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Myocardial Scarring (Case 1)

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0:01

Okay, this next case is unique in that it was, um, done

0:05

in part to look at the coronary arteries.

0:07

Um, in that every TAVR scan, uh,

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gets a look at coronary arteries, but it already had a cath.

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So I just wanted to show you, um, a late sequela

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of ischemic heart disease, difficult scan.

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Um, not really, uh, important that we look

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through all the coronary arteries,

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but, um, we can look through the bypass graft anatomy.

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This is not even gated, this part.

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And you can see there's a vi bypass

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graft going to the saphenous.

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There's the lima, which is very disease, very hard to see.

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REMA is not used. And then here's a right sided bypass graft

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and a native occluded RCA.

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Um, but what you notice on this delayed non-G gated series

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is hypoperfusion of the myocardium.

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So there's a couple ways to look at profusion.

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We talked about the ischemic cascade,

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um, and you even see thinning here.

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So I'm gonna, um, briefly look at the myocardial function.

0:57

So we have a multi-phase exam here.

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And the first thing I notice when I'm looking at the

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ventricle is I'm not looking at thin cuts for the arteries,

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but rather thickening the image

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to make it easier on the eyes to see sub endocardial

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or transmural infarcts.

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Now this here is a multi-phase exam.

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If I'm gonna measure wall thicknesses,

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I'm gonna make sure I'm an end diastole.

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And the first thing I could do is just draw a quick line.

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That's a very large ventricle.

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So if you look at echo normative databases, um, up

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to about six millimeters for a tall man,

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and I think 5.4 for a tall woman is about top normal.

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This is seven. So there's nowhere near any, uh, normal here.

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This is a, a dilated ventricle,

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could be from a number of reasons.

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In this case, I think it's probably due

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to aortic valve disease as well as, uh,

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ischemic heart disease.

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And the reason I I'm suggesting ischemia is you've got a

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bypass graft and you have some hypo enhancement.

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And you've noticed here I've made an average intensity

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projected eight millimeter image tight window within level.

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And that kind of highlights this first pass rest perfusion.

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Uh, and you can see a couple

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of sub endocardial abnormalities.

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And as you remember, the epicardial coronary

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artis flow from outer to inner.

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Um, and so the distal pressure head, the ischemic wavefront,

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is worst at the inner layer.

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That's why you get sub endocardial abnormalities.

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This is your first pass.

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So, um, you could indeed look at a delayed image and do this

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and, and see that it's more profound.

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Um, or you could go back

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and look at your functional information

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and look at how well does the heart contract.

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And so when I look at this wall motion,

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it's not quite a normal amount.

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And I also wanted to point out in the distal apical segment

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here, there's thinning and maybe perhaps kinesis.

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And there was an LED territory insult, uh, at some point.

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So, um, we don't necessarily need, uh, to look at

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that on every scan if it's known by echocardiogram.

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But when you have a limited echocardiogram

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or it's complimentary

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or you're just the first to know,

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finding sequela ischemia is important.

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Uh, you can even see the anterior

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septal wall is thinning here. That's the LED

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Territory. Uh, important

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pertinent negatives that I mentioned,

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if I see those findings are, um,

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is there a calcified aneurysm?

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Is there dyskinesis?

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Is there thrombus within the ventricle

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or really any part of the heart?

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But every, uh, cardiac CT is a chance to roll out thrombus.

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It's far easier than on an echo echocardiogram if you're

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lucky enough to have that with a ct.

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Just make sure if you need to,

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to sort out mixing artifact from delay with a delayed image.

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So here's a rapid scan immediately following the first scan.

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And then what we do for our, uh,

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structural heart scans is just always

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include a delayed series.

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So here's that. So a lot more equilibrium phase,

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but if there were any question of mixing artifact,

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it'd certainly be sorted out by one to two minutes.

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This is actually a nice look at the myocardial

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hypoperfusion here.

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Let me just give you another look at the, uh, subin cardium.

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So again, I'm thickening my view so

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that I'd sort out the noise.

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And you can see that fatty metaplasia and hypoperfusion

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and the basal infra septal

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and then the interseptal walls, so sequelae

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of ischemic heart disease

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and look at that little papillary

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muscle, uh, poking in there.

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So when you see calcification in a papillary muscle,

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it's nearly always from ischemic heart disease.

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So, uh, just something else to pay attention to.

Report

Faculty

Brian Ghoshhajra, MD, MBA, MSCCT

Academic Chief, Cardiovascular Imaging and Associate Chair, Operations Analytics

Massachusetts General Hospital / Harvard Medical School

Tags

Vascular

Myocardium

Coronary arteries

Cardiac Chambers

Cardiac CT (SCCT Cat B1 Video Case)

Cardiac

CTA

CT

Angiography