Interactive Transcript
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To finish up hyperthyroidism.
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Now you all learn about this disease
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as resonance in in radiology.
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Uh, I can remember very clearly,
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I think it was in the first week, whether I was a ra,
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a radiology resonance.
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I learned about subperiosteal resorption of bone
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and I was taught to look at the hands,
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to look at the phalanges, to look at the terminal tufts
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for the classic features.
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But as I show you here with this list, bone resorption
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and hyper power thyroidism can indeed involve different
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types of resorption, intracortical, endo,
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sub ligamentus, SubT tenderness and sub conval.
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So if you ever wondered why the sacroiliac joints widened in
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primary or secondary hyperparathyroidism
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or the synthesis pubis widens,
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that's not subperiosteal resorption.
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That's sub conval bone resorption, typically bilateral
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and symmetrical.
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It can simulate early stages of sacroiliitis as shown here.
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And just to show you the same phenomenon occurring
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elsewhere, these are the Sterno
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Pav joints you can see here by uh, image
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and also in a specimen subcon bone resorption
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involving the clavicle
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and sternum widening
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of the corresponding interosseous spaces,
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erosions surface irregularity and bone sclerosis.
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Same thing occurs about the acromioclavicular joints.
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Beautiful example.
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Uh, Karen Chen, uh, showed this one not too long ago.
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This is all subcon bone resorption,
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bilateral sacroiliac joint involvement,
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symmetric simulating sacroiliitis.
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This is related to chondral bone resorption in the glenoid.
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Here you can see the irregular surface of the femoral head,
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subc chondral bone resorption in hyperparathyroidism.
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And another phenomenon to remember,
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you get subc chondral bone resorption in this disease
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in the vertebral bodies.
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So multiple and large cartilaginous
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or sch smalls nodes may be seen particularly in those
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patients with chronic renal disease.
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And you can see that nicely in this example.
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And then to finish up our talk infection, of course,
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the classic distribution
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and infection is unilateral involvement
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in the peripheral skeleton monoarticular involvement.
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So to show you a couple of examples here,
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septic arthritis involving a sacroiliac joint
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with all the classic features that you would expect,
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the unilateral distribution being very, very important.
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And then one further example shown here,
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unilateral involvement, in this case,
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the right siliac joint. You can see
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The changes on the fluid sensitive sequence.
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And here with gadolinium,
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this is an abscess present anterior
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to the involved sacroiliac joint.
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So what I've done over a period of 45 minutes is
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to fulfill hopefully three objectives.
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We studied the anatomy of the sacroiliac neuros space,
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emphasizing the true portion, the sacroiliac joint,
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and then the adjacent ligaments.
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We've reviewed imaging methods throughout,
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although I have emphasized MR.
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Imaging in this particular talk,
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and we documented some
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of the characteristic morphologic features.
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But most importantly, we documented the distribution,
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distribution, distribution, distribution,
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very important in the differential diagnosis
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of sacroiliac joint disease.