Interactive Transcript
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And, um, my first case is a youngster.
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Um, he's 16 years old
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and his chief complaint was, he is a pitcher, um,
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diminished range of motion.
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And let's start out with the axial gradient echo.
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And I'm a big fan of the gradient echo to evaluate the labra
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with without contrast.
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And anteriorly, he does have these small
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minuscule little cracks in the anterior labrum,
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and it's not uncommon to see anterior labral signal in, uh,
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vigorous rigorous athletes.
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And in, in adults sometimes you can even get CPPD in there.
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You know, incidentally, that
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that produces some irregularity and signal intensity.
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Um, there isn't much swelling around this,
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and it's, it's not in the typical location
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where you would worry about a label tear.
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It's sitting right about at the equator.
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He's also got this pseudocyst in the back.
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And on day one, I did emphasize that I use as a roadmap,
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these patterns of pseudocysts to help determine
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where the biomechanics have gone wrong,
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especially in younger individuals
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who don't have necessarily obvious static morphologic
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abnormalities, but rather biomechanical ones.
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And I can infer the biomechanical problem from the cyst.
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This one's all the way in the back.
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And if we look all the way in the back
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and we scroll down on our calcium sensitive,
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blood sensitive gradient echo image,
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we see this very curious looking longitudinal dark snake
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behind the, the scapula.
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Now, uh, this is a Bennet like lesion,
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and the reason I say Bennet like is in my experience,
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80% of the Ben lesions, uh, are
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connected intimately with the bone.
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Uh, the bone appears heaped up
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and proliferative, whereas about 20% of them,
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maybe a little more, uh, you can see a cleavage plane
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between the lesion and the bone.
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And this is a person that has the right symptoms.
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And other than this cleavage p plane has the right shape.
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This, this was calcific on the ct, which I don't have
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to show, but we did get one.
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And you see how long it is from top to bottom.
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It's very long as ben lesions are, are apt to do.
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And I, I think about these as either capsular
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or periosteal hemorrhages
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that ossify when they have this plane.
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So I think there's really a, a heterogeneous group
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of Bennett like lesions.
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And when we look at the posterior capsule in this young man,
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it's a thick capsule as it should be in a thrower.
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But I think this capsular thickening along
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with the Bennett lesion, certainly with the Bennett lesion,
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and the capsule blends with the periosteum, uh, and,
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and perhaps contracts back here
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and shortens, contributes to this, uh,
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glenoid internal rotation deficit of this child.
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Now, I, I left this image upright here
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because, not just for the cyst,
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but if you look down below at the site
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where this Bennet like lesion exists, there's a fair amount
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of swelling, uh, present there.
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Um, he, he didn't complain of pain, by the way.
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His, his chief complaint was diminished range of motion.
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Any comments about this one, Don? Yeah,
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I'm just wonder, uh, whether, uh, uh,
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you say you've seen this particular kind of appearance.
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Have you followed them?
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Do they begin separate
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or could they start connected to the bone?
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Uh, or is there any possibility these are fractures
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that have developed from the posterior glenoid?
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It'd be nice to kind of trace the history of them over time.
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I think that's a great thought.
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If you look at the sagittal though, I,
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I do see a pretty good cortex here on the T one.
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And I have two cases where I've studied young individuals,
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11, 12, 13 year olds who complained of pain.
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They have edema on the proton density,
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fat suppression image.
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And then they come back three or four years later
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and they have a benit lesion.
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So I do think that these are acquired
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and they, they develop, you know, a,
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a little bit like a Nora lesion, you know,
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a hemorrhage along the edge
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of the bone, and then they ossify.
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Okay.