Interactive Transcript
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Okay, this was an SMA embolism
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causing mesenteric ischemia.
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Here on this 3D image, you can see the celiac axis,
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and the lack of enhancement of that proximal SMA,
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as we saw on the images previously. Acute arterial
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occlusion is usually from an embolic source, most
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commonly from a cardiac source with a patient
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who may have AFib or other cardiac abnormalities.
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So, we always look for intracardiac thrombus
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in the setting of emboli or embolic disease.
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I would say also, even when I'm trying to protocol
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these abdominal pains, sometimes if someone tells
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me that the patient has underlying AFib or a
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cardiac issue such as this, I will automatically
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protocol that case with an arterial and portal
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venous phase because these emboli are so much more
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easily visualized in the arterial phase of imaging.
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You can also get in situ thrombus.
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That's probably 15% to 30%—just develop
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at the level of an ostium, secondary
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to underlying atherosclerotic disease.
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Um, you can see that this patient
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has a lot of calcifications.
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They had actually already had surgical aortic
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graft, so they're definitely a vasculopath,
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so that is an opportunity as well.
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The fact that there were splenic infarcts
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there probably cinches the deal that
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it was more likely embolic in nature.
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Aortic dissections can occur and result in mesenteric
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ischemia as well, so you always want to look for
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that as a cause for your mesenteric ischemia.
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So, how does the bowel look with mesenteric ischemia?
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Because the bowel, you know, has lack of blood
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flow and the blood supply is reduced, it'll become
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ischemic, and it will have typical features of
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ischemic bowel, including this absent enhancement.
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You can see as the enhancement more
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proximally of these little folds just
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gets lost in these dilated ischemic folds.
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You're going to have a very thin
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wall in arterial enhancement.
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It's a blood
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inlet problem.
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You're not getting enough blood to the bowel,
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so the wall will be nice and paper thin.
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You can see that along where these dots of air are.
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Usually you'll have some ileus.
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The bowel isn't going to like to be
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ischemic, it's not going to do its work
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if it doesn't have the blood it wants.
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I mean, it's not going to be happy.
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So, as a result, you'll get some dilatation as well.
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You may go on to pneumatosis, where there's actually
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air in the bowel wall.
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As that mucosa breaks down from ischemia, it
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allows air from the intraluminal contents to
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go into the wall, which can then be reabsorbed
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into your portal venous tract as well.
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So, this is a beautiful appearance of
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arterial ischemia of the small bowel.
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This would be considered a watershed zone for the SMA.
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There are a lot of variable
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watershed zones that aren't as
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applicable to the general population because it
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depends on where your atherosclerotic disease is.
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But this would definitely be a location where
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I've seen a number of ischemic insults from
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SMA occlusions and/or the like, frequently
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affects the distal small bowel and cecum.
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Here's a case which is similar.
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But this time we can see that there's a small focus
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of enhancement within the lumen, and there's a nice
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crescent of lower attenuation. On the sagittal image,
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we can see just that nice small, um, lumen here.
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This is an SMA dissection.
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We do see celiac axis and SMA dissections at times.
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They don't have a really good natural
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history of who gets these or why.
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Definitely higher on your Ehlers–Danlos
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patients, but they can be somewhat sporadic and
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definitely not something that
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people would be expecting.
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So, I always look very carefully at the vessels.
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It's definitely one of my, uh, search patterns.
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I like to look at the celiac axis and SMA specifically
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on the sagittal view because I always feel like
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this view gives me the best image of those vessels.