Interactive Transcript
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So when we have injuries that are largely at the surface,
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they are mostly at the fascia.
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We don't have any big holes in the muscle, it's just emus.
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But the architecture's reasonably good.
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Small areas in here, these ones tend
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to do better than when the tendon is involved.
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And these injuries that are predominantly
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around the epimysium, nice example here involving the soleus
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and a runner, uh, make up about somewhere between four
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and 20% of muscle strains.
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And the patients are often able to return
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to their activities relatively quickly once you start
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to get into tendon damage.
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And here you can see that the tendon
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of the gastrocnemius is disrupted
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and we have voids in the muscle, not just edema,
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then you're looking at a significantly longer
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return, uh, to play.
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And again, uh, the validations that I've mentioned here,
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they have recently done some validation work in the
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quadriceps as well.
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Um, just this is, uh, this tendon damage seems
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to correlate really best, uh, with the longer,
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uh, return to play.
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And then the worst prognosis is damage to the free tendon.
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And here we see a free tendon disruption
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of the conjoining tendon
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and high grade of the semimembranosus more anteriorly
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with a large fluid collection.
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And obviously this is going to take a long time to recover
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and many of these may require a surgical, uh, reattachment
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of the tendon when the tendon is torn, uh,
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near the footprint, uh, as it has, uh, in this, uh, example.
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So what do I like to report? Uh, I don't give a number.
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I basically say which muscles involved where it is,
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whether it's proximal, mid distal, a rough idea of size.
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I just do it as a cross-sectional area.
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I don't bother with diameter. That makes no sense to me.
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'cause muscles are different sizes.
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The length of the area really focus in on whether there's
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architectural distortion.
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The tendon itself is abnormal.
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The length of the tendon abnormality in which tendon
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region, uh, is affected.
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I do like the feature of, uh, FC Barcelona
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that they consider previous injury
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because we know that an athlete
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that's had an injury in one location is at higher risk
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of getting an injury in the same location.
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This was an athlete who continued to play.
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Uh, there's a lot of financial downside
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to these athletes not playing.
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So they, they, they, they don't sometimes do rational things
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and he's reinjured himself 12 days later
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and now has a much more extensive injury
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with the frank tear, uh, in the muscle.
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We also know that re injuries take longer to heal.
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And really that's the only thing from FC Barcelona
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to add into the baac.
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And I always look for signs of prior injury because
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Healed does not mean normal.
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Uh, tendons that have been damaged when they heal
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are often stiff.
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And we have loss of tissue compliance.
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So here we can see that there's a lot of tendon thickening.
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There's not any fluid around it. Perhaps a tiny bit here.
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The patient at this point did clinically have a low grade
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strain, uh, injury, just a halo of fluid, uh,
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next to the tendon.
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But you can think about this kind of as a rubber band,
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it's really hard to tell by looking at a rubber band if it's
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gonna stretch or if it's gonna break
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or if it's gonna be stiff.
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And, um, so we don't really know by looking at this tendon
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how it's going to behave.
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So just because there's no fluid does not mean that
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that tendon is gonna be normal.
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Here's another example of a previous injury.
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No edema in this case.
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Lot of thickening in the interface
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between the gastroc anemia and the soleus.
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I don't see the plantar in this case.
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I don't really bother about the plantar actually.
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Um, but uh, we know
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that there's been a so-called chronic tennis leg injury.
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And then we also have muscle atrophy.
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The medial head of the gastroc anemia add,
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its a neurotic insertion, uh,
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to the tibia is Foley, uh, atrophic.
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So those are the findings that you really wanna look for.
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Tendon thickening and irregularity, focal regions
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of muscle fibrosis or atrophy.
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Just another example here in a rectus injury, we have, uh,
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the higher levels abnormalities involving the direct head
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of the rectus, which is fat replaced
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and further distally abnormalities involving the central
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indirect head, which has fatty replacement,
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perhaps a little bit of tendon thickening, uh,
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and, uh, irregularity, uh, as well, more distally.
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So those are what you're gonna look for.
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And remember that this doesn't really have any mass effect.
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You're gonna be looking for areas of atrophy
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that don't cause a mass like a lipoma would.
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Uh, that's simply fat replacing the damaged, uh, muscle, uh,
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tissues.