Interactive Transcript
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We now move on and we'll discuss the tendon pattern,
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the tendon patterns of impingement.
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Now these are generally divided into two types, external,
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something perhaps outside of the cuff itself
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with two subtypes that are emphasized, subacromial
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and subor, and then internal.
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And there are several varieties now of internal.
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The two that are most commonly described are posterosuperior
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and anterosuperior.
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I'm gonna discuss three of these four patterns
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of impingement.
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So let's talk about external impingement.
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The general abnormality that is agreed upon
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by most people about this pattern of impingement
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is narrowing of the subacromial subcoracoid space.
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But it is controversial as to what causes that narrow
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with two theories.
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One theory suggests that it's an intrinsic problem
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of the tendons of the rotator cuff
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that produce the initial abnormalities, degeneration,
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and tearing of the tendons, weakening of those muscles
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because of those tendons
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and an elevation of the humeral head
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with secondary narrowing of the subacromial
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sub corticoid space.
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The alternative theory says, no, no, wait a minute.
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That's not what's going on.
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The initial abnormalities are abnormalities
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of the corco acromial arch
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and those abnormalities lead to narrowing
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of the subacromial subc corticoid space.
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And because of that narrowing,
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and I'll show you exactly why there are abnormalities
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that develop within the subj tendons, perhaps beginning
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as degeneration, but ending up as partial
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or full thickness tearing of those tendons.
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So that is an extrinsic cause, not an intrinsic cause.
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I strongly believe that shoulder impingement is a clinical
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diagnosis, not an imaging diagnosis.
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And I would argue that there may be no foolproof signs.
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One fairly reliable sign that the symptoms
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of impingement are present.
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Now the orthopedic surgeon is armed with a variety of tests
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and they're good near test and the Hawkins test,
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and there are others that are, are listed in the literature.
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Okay? So there are a number of tests.
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Generally they relate to painful elevation
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of the arm in certain positions of that arm.
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So that is the way the diagnosis is made.
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So I really believe it is not our diagnosis to make
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we provide evidence for that diagnosis.
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Now, if you are a believer in the external theory of
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impingement on the rotator cuff tendons,
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this is the story that you would tell.
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There are gonna be some abnormality in the
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cortical acromial arch.
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And that includes of course, the acromium, the clavicle,
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the corco acromial ligament, and other nearby structures.
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And that abnormality produces some sort of mass,
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and that mass pushes down on the external
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surface of the tendon.
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Now the foursome showing
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by the blue arrows here is compression.
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Compression will injure tendons,
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but it is tensile force that is most important
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for tendon tearing.
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So if you believe in this pattern, you're gonna have
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to explain why within external force we get such failure
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of the tears, uh, of the tendon.
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Certainly we can get sal sided fraying
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with this particular picture as I show it.
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Well, the argument that the believers in this theory would
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suggest, oh, wait a minute, as I push down on the top
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of this tendon,
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what will occur is a convexity involving
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the inferior or articular surface of that tendon,
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and through a process of tend
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tensile undersurface fiber failure known as tough.
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That is why articular cited tearing is the
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initial abnormality.
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And indeed what occurs can be full thickness tearing.
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If you believe in this, then any process
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that narrows the subacromial space could lead to
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external subacromial impingement.
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Now, the major finding we see statistically is usually
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osteoarthrosis of the acromial vic of the joint,
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particularly when there is capsular distension
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and osteophytes that are contacting
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or even depressing the myo tendonous junction
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of the supraspinatus.
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Uh, it certainly looks like there is impingement,
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but I would counter, again, I don't think that is diagnostic
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of the clinical features of the impingement syndrome,
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but there are other causes certainly that could lead
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to the structural findings that are required
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for external subacromial infringement.
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And that is why there's been a lot of interest in the shape
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of the acromion, and particularly with the diagnosis
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and identification of an outgrowth
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that is called a subacromial and feso fight,
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or simply a subacromial spur.
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Here's a beautiful example of
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what it looks like in a specimen. Here.
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We can see it in images right here.
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This represents ossification at the acromial attachment
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of the cortical acromial ligament.
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Alright? That's what it looks like.
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That's what it is, and that's why it points
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to the cricoid process
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and of all of the imaging findings that we look for,
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this is the most specific,
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although it is not totally diagnostic,
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it's the most specific.