they've actually identified up to 11 or 13 bundles.
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And for those that are interested in knee and AACL reconstructions,
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while you're studying, you may run into the what's been turned,
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the double bundle technique where they take parts of where, wherever graft from,
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wherever, and they loop it over, and they, they, they, they, uh,
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double it over on itself.
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So now what some people are doing are double and triple bundles, or even a,
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um, uh,
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increasing the number of bundles to make things more anatomic. But granted,
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once you tear an ACL, you re you're really never gonna be the same. Okay?
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So right after the ACL, obviously I go to the, the PCL. Okay?
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Next, I, what I find, uh, is the medial and lateral supporting structures,
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and the reason why I call it a medial and lateral supporting structures,
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and what I've been trained by my mentor and now colleague, Dr. Resnick,
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is because of the different layers, okay?
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So walking probably the more important things, okay?
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The second and third layers, okay?
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That is gonna be the tibial collateral ligament, okay?
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And the deep medial meniscal femoral,
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and the deep medial meniscal tibial ligaments, or AKA, the coronary ligament,
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walking a little bit more posteriorly, okay? Sorry,
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my laptop is little, okay? We have the posteriorly ligament, okay?
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And then obviously we have, you know, starting to get into the OPLL,
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the semimembranosus tendon, and all of its arms, okay?
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And obviously the posterior joint capsule,
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walking around the posterior joint capsule and coming back from posterior to
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anterior, okay? Looking at the lateral supporting structures, okay?
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I look at the pop fib ligament, okay? Probably, uh,
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apologies. So this right here,
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okay? This little band right here, the pop lidio fibular ligament, okay?
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Then we have the pop lius tendon, then obviously the biceps pems.
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And if you read the literature, there's about three or four insertions. Um,
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but the majority of them are going to happen, uh, at the, uh, the fibula,
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obviously the, the lateral aspect of the proximal tibial epiphysis, okay?
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Coming out more, uh, anally than now.
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We have what's been termed in the popular literature,
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the anterolateral ligament, okay? And that's what,
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when that tears or pulls off a piece of bone that's been called the, uh,
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sagon fracture, we know it better as a sagon fracture. Okay?
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And then now mind you too, the, the proper term, uh,
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even though the pop culture or the popular news has coined this as the
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anterolateral ligament, it's,
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it's better known as the mid third lateral capsular ligament. Okay?
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That's the original term. But, uh,
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people in the popular literature have been trying to, um,
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show that this is a new ligament, but it's actually not.
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And we've actually written about that for those that are interested.
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Skeletal radiology a couple years back. Okay? And then finally,
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obviously you have wisp of the, that biceps femme, but far anteriorly, okay?
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The main insertion of the IT band or the ileal tibial band distally is gonna be
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at gertie's tubercle, but recent literature, um, is, uh, and also review,
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nice review article in skeletal radiology. Um, and please feel free, you can,
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you can, um, uh, reach out to me via email, Olivia and the others can,
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can direct you. But, and I'd be happy to share some of these PDFs or,
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or citations with you. But, uh,
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relatively recent literature has shown that the it band actually has five
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insertions,
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but the main important one is gonna be at the gerdes tubercle right here.
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So that takes care of the medium lateral supporting structures, okay?
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Moving right along and, and sort of going into our case and, and we'll go,
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come back to the findings in a bit, but moving right along to my next c check.
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Uh, next thing on the checklist is gonna be the extensor mechanism, okay?
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And that's obviously gonna be the quadriceps, which consists of the VA triplets,
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vass medias, intermediates and laterals,
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and the rectus fous bar anteriorly inserting upon the patella and the
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quadriceps, uh,
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pre patella p plate or what some people also call the patella quadriceps
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continuation written originally in the radiology literature by Dr. Resnick,
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okay? And Dr. W back in, I think it was 2009 ish or something like that,
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a nice cric study in AJR for those that are interested. And that, uh,
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continuing on the patella tendon, obviously, which inserts upon the ligament.
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Now, some of you, I've noticed, have called this a, um,
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a patellar ligament or even a quadriceps ligament. That's fine, right?
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It's bone, bone to bone, you know, it's gonna be a ligament. So again,
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I just ask, you know, just know what your clinicians are using,
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and I kind of just dictate to what the clinician is, is used to hearing.
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I'm fine either way, calling it tendon or ligament,
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especially when obviously it was between bone and bone. Next, I check the, the,
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the fat pads, okay?
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And the three or four fat pads that I like to check on a knee are gonna be the
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pre femoral fat pad, which is right here, the super patellar, uh, fat pad,
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the hoffa fat pad, and then the paraship fat pad. Okay?
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Depending on who you read the super patellar fat pad,
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when EMS can be problematic, some people believe in it, some people don't. Okay?
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Pre femoral fat pad, you really don't see any problems here.
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Usually the ones that I really pay attention to are Haas fat pad,
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where you see the, uh, the edema,
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particularly sup laterally with patella mal tracking,
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but you can also see it in things with Haitis, okay? Uh,
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originally described by our group back in the, uh, I think the two thousands,
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or maybe late nineties, especially with HIV patients, they can get haitis.
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Okay?
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The other thing that they can get and more recently written about is PeriShip
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fat pad edema, okay? And then th this,
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basically this fat pad right here in between the tendons, or sorry,
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the ligaments here, it is an intraarticular, extra synovial structure,
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okay? Can get inflamed, particularly with patients that like to bend the knee.
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So a lot of, we get this in a lot of soccer players, okay?
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So peri cruciate, fat pad edema. All right? So that's my, uh,
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so just to backtrack real quick, so menisci cruciate ligaments,
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medium lateral poring structures, extensor mechanism, fat pads.
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Now the joint, I look at the joint, I look at the alignment, okay? Granted,
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it is a static image, okay? We're not gonna, you know,
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we're not doing the physical exam, but sometimes, especially with a, uh,
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anterior cruciate ligament tears,
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we're obviously gonna see that anterior translation of the tib with respect to
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the femur, with uncovering of the menisci. Right? Next I check the, uh,
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cartilage, starting with the patella femoral compartment, then using, uh,
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and I evaluate that with axials and sagittals, that compartment.
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And for the coronal, uh, sorry for the, um, for the, uh,
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medial and lateral femoral tubal compartments, I like to obviously use the, um,
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the sagittals and coronals. I will, however, use the axials,
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especially for these far posterior, uh, con or lesions, you know, um,
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sometimes, you know, we miss a little far, uh, posterior lesion.
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And some of my, um, some orthopedic literature will say, you know,
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if we don't mention it, they will miss it,
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because they won't get the scope all the ways back there,
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or entirely flex the knee to try to get back there. Okay? Next,
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along with the joint, I look for the Popal cyst, and I harp,
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I harp on my trainees here at UCSD to make sure that the neck comes out between
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the m and m muscles, that is the medial gastroc anemia, and the semimembranosus,
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uh, muscle or tendon. And that is the direct arm here, okay?
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Depending on who you read, there are actually about six or seven attachments,
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okay? Or arms of these emin ss tendon. But here, for this purposes,
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I'm talking about the direct arm, which inserts upon the, uh,
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posterior aspect of the, uh, uh,
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medial tibial, uh, pip proximal, medial tibial pips right here. So this as,
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in this case,
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this is where you want to see the neck of the Pope cyst arising from,
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from the medial gastroc anus,
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from in between the medial gastro anus and the semimembranosus muscle,
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or the m and m muscles, kind of like the candy. Okay?
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If you don't see that neck there, then you gotta worry about something else. Um,
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uses neoplasm, a ganglion cyst, what have you, something like that. Okay?
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Something more, potentially more ominous,
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especially with the mus neoplasms or potentially, let's say a synovial sarcoma,
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right? All right. So finally,
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the last few checks I have are for any incidentals.
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I look for any muscle anatomy, uh, variance,
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particularly third head of the gastric anemia muscles,
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which typically will arise right here, can, can, uh, uh, uh,
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lead to popliteal artery entrapment syndrome if you believe in that.
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Other things I look for are, uh, accessory plant terrace,
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which is thought to contribute potentially to it band prien syndrome.
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And another thing I also look for, especially if someone's going for, let's say,
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a high tib osteotomy, or they have really overt degenerative changes,
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I look for an abberant, anterior TBIs artery, okay?
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Which is depending on who you read, seen in, in the knee,
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about 2.1% of the time, that's according to an article from the,
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the American Journal of Sports Medicine.
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And basically it's a little early takeoff of the angio tibialis artery that
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interposes itself between the posterior aspect of the tibia and the populous
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muscle right here. So you would see it right here.
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Why is that important to mention?
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I've actually been involved in a couple of cases right now, not me personally,
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but testifying that, uh, the, that artery was missed.
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Surgeon went in, it was Nick,
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and the patient later went on to bleed out and got compartment syndrome.
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And obviously the, the problems with that. So if you do see, uh,
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an anterior an an aberrant anterior anterior TBIs artery, I would,
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I would highly suggest, uh, uh, mentioning it in your, uh,
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in your findings at least, if not in the impression, especially,
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especially if the patient is planning to go for, you know, uh, surgery, um,
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a high tibial osteotomy, knee replacement, what have you.
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So something to be aware of. And finally, lastly, I like to look at the,
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uh, bones, uh, bone, uh, bone morphology and marrow signal,
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and obviously I like a nice T one for that. Okay, so going back to our case.
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Okay. Uh, sorry,
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any questions on how I kind of approach a knee before we jump into the
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actual case or the first case?
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Well, you, you talk about the complexity of structures,
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especially in the postal lateral corner in the EM prognosis,
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having multiple attachments.
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Is it crucial to identify each of these attachments and structures,
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these small structures?
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I would say not, not really, sir. Uh, great question. I, I,
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I think that was coming from Hari. Great question. Uh,
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I'm happy to provide that, uh,
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literature for literature to you if you're interested. But really, I would say,
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uh, in my experience, the big things are going to be identifying the, the, uh,
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the, uh, direct head. Okay? This one right here, okay.
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Or sorry, the direct arm of the semiosis, which obvious, uh, oftentimes,
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uh, gets, uh, degenerated tendon otic or partially torn.
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And I would say the other arm that I would, uh,
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like to know about is the, uh, anterior arm,
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which is this arm right here,
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which inserts upon the lateral aspect of the proximal tibial epiphysis. Okay?
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And if you view it on a sagittal, it's actually been likened to, uh,
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a hockey stick. Okay? And why is that important? This,
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this arm is important. It's right here, okay? And going back to our coronal,
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it's right sort of, uh, here, this sort of thin structure right here,
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it, it can become tendon otic, and sometimes people will come in,
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say, uh, with a rule out mass, and it just turns out to be a really tendon otic,
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uh, long, uh, sorry, anterior arm of the semimembranosus tendon. And,
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uh, it is been confused for, um, a mass, for instance, like, uh, you know,
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tens synovial, giant cell tumor, focal type intraarticular,
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or even like gout or something like that. So those,
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those are in my experience, are probably the two more important arms to,
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to think about for the, uh, the posterior medial corner here.
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So it's interesting to, to,
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to find out that it's actually 11 bundles within the ACL.
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Yeah, yeah, yeah. There's, there's up to 11 or 13 now and stuff like that. And,
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and as you'll see during my office hours, and I'll point out the, the more,
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the more I study, at least the more I find out I, I, I, I have more to learn.
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So, but that's why, that's why we do this, right?
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And the
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Anterior posterior bundles of the, um, ACL,
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how do you identify that?
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Uh, just where they insert, right? So the anter medial is just,
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is just gonna be more anterior and medially located,
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and the posterior lateral is just gonna be post, uh, insert, uh,
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sort of post more posterior and lateral. And if you think about it, right,
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the cruciate ligaments and pretty much everything, even the meniscal,
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meniscal ligaments right,
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are named for its insertion upon the tibia more anteriorly,
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if you want to think of it that way. Okay?
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So going right to the case. Okay? So,
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so now that I've shown you how I kind of approach a case, that being said,
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okay, besides looking at the localizer and the, uh,
18:08
my sequence or structural analysis,
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I do initially open up every study, um,
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looking at, uh, usually AT one and a fluid sensitive sequence. Okay?
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And that just gives me a flavor of the case, okay?
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Just where to direct my eyes and where I,
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I figure I'm gonna direct most of my intention, okay? And in this case,
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in our 72 year old male with problems squatting, what we see here, okay,
18:35
we will go from proximal to distal, okay? The quadriceps tendon, uh,
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some of you may have called, um, some tendonosis, that's fine,
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maybe even a little bit tearing.
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But what I wanna highlight is the quadriceps can look quite striated,
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sort of almost like a step celery stock that we see with the ACL. Okay?
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I like to see a lot of edema. I like to hear that,
18:56
that history of anterior knee pain if possible too as well. Okay?
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But continuing distally part of the extensor mechanism or the heart of the
19:04
extensor me mechanism, what we see here is a really emus okay,
19:08
or patella with osteo, right?
19:11
Without any good history and any other good, uh, other findings to corroborate.
19:16
Yeah, you can think of contusion if there's an ulcer, you know, you could,
19:19
you could start thinking about osteomyelitis,
19:22
septic arthritis with pre patellar bursitis, what have you.
19:25
But going more distally, we've noticed that the quadr, uh,
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pre patellar quadriceps, uh,
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continuation or what some of you have termed the, the pre patellar plate,
19:36
okay? We, uh, know this, okay,
19:38
as basically a chondro,
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a pope zone, okay? And I kind of think of it like,
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um, I love sweets, okay? And I love baklava. Okay?
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I don't know if you guys have baklava before or like, uh,
19:53
croissants or fine pastries. It's, it's multiple,
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it's actually multiple layers and bursa overlying this, uh,
20:01
anterior patella, and I always have to look it up.
20:04
There's like a subcutaneous bursa, a subfascial bursa,
20:08
and a three or four more, more bursa. But you can look it up in the literature.
20:13
But what can happen is with injuries, this is, this can actually tear,
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or as in our case, okay,
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we have a little bit of enthesopathy perhaps from the tugging,
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the pre patella quadriceps.
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This entire plate is sort of dispersing or helping to disperse the force
20:29
of that quadriceps and patella tendon across the Accenture mechanism, right?
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Think of this patella as basically a pulley, right?
20:36
Providing more mechanical advantage to our extensor mechanism,
20:41
allowing us readily ready or easier extension, okay?
20:45
Between the, uh, of the lower extremity, obviously. But in this case,
20:49
this patient obviously has osteitis or reactive edema,
20:53
some enthesopathy, some stripping,
20:55
and some arguably low grade or some partial tearing of that pre patellar plate
21:00
or the quadriceps continuation. And then more distally,
21:03
it cuts into the sort of interstitial deamination delaminating sort of
21:08
tear of the proximal teller tendon superimposed upon, um,
21:12
tendinopathy. I used to grade tendinopathy and sort of,
21:16
sort of differentiate tendinopathy, but after talking to a lot of my surgeons,
21:20
they don't mind if I don't, uh, sort of, uh,
21:25
better classify or come down hard on the typical, the,
21:28
the exact type of tendinopathy you may hear terms of such,
21:31
such as hypertrophic, uh, degenerated, fatty,
21:35
replaced calcific ossific, you know, sometimes even, you know,
21:39
you can have crystal deposition or fatty deposition in, in some tendons,
21:43
what have you. Um, but I just say tendinosis. Some, some of my surgeons, yes,
21:48
they like measurements. So in this case, I would maybe measure the,
21:53
not only the AP thickness,
21:55
but the cranial coddle thickness as well and just really try to be specific.
21:59
But this does fall in the realm, okay, of, uh,
22:03
arguably attraction apophysis. Okay? And someone older, if, sorry,
22:08
if it was a very young patient, uh, uh, a,
22:11
a little kiddo where the patellar, uh, um,
22:14
the epiphyseal cartilage is not yet mature,
22:16
you get the patellar sleeve evulsion fractures, okay?
22:20
But as they get older, and at the proximal uh, end,
22:24
we're gonna worry about jumper's knee,
22:26
but basically you can think of them as traction of pope or traction sort of
22:30
injuries across the Accenture mechanism. If you have it happening more distally,
22:35
then that's gonna be that classic SLJ or syn larsson johanson, uh,
22:40
syndrome for those that like eponyms, okay? And,
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and obviously a little bit younger, you're gonna have, especially when you have,
22:46
uh, typical tubercle edema and uh,
22:49
oif osci fragmentation of the tibial tubercle,
22:52
then that's gonna fall in the realm of os Oscar slaughter's disease.
22:55
But I sort of think of these, uh, injuries as a continuum,
22:59
much older patients, especially if they have bilateral disease.
23:03
I'm gonna start thinking of systemic disease processes, right?
23:06
Whether it is renal failure, diabetes, what have you, uh, maybe, you know,
23:12
rare medications, but as we know, medications,
23:14
especially fluoroquinolones has typically been described with your achilles
23:18
tendon tears. But that, uh,
23:19
and that we may see that in another course if you guys are with us. Okay?
23:24
So this just a, just a nice case within the realm of jumper's knee,
23:28
but also some nice, uh, tearing or stripping of the, uh, uh,
23:32
patellar quadriceps continuation. Okay?
23:35
And you can see that obviously too nicely on our axial sequences.
23:40
Again, coming from proximal to distal, we see some of that quadriceps, uh,
23:45
tendinosis, maybe even little foci of tearing out medially there,
23:50
going out, uh, more distally, we see that abnormality,
23:53
that enthesopathy and osteitis underneath that patella's quadriceps
23:57
continuation,
23:58
we see some of that uplifting of the three or four layers of the bursa anterior
24:03
to the patella bone and more distally, we see that, uh,
24:06
partial mainly intrasubstance hearing of the patellar tendon. Okay.
24:12
Questions on this case.
24:15
Is this commonly seen in racket spots, uh, particularly with tennis players?
24:20
Uh, I, I have seen it. I have, yeah, I have seen it. But here in, uh,
24:25
San Diego, I typically see it more with, uh,
24:28
basketball players and volleyball players.
24:31
So what are the treatment adjuncts for that?
24:34
You know, I i, I don't know the, the, uh,
24:38
treatment adjuncts for that. Um, but um,
24:44
I think I,
24:46
I have been asked at times to like inject, um, you know,
24:51
steroids if, you know,
24:52
to see if that's like a pain generator or lidocaine at times under ultrasound.
24:57
But more often than not, I, I would say, um,
25:03
I would say definitely repaired when the rest of the, uh,
25:07
other components of the extensor mechanism are involved.
25:10
But I have not in my, uh,
25:13
experience or anecdotally seen a surgeon go in just to fix that, uh,
25:18
quadriceps continuation.
25:21
And certainly there's some controversy with, uh,
25:23
corticosteroid injections because they tend to cause weakening and possibly even
25:28
tears in the future.
25:29
Yeah, I, I think I, and, and depending on who you read too, um,
25:36
a long term, and there's a, there's a couple recent articles.
25:41
Longer term,
25:42
if you follow some patients out tendon tears at the hips and or
25:47
tendonosis and tendinopathy, long-term, um,
25:51
what they found is long-term steroids may be detrimental.
25:57
'cause it does, as you had mentioned,
25:59
lead ultimately to tears and what have you.
26:02
And it sort of accelerates the process, some of my colleagues and,
26:07
and listening to authors at various, um, meetings.
26:11
Some people believe that, um, you know, we are just treating friction with,
26:16
you know, saline, lidocaine or steroids. But, um,
26:20
I have not heard of a good, uh, prospective study. Uh,
26:25
I, I, that would be a nice thing to do. Just, you know,
26:28
in do a sham injection where someone's getting, um,
26:32
no injection, uh, maybe lidocaine, um,
26:37
uh, maybe lidocaine saline or just saline injections and then, you know,
26:42
steroid injections and just see longer term what, uh, um,
26:47
how these patients do in the various, um, uh, you know,
26:51
in with these various injections in, in various locations. I, I'd,
26:55
I'd really be interested to, to see that. Yeah.