Interactive Transcript
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Before I start, I just wanted
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to show you this high resolution one millimeter on,
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on just a regular man magnet with an arthrogram.
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And frequently we don't perform arthrography in the wrist,
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but just so you know, I know you're passot by ligaments,
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but I wanna inculcate you with, with the terms
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and, and prove that Dr.
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Resnick is not making these up. They really do exist.
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And here on an mr you can see
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that radio SCA O capitated sling ligament.
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There's your long radio NATO triquetral ligament,
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not showing you the triquetral parts.
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So there are two pretty important ones right there.
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And you, you make an inverted V for the,
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for the deltoid ligaments, you know, the superficial layer.
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And then as you get deeper, you make a very, a smaller V.
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And you can see that V here's one limit of the V right there
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as part of the arcuate.
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The other one not as well seen, uh,
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he wasn't making up the radial collateral ligament.
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It really, really is there.
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And then as you get onto the dorsal side, you see
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that your V is no longer inverted.
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It's kind of sideways just to help you young
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and sort of memorize some, some of these ligaments.
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Here's the dorsal transverse carpal ligament,
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and here is the long LAR radio lunate
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or radio lu NATO triquetral ligament.
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And there's your, there's your sideways beam right there.
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So now let's get out of that and into the case.
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Uh, there are a couple of views I use in the wrist when I'm
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looking at scapholunate ligaments.
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And, you know, I especially don't necessarily want
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to put contrast in the joint.
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It's not hard to do.
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We've been doing 'em for a long time,
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but we will use sometimes to elucidate dynamic and,
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and static forms of instability, a steep radial
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and ulnar deviation view.
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And we'll bring the patient back.
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And that's more efficient than, than doing the arthrogram.
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Another thing we'll do
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for radial ulnar instability is we'll do steep pronation
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and supination of both wrists and compare the two sides.
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Uh, let's look at our first case style.
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And this is which one? 34. 34.
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So this is a 25-year-old a youngin, uh,
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with pain in the wrist.
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He fell, he fell through a trailer
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and landed on both of his outstretched hands.
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He's got a, a, a history, a previous history of,
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of fractures from it.
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This case is interesting on a number of levels.
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Uh, this happened several months ago, so most
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of the fractures have bridged.
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You can see he's got a little bit of residual bone edema.
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But we do have a wide, uh, scapholunate interval.
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So we have an example of at least sit, CID
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and it looks like he's avulsed.
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He has avulsed the scapholunate ligament from
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the scaphoid side.
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Remember, you can get it from the scaphoid side,
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from the lunate side, uh, or, or, or in the middle.
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Um, and you, you can volts from,
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from either side of the bone.
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So there are a few variations, I think, described
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by Anderson in that classification system.
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And then there's another finding
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that I'm gonna come back to in a minute.
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Look at the marrow. It looks, it looks a little strange.
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He's a youngster. And you're seeing this sort
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of window framing a little bit reminiscent
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of say, Paget's disease.
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This edema around the periphery in virtually every
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bone in a young individual.
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Look at these little stippled areas of hyperintensity.
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When you see this, especially in somebody with persistent,
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uh, pain in the extremity.
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You have to think about CRPS
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or complex regional pain syndrome.
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Type one, formerly known as the artist,
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RSD reflex sympathetic dystrophy.
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Now let's look at our sagittal projection.
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And we've got a, we've got a, um, a lunate
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that is facing dorsally and, and why is that?
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Earlier we described that some of these instabilities a
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and changes in bone position don't occur just from, uh,
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intrinsic disruption.
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You have to have frequently extrinsic disruption.
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Now remember, extrinsic doesn't mean extracapsular.
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There aren't that many extracapsular ligaments,
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the pizzo hamme, the pizzo metacarpal
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and maybe one other one.
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Uh, so how did he get this?
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Well, you saw earlier, I know it was a blur
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'cause it's a lot of ligaments,
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but where is the radios scfo capitate ligament.
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Uh, we can see a little bit
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of the short radio lunate ligament here,
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but the radio sca capitate ligament should come
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plugging in right about here.
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And you ne you never see it. Uh, come in.
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Let's look at the coronal projection. Let, let's, sorry.
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I, the radio scap capitate ligament is here.
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My apology here on the lar side of the scaphoid
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where it should be obvious right there.
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And then as we get to the coronal projection,
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we see the long radio lunate ligament, also known
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as the radio triquetral ligament.
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But look at the weird, almost sagging like position
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of the radio scap capitate, uh, ligament
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and even its base looks a little bit strange
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as it comes off the, uh, periphery
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or stylo styal portion of the radius.
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So this patient has torn the radio
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sca ffo capitate ligament, a very important volar extrinsic.
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And they've also avol the scapholunate ligament off
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of the sca filling.
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Remember in the scapholunate ligament, it's the dorsal SLD
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that's most, most important.
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On the lunate side, it's LTV, like a TV
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and that kind of has helped me through the years,
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remember which one is more important?
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Vola on the lunate side.
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Dorsal on the scaphoid side are most important.
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Don, do you have any comments on this one?
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Yeah, I'm gonna comment on, on one
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of the findings you mentioned,
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which kind of had an interest.
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And, and that's the marrow, uh, changes and,
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and, uh, this is a very distinctive pattern
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where you have a, like a bone within bone appearance where
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the major abnormalities along the periphery.
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And, uh, this is an interesting finding.
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It was actually emphasized probably about five
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or six years ago by Larry White in the knee,
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not in the, in the wrist.
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And he was talking about patients who had had
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injuries, uh, to the knee.
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And in the subacute stage he was pointing out the hyperemia
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that occurs and the border like, uh, finding.
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And in the knee it's usually the patella where you see
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that in major, uh, it seemed best in the patella
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and it is hyperemia.
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And it certainly, when you see it, you have to think
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of complex regional pain syndrome.
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It also can occur particularly in the subacute stage of
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injuries in young people without having
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complex regional pain syndrome.
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Um, so I usually give 'em a, a, a differential
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for that, uh, finding.
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Once you give someone complex regional pain syndrome,
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I think it's kind of like a lifelong diagnosis.
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It's in, in many pathway of, my associate always told me,
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uh, don't give it as a single diagnosis.
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That that often.
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The other point, if you think back to radiographs
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and you look at patterns of osteoporosis
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that occur fairly rapidly
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as in complex regional pain syndrome.
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The other one are bands of lucency
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that occur either in the epiphysis
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or at the metaphysis where the physio plate was.
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So that's another pattern sometimes you'll see with Mr,
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where you see bands
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and that do suggest hyperemia
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as you indicated in this particular case.
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And I, and I don't think this patient turned out to have,
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well this patient did not turn out to have CRPS,
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but I unfortunately, I've had
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very deep personal experience within my own
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family with this condition.
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It affecting the essay node in this person, the intestines,
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the bladder, the throat,
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and of course the lower extremity, which
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she nearly lost as a result of the condition.
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So I'm very sensitive to it.
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Anytime I see this peripheralization, uh,
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of marrow hyperintensity,
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it's at least something you've gotta think about.
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'cause if you miss it, the longer it goes, the harder it is
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to get rid of and treat.
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But it is a real thing. Yeah,
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Yeah. Okay,
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let's move on to the next one. I.