Interactive Transcript
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It's a 58 year old woman complaining of right posterior hand
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pain and a history of carpal tunnel, uh, surgery.
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And most of you correctly picked up on some changes in the flexor
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macula and some changes in the peritus soft tissues. For instance,
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these circumferential areas of high signal represent fluid
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in the, uh, extensor sheaths. And if you look very carefully,
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there's a similar phenomenon anteriorly. This is real.
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There's some fluid in the flexor sheaths. Now,
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sometimes when you have fluid in say, rheumatoid or related conditions,
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you'll see actual synovitis. Um,
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most of these sheaths are synovial lined in the foot.
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Not all the tendon sheaths are synovial line. For instance,
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the Achilles is not synovial line. So that can get a little bit confusing.
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What's also confusing about this case is that the,
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the history of posterior wrist pain kind of dominates the
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introduction. And so you would have to come up with this explanation,
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namely that there's teno synovial or peritendinitis fluid accumulation in the
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extensors. If you're gonna correlate with the history, there's also,
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and if you correlate with the T one on your right,
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the fat weighted image right here, you'll see that the capsule is distended.
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And this little gray sliver and this little sort of round sliver right
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here, uh, that's what very mild, uh, synovitis looks like. Now,
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synovitis does not automatically mean you have, um,
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inflammatory arthropathy 'cause you can get mechanical synovial hypertrophy.
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And likely that's what's happened here.
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A lot of you are looking at this round object in the capitate,
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which most people have. It is a, because the capitate is a central bone,
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it is subject to friction from all sides.
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So this is a friction induced intraosseous ganglion. Uh,
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you'll see them on conventional radiographs all the time in the wrist.
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It's of no consequence.
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I often comment on it just so people don't get excited about it,
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and sometimes I forget to 'cause I see it in almost every case,
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and it's not really relevant to the patient's, um,
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clinical syndrome 99% of the time. Now,
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the main finding in this case is not on the dorsal surface,
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which makes it challenging. And, um, perhaps explains why, uh,
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quite a few of you did not, uh, tap into the major finding.
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The major finding is that the patient has had a carpal tunnel release.
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And I'm gonna use my pen here.
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One of the descriptors for carpal tunnel release is that you get a
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bhap fibro fatty cleft. So there's the B, there's the middle of the B,
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and there is the other part of the B. Um, and
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Then the cleft is right in the middle.
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You can kind of see that on the T one weighted image,
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there's the b there's the fibro fatty cleft, there's the fat,
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and then there's the other portion of the b. Now around the median nerve,
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which is right here, you should have a rim of fat on the T one.
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So that is lost. It might say no big deal,
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somebody was in there releasing the carpal tunnel space. Of course,
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you're gonna get a little inflammatory tissue there, and that's okay. You know,
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that by itself would not bother me. Now the nerve is normally gray and,
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and there is the gray nerve.
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That's what the nerve should look like on the T two weighted image.
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That's what it should look like on the T one weighted image,
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although it is obscured by the post-surgical granulation tissue.
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Let's keep scrolling as we get to our fibro fatty cleft where we did
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a flexor vernacular release, which by the way,
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you can do under ultrasound now non-invasively. And, uh,
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that is done in several major centers around the United States,
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uh, popularized by, um, Dr. Nazarian in, in Philadelphia. And,
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and then wait a minute, all of a sudden, and without warning,
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look what happens to our median nerve. It goes bright on us like a light bulb.
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Now it is not supposed to do that. Not only does it go bright, it gets bigger.
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So when I'm looking at nerves longitudinally,
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and right now I'm looking at them in cross-section,
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I don't wanna see the nerve do this.
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I don't wanna see the nerve get bulbous, almost like it has an aneurysm.
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And that's exactly what's happening here. So the nerve is swollen.
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I also do not wanna see the nerve go, go bright on me.
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I don't wanna see it go white on me, so I don't wanna see this.
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And that's exactly what's happening here.
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So there has been a focal injury to the median nerve.
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It's not transected, but it's injured. Now here's, here's a weird thing.
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Sometimes when the nerve has been injured on a chronic basis,
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and this applies no matter where you are in the body, we're talking Mr.
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Neurography. Now sometimes the nerve will get smaller,
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so-called neuro esis of the nerve. So the nerve will,
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will be indented almost like it's scarred, it'll shrink.
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And very often when this shrinkage occurs, what do you get at that site?
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You don't get white, you get black or dark gray,
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something like this.
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And that's paradoxical fibrosis o o of the nerve.
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So these are the things you should be looking for. Swelling with high signal,
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not on a stir, not on a spur, not on a gradient,
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but on a T two spin echo,
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which this is so bright signal in the nerve on a T two
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spin echo without nerve transection is consistent with a
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traumatic neurotic phenomenon of the median nerve at a site of carpal tunnel
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release. Now, when I look at carpal tunnel release,
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I wanna see that that cleft go all the way to the hook of the handmaid.
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And it does.
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And then I wanna see it back at the level of the pisiform and maybe not so much.
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So they,
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they probably could have taken the release back a little further to the pisiform
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level, but I, I'm more concerned that they,
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they don't release it at the handmaid level. And they did.
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So this could have had a little more release, more proximally,
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and that probably should have been commented on.
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But no doubt this is a major finding.
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Now what do you do about it? There's not a lot that you can do at this point.
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Um, you can try nerve stimulation, you can inject steroids,
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being very careful not to hit, hit the nerve directly.
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Now another thing you can see here is that there is in the deeper fibers of the
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thenar eminence, there's some atrophy and fatty replacement.
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Now let's keep going on these axials and see if there's anything else.
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We've already established that there's flexor peritendinitis or teno
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synovitis. There's extensor the same thing.
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Here are the extensors and they're all,
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they've all got a little fluid around them.
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And here's a little pearl when you fracture the wrist,
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because the periosteum of the dorsum of the wrist communicates with the sheath,
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the sheaths will always distend.
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So you shouldn't call that tina synovitis,
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you should call it reactive fluid secondary to a fracture.
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Now this patient doesn't have a fracture that doesn't apply here,
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but that's just a pearl for the future.
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And you can see a fair amount of fluid around the extensor carpe ris.
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The extensor carpe ris sub sheet is right there and it stays, you know,
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pretty nicely where it's supposed to. If we go a little more towards the groove,
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we didn't get there, but it's in, it's in the right place.
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Now let's turn our attention to the coronals,
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see if there's anything there of interest. So let's take a look at
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the triangular fibrocartilage for a moment.
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And I admit it's a little difficult to see at low field,
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but there it is right there.
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And you can see there's some tissue thickening right here.
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Now this right here is hyland cartilage. That's normal,
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but this tissue thickening is abnormal and there's fluid in the distal radial
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ulnar articulation.
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So this patient has a chronic central third perforation
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of the triangular fibrocartilage. Once you see that, you should look for ul,
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no malacia doesn't have it.
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You should look for lu triquetral ligament tear doesn't
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have it, uh, to see if the patient has, you know,
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some type of ulnar lunate abutment syndrome.
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Let's see, what else do we have in this case? We've got some generalized carpal,
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uh, capsulitis that, that fluid that's present there.
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That's a term I use frequently unless I see frank synovitis.
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There's our intraosseous friction related ganglion. Uh,
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of the capitate.
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We've already established that there is fluid in the D R U J,
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there's some fluid in the carpus, there's some fluid in the flexors,
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there's some fluid in the extensors and there's fluid in the metacarpal
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phill joints. There, there, there, there, and there.
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The white stuff shouldn't have that either. So why is that there?
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I'm not really sure. I don't know the answer to that. But if this, um,
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is, is a woman, which it is, and I I didn't have another explanation,
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I would absolutely vet this patient for a rheumatologic disorder.
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And remember, people with rheumatologic disorders, including RA,
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will get secondary carpal tunnel syndrome. Now,
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that wasn't mentioned here and it probably should have been in, in the,
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in the official report. So that, that is something to consider.
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There's no good reason why this patient should have fluid in the carpus,
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fluid in the sheath and fluid in the cps.