Interactive Transcript
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The next case is, um, a fairly young gentleman
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who had already had a stenting procedure.
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And you can see that stent right here in the LED.
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And, um, rather than worry too much about the coronaries,
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I wanted to talk more about the myocardium.
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So it was known, I believe,
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that this patient had an occlusion.
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Let's go back and just review
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the myocardial segmental anatomy.
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If you take these axial dataset
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and you put your cursor in the middle
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of the mitral valve plane and then go parallel
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to the interventricular septum on this axial image,
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you'll create the two chamber view.
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And on this view, you wanna go perpendicular
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to the long axis.
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So that green plane is defined here.
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This is your short axis.
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And then if I find the middle of the, um, right ventricle
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and bisect the acute margin, this view resulting, um,
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defines the true four chamber.
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And if I take it up to the basal level
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and bisect the left ventricular outflow tract,
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I would define the three chamber.
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But no matter which long axis view I look at,
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I see the apex vial, and I don't like what I see.
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And what I'm seeing here is a
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hypokinetic left ventricular apex and a thrombus.
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So the most common cardiac mass is thrombus,
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and the most common cardiac disease is
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ischemic heart disease.
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So really not a shock.
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Um, but the apex can be close to the echocardiography probe
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and too close to the chest wall, to the point
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where you can miss that due to artifacts.
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So you might wanna warn if you're the first to know,
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because the echocardiogram can be done differently
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or more carefully to look at the apex.
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Um, but also, uh,
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if I wanna look at the
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myocardial function, I can help out here.
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And so I might go to an eight millimeter average NPR,
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and I would mention that the mid to apical, uh,
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interseptal walls as well as the,
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I can just look at each one and kind
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of do it like a checklist, really.
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The anterior, the lateral, the inferior,
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and the septal, uh, walls
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of the apical segment are hypokinetic.
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So I have this noncalcified thrombus,
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and I have regional abnormality in the, uh,
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territory of the LED.
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And if you remember, I just glossed through it,
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but there was an LED stent.
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So this is not, uh, a shock that it fits the territory.
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Uh, but important to note this complication.
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Say you're not sure if there's mixing artifact.
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This is well defined, and, uh, I'll stop the syn
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and just show you here that's sharply de marketed.
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I can always just do a delayed image and see that persists.
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So one to two minutes is all you need.
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Um, if you check the images and find them, uh, great.
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If you don't, then you would just wanna make sure you
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add that to your protocol.
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And I'm second guessing.
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Oh yeah, there might actually
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just be extensive atheroma and not a stent.
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I can't remember whether this person had a,
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a stent that was occluded.
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In fact, it was not a stent, it was just tram track calcium.
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So I, what I was showing you, there was stenosis
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and, uh, extensive on of plaque.
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The dis LLAD is probably occluded,
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but there's certainly not, uh, a stent,
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but rather just a, a fair amount of plaque important
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to make a phone call if you do
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Find this and you're the first to know.
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Uh, and the other interesting thing about this patient is
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that there is a, uh, uh, lot of disease in the RCA.
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And I believe when we sorted out the wall motion, uh,
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we looked and we went through segment by segment,
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and we noticed a little bit of lateral wall, uh,
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hypokinesis and thinning too.
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So really, uh,
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the most profound abnormalities in the LED territory.
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But you also have, uh, this lateral wall finding,
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which would suggest, uh, circumflex disease.
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And as you can see, the proximal circumflex,
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we'll show this jump to real quickly.
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Circumflex has its share of disease as well.
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So again, important sequela of infarction, uh,
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thrombus formation.