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Wk 3, Case 4, Knee MR - Review

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This was a 72 year old male with knee pain, no injury.

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Starting from the medial side. We have soft tissue inflammation.

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The medial meniscus is looking okay. I didn't see a definite tear. 72 year old.

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Uh, the articular cartilage, um, doesn't look healthy. If you see there, there,

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there is some abnormal signal in the cartilage. So this,

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this will be areas of gait. Grade two to three cartilage loss over here.

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Looks like it's almost full thickness cartilage loss. Over here,

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there is superficial cartilage freeing over here. Also,

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there is superficial cartilage freeing and some loss.

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So it is of grade two to three cartilage loss in the medial compartment.

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Your cruciate are normal,

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but you can see that gray signal in the interoral notch.

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So there is some synovitis in the joint

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coming onto the lateral side against lateral meniscus,

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no big tear. The, there is like free edge, uh, frank, uh,

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and then abnormal signal in the anterior hand of the lateral meniscus.

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So abnormal signal in the anterior horn of the lateral meniscus can often, uh,

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be result of, um,

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degenerative changes in the ACL because both the tibial insertion of ACL and the

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anterior hand of, um, lateral meniscus, like those fibers intermingle.

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So there's ACL degeneration that signal, um,

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or the degenerative changes involve the anterior one of lateral meniscus as

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well. So here I don't see a lot of, um, um, changes in the ACL.

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So this might be like a small degenerative tear of the anterior,

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one of the lateral meniscus. Um, then we have joint effusion,

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and what we see is a lot of abnormality in the extensor, uh,

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component of the knee joint. So the quadriceps tendon is thickened,

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and if you see there's abnormal soft tissue that's, uh,

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interspersed in between the fibers. We have, um,

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pre particular soft tissue single abnormality as if there's like intermediate

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signal soft tissue deposit here. And similarly, even the particular tendon,

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uh, is thickened shows abnormal intermediate signal as if, um,

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there's some infiltration of the particular tendon.

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And we have this, uh, can make out what is it on sattal images. So we can, uh,

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look at the axial images. And as I was saying about that,

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v this is your nice fibular collateral ligament seen on the sagittal images.

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And this is the biceps tendon.

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So that's your posterolateral complex structure.

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Both insert on the tip of the fibula,

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starting strong pen. Again, we see that quadricep tendon abnormalities.

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If you see there's something external, uh,

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on the surface of the quadricep tendon that's infiltrating the fibers,

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anteriorly joint effusion,

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soft tissue edema,

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then you have signal abnormality in the ular tendon. And, uh,

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remember we saw something on the lateral side. So that's the abnormality along.

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Um, this is where the, the, the popliteus notches.

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And if you see this is the fibular, uh,

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fibular collateral ligament attachment on the lateral femoral condyle. So again,

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there is like an erosion, um, along the lateral femoral condyle with, again,

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that soft tissue, which is intermediate signal. So, um,

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this, uh, is actually a classic. It's a really nice case, a classic imaging, uh,

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appearance of gout in knee. Um,

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the demographics are perfect and early males have very high chances of, uh,

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especially if they're, um, um,

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if high protein and take their hyperuricemia from uh, uh, whatever reason. Uh,

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if they have, um,

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like elderly males are more predisposed to have gout, uh,

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specifically if they have, uh, hyperuricemia things like leukemia, um,

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renal disease, uh, where, um, or inborn errors of metabolism where the,

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the urate levels are high. And you, um, uh,

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what is gout is deposition of monos sodium urate crystals. Um,

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and a gout classically gives rise to, um,

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juxta articular marginal erosions. And, uh,

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another classic manifestation of gout is deposition of tophus. So this,

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um, deposition of crystals, um, is usually the per articular location.

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Um, and they give rise to these intermediate signal intensity masses around the

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joint that can eventually cause, uh, bone erosion.

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So that's your example of a tophus deposition.

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You have this intermediate signal,

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intensity soft tissue resulting in bone erosion. And then sometimes these, um,

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so your classic location is the first MTP joint of the, the foot. Again,

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you'll have nice, um, um,

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just strat erosions with overhanging margins because the articular surfaces, uh,

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um, get affected late in the, uh, disease process. The articular cut, uh,

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uh, cortex remains intact for a long time.

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So the joint space narrowing sets in really late.

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But what you get is a ni nice punch out erosion in the, uh,

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juxta articular, um, cortex with, uh,

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an MR is excellent in demonstrating these tophi. So on on, so, uh,

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radiographs you see dense soft tissue overlying these erosions and an mri.

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You can see this intermediate signal, intensity soft tissue, and,

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um, uh, deposits in, in soft tissues as well. And your pre particular,

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um,

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areas is a classic location for gout tophus deposition.

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So if you see an intermediate signal soft tissue along the superior pole

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anterior aspect of the particular in an elderly male, uh, that's, that's a,

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um, a very classic presentation of gout. Another, uh,

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great location for gout is, uh,

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the reon bursa of the elbow. So if you have, uh,

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reon a person who presents with reon bursitis, but that bursa has dense, uh,

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appearance on, on radio grabs.

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And if you do Mr and if you see similar looking intermediate signal,

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intensity soft tissue in the lone bursa, um,

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that's another classic manifestation of gout. Uh,

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and then it can eventually cause erosions here. So again,

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erosion along the anterior superior patella is, uh,

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if they short on radiographs,

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it's some soft tissue intensity overlaying it that suggests gout. Um,

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even an elbow, uh, it can result in erosions, um, shoulder.

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You get that hatchet deformity, uh, when you have AS lateral erosion in the,

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in the shoulder. So, yeah, uh,

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so this, this will be gout where we have the tophus deposition, uh, um,

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in the distal, uh,

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soft tissue infiltrating the distal quadriceps tendon tous overlying the

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anterior superior pole of the patella, um, thickening of the patula tendon,

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and you have a nice erosion along the lateral femoral condyle close to theus

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tendon insertion with small tous deposition.

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I, I found this case very challenging. I I missed the diagnosis. Mm-Hmm mm-Hmm.

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So, mm-hmm. With all the changes within the soft tissue structures,

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I was confused whether this is actually chronic trauma and, uh,

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tendinosis rather than, um, a crystal deposition disease. So,

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um, what's the actual, uh,

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important point to pick up when you see these cases as, uh,

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as the most major finding?

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Um, uh, I mean it's the, yeah. I mean,

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the thing with gout is, uh, we always, uh, read about the,

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the classic like involvement of the first meta of fedal joint, but we, we don't,

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uh, get to see, uh, more often in teaching, um,

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is these kind of, uh, manifestations of gout. Um, and this is a, when you, um,

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read, uh, a book on, um, arthritis or, um, um,

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a fellow level, uh, reading and, and then MSK imaging,

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you'll realize that this is a, it's, it's,

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it's a classic presentation of gout where we have this tofus deposition. So, uh,

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trauma should not, like, can result in tendon changes,

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but should not give rise to this, uh, soft tissue, right? If there's, there's a,

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there's a nice soft tissue, intermediate signal,

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or almost likely to dark signal, uh, soft tissue, um, tissue that is getting,

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uh, deposited along the anterior aspect of the patula.

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So, um, that, and knowing, uh, that, I mean, this is a,

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a known, uh, presentation of gout and just knowing the, the other locations,

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uh, classic locations for gout, I think that, that, that will help in, uh,

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making the diagnosis is

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That involvement in this case also,

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it looks a very ground glass and thickened is, is that another thing that could

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Possibly, sorry, what appearance of what?

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The cartilage.

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Cartilage, okay. Let's see. So cartilage and, uh, this patient is,

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I think he's 72. It's, it's not a very healthy looking cartilage. There are, uh,

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areas of grade two to three cartilage loss. Um,

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and then if you see there are these other dark fo so this is, uh,

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I sometimes when there is old cartilage injury, um, uh,

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that gray appearance is for normal highline cartilage.

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And when there's cartilage injury, sometimes that can heal on itself, but, um,

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it'll never give you back your highline cartilage. Uh,

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the highline cartilage gets replaced with fibrocartilage.

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So those T two duct areas are either fibrocartilage, um, and, and sometimes, um,

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there's crystal deposition on the articular cartilage surface itself.

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So all these things are making the cartilage look, yeah, definitely abnormal.

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This is not healthy cartilage that you've seen in younger individuals with no

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cartilage injury.

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Uh, on sagittal proton density, uh, sequences.

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The cartilage looks a bit thickened on the sal proton density

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pd, uh, non non-fat, just the pd,

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No. Here,

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I think you are losing the contrast resolution between the articular cartilage

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and the cartilage itself. So the articular cortex and the cartilage.

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So you're seeing both combined as this high point and signal.

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I think it's just to do with the, I don't, uh,

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I'm not seeing the TETR time on this. It has to do with the, the settings,

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like the parameters of the sequence. Uh, but this, this is a good sequence, uh,

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at, at least in this, uh, study, uh,

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that tells you the articular cart and it doesn't look thickened.

Report

Patient History

72-year-old male with extreme posterior knee pain with no known injury

Findings

Cruciate Ligaments: ACL and PCL with mild mucoid degeneration. Otherwise, intact.

Central Compartment: Diffuse synovitis with tibial eminence spurring and multifocal chondral erosions with subchondral arthropathic cysts along the notch floor.

Medial Tibiofemoral Compartment: Class 2-3 chondromalacia. No osteochondral defects. No osteoarthrosis. Meniscal fraying without meniscal tears. Intact medial collateral ligament. Mild swelling along the deep crural fascia with nominal tibial collateral bursitis.

Lateral Tibiofemoral Compartment: Class 2-3 chondromalacia. No osteochondral defects. Complex macerated tear of the anterior meniscal horn which is partially extruded into the gutter and a thin and delicate radial/flap undersurface tear of the posterior horn and body.

Proximal Tibiofibular Joint: Mild osteoarthrosis with penetrating chondral fissure and subchondral arthropathic cyst formation.

Posteromedial Corner: Mild to moderate insertional tendinosis of the semimembranosus tendon.

Posterolateral Corner: Severe infiltrative tendinosis at its insertion on the popliteal hiatus with a punched-out cortical erosion and synovitis. The fibular collateral ligament, biceps femoris, arcuate ligament and popliteofibular ligament are intact.

Extensor Mechanism: Severe diffuse hypertrophic infiltrative tendinosis with peritendinitis and interstitial splitting of the mid to distal quadriceps tendon and the entire patellar tendon.

Flexor Mechanism: Nominal fluid distention of the gastrocnemius and semimembranosus bursa without dehiscence. The neurovascular bundle is unremarkable.

Traction enthesophyte with cortical erosion of the inferior patellar vertical ridge.

Patellar plate delamination with severe prepatellar bursitis and diffuse anterior soft tissue swelling.

Small to moderate joint effusion with reactive synovitis. No conspicuous internal debris or free bodies.

Diffuse periarticular soft tissue swelling.

Impressions

1. Crystal deposition arthropathy of the right knee (gout).

2. Marked severe infiltrative hypertrophic tendinosis with peritendinitis and interstitial splitting of the mid to distal quadriceps tendon and the entire patellar tendon.

3. Patellar plate delamination with severe prepatellar bursitis.

4. Severe infiltrative tendinosis of the popliteus with a punched-out cortical erosion and synovitis at its insertion on the popliteal hiatus.

5. Small to moderate joint effusion with reactive synovitis.

6. Diffuse periarticular soft tissue swelling.

7. Generalized lateral femorotibial class 2-3 chondromalacia.

Case Discussion

Faculty

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Gitanjali Bajaj, MD

Assistant Professor

University of Arkansas for Medical Sciences

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Brian Y. Chan, MD

Assistant Professor of Musculoskeletal Radiology

University of Utah

Todd D. Greenberg, MD

Radiologist

ProScan

Tags

Musculoskeletal (MSK)

MRI

Knee