Interactive Transcript
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This was a 72 year old male with knee pain, no injury.
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Starting from the medial side. We have soft tissue inflammation.
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The medial meniscus is looking okay. I didn't see a definite tear. 72 year old.
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Uh, the articular cartilage, um, doesn't look healthy. If you see there, there,
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there is some abnormal signal in the cartilage. So this,
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this will be areas of gait. Grade two to three cartilage loss over here.
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Looks like it's almost full thickness cartilage loss. Over here,
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there is superficial cartilage freeing over here. Also,
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there is superficial cartilage freeing and some loss.
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So it is of grade two to three cartilage loss in the medial compartment.
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Your cruciate are normal,
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but you can see that gray signal in the interoral notch.
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So there is some synovitis in the joint
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coming onto the lateral side against lateral meniscus,
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no big tear. The, there is like free edge, uh, frank, uh,
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and then abnormal signal in the anterior hand of the lateral meniscus.
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So abnormal signal in the anterior horn of the lateral meniscus can often, uh,
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be result of, um,
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degenerative changes in the ACL because both the tibial insertion of ACL and the
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anterior hand of, um, lateral meniscus, like those fibers intermingle.
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So there's ACL degeneration that signal, um,
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or the degenerative changes involve the anterior one of lateral meniscus as
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well. So here I don't see a lot of, um, um, changes in the ACL.
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So this might be like a small degenerative tear of the anterior,
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one of the lateral meniscus. Um, then we have joint effusion,
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and what we see is a lot of abnormality in the extensor, uh,
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component of the knee joint. So the quadriceps tendon is thickened,
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and if you see there's abnormal soft tissue that's, uh,
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interspersed in between the fibers. We have, um,
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pre particular soft tissue single abnormality as if there's like intermediate
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signal soft tissue deposit here. And similarly, even the particular tendon,
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uh, is thickened shows abnormal intermediate signal as if, um,
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there's some infiltration of the particular tendon.
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And we have this, uh, can make out what is it on sattal images. So we can, uh,
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look at the axial images. And as I was saying about that,
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v this is your nice fibular collateral ligament seen on the sagittal images.
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And this is the biceps tendon.
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So that's your posterolateral complex structure.
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Both insert on the tip of the fibula,
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starting strong pen. Again, we see that quadricep tendon abnormalities.
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If you see there's something external, uh,
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on the surface of the quadricep tendon that's infiltrating the fibers,
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anteriorly joint effusion,
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soft tissue edema,
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then you have signal abnormality in the ular tendon. And, uh,
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remember we saw something on the lateral side. So that's the abnormality along.
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Um, this is where the, the, the popliteus notches.
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And if you see this is the fibular, uh,
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fibular collateral ligament attachment on the lateral femoral condyle. So again,
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there is like an erosion, um, along the lateral femoral condyle with, again,
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that soft tissue, which is intermediate signal. So, um,
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this, uh, is actually a classic. It's a really nice case, a classic imaging, uh,
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appearance of gout in knee. Um,
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the demographics are perfect and early males have very high chances of, uh,
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especially if they're, um, um,
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if high protein and take their hyperuricemia from uh, uh, whatever reason. Uh,
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if they have, um,
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like elderly males are more predisposed to have gout, uh,
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specifically if they have, uh, hyperuricemia things like leukemia, um,
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renal disease, uh, where, um, or inborn errors of metabolism where the,
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the urate levels are high. And you, um, uh,
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what is gout is deposition of monos sodium urate crystals. Um,
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and a gout classically gives rise to, um,
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juxta articular marginal erosions. And, uh,
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another classic manifestation of gout is deposition of tophus. So this,
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um, deposition of crystals, um, is usually the per articular location.
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Um, and they give rise to these intermediate signal intensity masses around the
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joint that can eventually cause, uh, bone erosion.
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So that's your example of a tophus deposition.
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You have this intermediate signal,
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intensity soft tissue resulting in bone erosion. And then sometimes these, um,
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so your classic location is the first MTP joint of the, the foot. Again,
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you'll have nice, um, um,
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just strat erosions with overhanging margins because the articular surfaces, uh,
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um, get affected late in the, uh, disease process. The articular cut, uh,
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uh, cortex remains intact for a long time.
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So the joint space narrowing sets in really late.
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But what you get is a ni nice punch out erosion in the, uh,
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juxta articular, um, cortex with, uh,
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an MR is excellent in demonstrating these tophi. So on on, so, uh,
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radiographs you see dense soft tissue overlying these erosions and an mri.
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You can see this intermediate signal, intensity soft tissue, and,
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um, uh, deposits in, in soft tissues as well. And your pre particular,
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um,
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areas is a classic location for gout tophus deposition.
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So if you see an intermediate signal soft tissue along the superior pole
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anterior aspect of the particular in an elderly male, uh, that's, that's a,
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um, a very classic presentation of gout. Another, uh,
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great location for gout is, uh,
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the reon bursa of the elbow. So if you have, uh,
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reon a person who presents with reon bursitis, but that bursa has dense, uh,
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appearance on, on radio grabs.
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And if you do Mr and if you see similar looking intermediate signal,
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intensity soft tissue in the lone bursa, um,
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that's another classic manifestation of gout. Uh,
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and then it can eventually cause erosions here. So again,
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erosion along the anterior superior patella is, uh,
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if they short on radiographs,
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it's some soft tissue intensity overlaying it that suggests gout. Um,
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even an elbow, uh, it can result in erosions, um, shoulder.
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You get that hatchet deformity, uh, when you have AS lateral erosion in the,
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in the shoulder. So, yeah, uh,
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so this, this will be gout where we have the tophus deposition, uh, um,
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in the distal, uh,
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soft tissue infiltrating the distal quadriceps tendon tous overlying the
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anterior superior pole of the patella, um, thickening of the patula tendon,
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and you have a nice erosion along the lateral femoral condyle close to theus
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tendon insertion with small tous deposition.
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I, I found this case very challenging. I I missed the diagnosis. Mm-Hmm mm-Hmm.
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So, mm-hmm. With all the changes within the soft tissue structures,
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I was confused whether this is actually chronic trauma and, uh,
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tendinosis rather than, um, a crystal deposition disease. So,
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um, what's the actual, uh,
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important point to pick up when you see these cases as, uh,
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as the most major finding?
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Um, uh, I mean it's the, yeah. I mean,
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the thing with gout is, uh, we always, uh, read about the,
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the classic like involvement of the first meta of fedal joint, but we, we don't,
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uh, get to see, uh, more often in teaching, um,
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is these kind of, uh, manifestations of gout. Um, and this is a, when you, um,
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read, uh, a book on, um, arthritis or, um, um,
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a fellow level, uh, reading and, and then MSK imaging,
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you'll realize that this is a, it's, it's,
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it's a classic presentation of gout where we have this tofus deposition. So, uh,
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trauma should not, like, can result in tendon changes,
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but should not give rise to this, uh, soft tissue, right? If there's, there's a,
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there's a nice soft tissue, intermediate signal,
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or almost likely to dark signal, uh, soft tissue, um, tissue that is getting,
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uh, deposited along the anterior aspect of the patula.
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So, um, that, and knowing, uh, that, I mean, this is a,
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a known, uh, presentation of gout and just knowing the, the other locations,
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uh, classic locations for gout, I think that, that, that will help in, uh,
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making the diagnosis is
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That involvement in this case also,
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it looks a very ground glass and thickened is, is that another thing that could
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Possibly, sorry, what appearance of what?
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The cartilage.
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Cartilage, okay. Let's see. So cartilage and, uh, this patient is,
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I think he's 72. It's, it's not a very healthy looking cartilage. There are, uh,
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areas of grade two to three cartilage loss. Um,
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and then if you see there are these other dark fo so this is, uh,
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I sometimes when there is old cartilage injury, um, uh,
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that gray appearance is for normal highline cartilage.
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And when there's cartilage injury, sometimes that can heal on itself, but, um,
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it'll never give you back your highline cartilage. Uh,
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the highline cartilage gets replaced with fibrocartilage.
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So those T two duct areas are either fibrocartilage, um, and, and sometimes, um,
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there's crystal deposition on the articular cartilage surface itself.
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So all these things are making the cartilage look, yeah, definitely abnormal.
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This is not healthy cartilage that you've seen in younger individuals with no
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cartilage injury.
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Uh, on sagittal proton density, uh, sequences.
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The cartilage looks a bit thickened on the sal proton density
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pd, uh, non non-fat, just the pd,
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No. Here,
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I think you are losing the contrast resolution between the articular cartilage
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and the cartilage itself. So the articular cortex and the cartilage.
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So you're seeing both combined as this high point and signal.
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I think it's just to do with the, I don't, uh,
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I'm not seeing the TETR time on this. It has to do with the, the settings,
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like the parameters of the sequence. Uh, but this, this is a good sequence, uh,
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at, at least in this, uh, study, uh,
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that tells you the articular cart and it doesn't look thickened.