Interactive Transcript
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Hello and welcome to Noon Conference,
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hosted by M R I Online Noon Conference connects the global radiology community
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through free live educational webinars that are accessible for all.
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And as an opportunity to learn alongside top radiologists from around the world,
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You can also sign up for a free trial of our premium membership to get access to
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hundreds of case-based micro-learning courses across all key radiology
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subspecialties. Today we are honored to welcome Dr.
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Stephen j Pran for a lecture on wrist M r I. Dr.
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Pomerantz is the c e o and Medical Director of ProScan Imaging,
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chair of Naples, Florida Community Hospital Network,
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and the founder of M R I Online.
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He's authored numerous medical textbooks and M R I, including the M R I,
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total Body Atlas. Dr. Pran is also an AVID conference,
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lecturer and chairs. This the fellowship training program in MR.
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And Advanced Imaging.
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We're thrilled he's here today to share his expertise with us.
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At the end of the lecture, please join Dr.
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Pran in a q and a session where he will address questions you may have on
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today's topic.
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Please remember to use the q and a feature to submit your questions so we can
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get to as many as we can before our time is up. With that,
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we are ready to begin today's lecture. Dr. Pomerance, please take it from here.
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Okay, great. Good afternoon, everyone. Good morning everyone.
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Wherever you are in the world, we're talking about wrist, M r I,
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which is a complex subject. The further away you get from the trunk,
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the more detailed the anatomy, but,
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but it is a joint unlike the ball and valve joints. That makes some sense.
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So it's not quite as difficult that as it appears to be.
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And I'm going to focus on, uh, a few issues. But before I do,
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I wanna remind you that on September 10th through 14th, Dr.
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Don Resnick, uh, will be presenting a course on the upper extremities,
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uh, along with Megan Mills and Christine Chung for the hand, wrist, and fingers.
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And I will be doing the case reviews in between the lectures.
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Uh, the lectures will be interactive.
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There'll be case reviews that are scrolled, uh,
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approximately three hour sessions each day.
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And we're gonna be focused on the shoulder, the elbow, the hand and wrist,
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the fingers, and entrapment neuropathy. So, uh,
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hopefully you'll tune in and get some of your pressing questions regarding, uh,
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the upper extremity, uh, in front of us.
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So what we cover in this lecture is technique and anatomy,
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the triangular fibrocartilage, ulnar variants, simple carpal instability,
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complex carpal instability,
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and slack and snack wrist.
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We're not gonna spend a lot of time on, uh, uh, things like, uh,
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masses and soft tissue lesions and arties since we're we're time
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constrained. Uh, let's start out with technique. And,
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um,
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I am showing you on the right a rigid wrist coil with the arm at the side.
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I typically like to have the arm a little bit closer to the body. Uh,
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I scan my patients with either the thumb up in the neutral position or in
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pronation. Uh, most people cannot hold still in supination.
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And the goal is to get something, you know, like this,
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which is a high resolution image, two millimeter slice, thickness high matrix,
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and you can even see the, the trabecula of the bone,
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the triangular fibrocartilage with its radial and peripheral and distal
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attachments. But I'm showing it for the anatomic detail, uh,
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which is greatly influenced by the field of view and the wrist.
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I don't like to have fields of view of greater than 12, uh,
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on the average with a, with a high quality instrument.
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I like my fields of view to be around eight, maybe nine,
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and that one was about seven or eight. Here's a patient, uh,
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with the arm at the side, another rigid, uh, wrist coil.
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This time the the hand is in pronation and we're asking 'em to perform a
4:13
clenched fist view to look at the ulnar variants. More on that in a few moments.
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Here's another, uh, rigid coil with the thumb up in the neutral position.
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Now, as far as the sequences go, this applies across the board.
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T one is used for bone marrow in any, in any body part. Uh,
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we'll use gradient echo, either two D or three D uh,
4:35
for intraarticular assessment, the capsule and the cartilage.
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And there are some fancy new gradient,
4:41
echo sequences known as adage from Hitachi,
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merged from GE Merge Fast Field Echo from Phillips and Medic from
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Siemens,
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along with a related sequence called steady state free pre procession or fiesta
4:55
from General Electric that are used in the risk to get an art arthro graphic and
5:00
joint effect at high field.
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I personally like the proton density, uh,
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tally sensitive inversion recovery over the T two fat suppression.
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Some of my colleagues on the west coast prefer the T two fat suppression, but I,
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I feel like I get a little more signal, a little more pop with,
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with this sequence. That's not to say I don't have a T two, uh,
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but I always have this as part of my workhorse sequence for any joint
5:28
at low field. I'll use short time inversion recovery or stir,
5:32
and then I'll use T two to characterize, uh, tendon injuries and to date them.
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When I say data, I mean acute, subacute, chronic, remote, and so on.
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So when I start out,
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I put up my coronal sequences first because I'm most comfortable there,
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you know, from, from doing radiography for so many years. Uh,
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I'll put up, say the T one, the gradient echo,
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the T two and the proton density spur if I have all of them.
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But I will initially focus on the heavily water weighted sequence,
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the proton density, fat suppression,
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looking for hotspots that makes it particularly easy and quick and expedient and
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pragmatic to interpret. So here's a hairline fracture at the distal, uh,
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waist and tubercle region, uh, of the scaphoid. It's so easy to see.
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There's the crack and there's the edema surrounding it.
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Here are three sequences, the T one that you've already seen,
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an anatomy sequence. And then in the center is a low field sequence,
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A two D gradient echo. Some of you know it, know it as uh,
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field echo gradient, echo flash.
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And then on the left is a three D gradient,
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echo sequence with slightly thinner sections.
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And I'm sure you're all seeing this, um,
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this granulation tissue that's involving the lunate.
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That's not why I'm showing it.
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I'm showing it for the shininess of the gradient echo,
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the ability to see into the joint.
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These linear black areas are the collapsed capsule.
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The slightly brighter areas on either side represents synovium and the hylan
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cartilage. And you can see that best on this three D gradient echo sequence.
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By the way, you can also see it on the T one.
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There's the very thin collapsed capsule in black,
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and there are the hyland cartilages on either side that are gray.
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Now a related sequence to these,
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these gradient echo sequences is the steady state free procession,
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also known a a as fiesta.
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And this sequence also gives you heavy water weighting. It's very robust,
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so it allows you to get the field of view down to around 7, 6, 5.
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These are one millimeter slices, 1.2 millimeters.
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And look at the band like portion of the scap lunate ligament, that's gorgeous.
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The radial collateral ligament, the lu NATO triquetral ligament,
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which is intact. And then some tearing out here in the periphery of the T F C.
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But I'm showing it for technique,
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not necessarily for pathology just yet. Now,
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some extreme situations, um,
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I'm going to use extreme c pronation and, and supination,
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uh, to evaluate the radial nerve joint. And I'll do it with both hands. Now,
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I can't see myself, am I on video guys? I'm on video.
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So extreme pronation and extreme supination will allow me to
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assess the radial ulnar excursion on the two sides.
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So it's really hard to make that interpretation, uh, on one side only.
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So I will do it bilaterally. You can do it with ct, you can do it with M R I,
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and I haven't had any trouble getting that reimbursed by, uh,
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insurance companies here in the United States.
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Then we've got steep radial and ulnar deviation that I use for the proximal
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carpal intrinsics, the scapholunate and the lu NATO triquetral ligaments,
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such that I very uncommonly have to do an arthrogram of the wrist.
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Then I've got scaphoid views,
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what I call compound oblique images that I perform on CT and mr.
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And then I also have some sequences and positions that I use for radial
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ulnar relationships. This is known as variance,
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and sometimes I'll use a clenched fist view for, for that purpose.
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And sometimes I'll bring the patient back for some of these sequences.
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So here we are,
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and supination and pronation showing you that there can be quite dramatic
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excursion between the radius and the, the center of the ulna.
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And it is extremely helpful and much more reliable to have bilateral
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assessment of both risks, uh, to make such a diagnosis.
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Now here is a, a coronal reconstruction of a, a wrist.
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But if you're imaging that wrist axially and it's for a scaphoid problem,
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you do not wanna,
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you do not want to image in the orthogonal axial projection.
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You really want obliques, what, what we call compound oblique scaphoid views.
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So you take your, your scaphoid, you draw long axis down it,
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there's a compression screw in place, and then you get this,
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which is a short axis oblique. You then take another oblique off that.
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So you can see it's a very compound, uh, sequence.
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We're starting to see the fracture, we're starting to see the screw.
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We get something that looks like this,
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another compound oblique along the long axis of the screw.
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There's the fracture right there.
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And now we have the entirety of the scaphoid in view. We can scroll that.
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We can count slices.
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We want 50% bridging 5.0 to allow a player to return to
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any type of contact sport, lacrosse, American football and, and so on.
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You can see a lucency here. I'm showing it for the technique.
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There is the compression screw seen. It's in entire entirety,
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in normal position. And then you can scroll through that.
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And as you get to the next slide, you can see there is an area of, of malunion,
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and you have to count slices to get to that 50% number
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that allows that patient go to go back to their activity. Similarly,
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compound oblique on m r i, there's the first oblique.
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There's the second oblique, and then here is the final oblique result of that,
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which is a long axis view of the scaphoid laid out in profile.
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This one being normal. Here's another example of a special sequence.
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Again, i i, I don't often inject the wrist
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when I do,
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it's either a scap o radial injection or a radial ulnar joint injection.
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Um, maybe one out of a hundred w would be as frequently as I do it.
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Here's a patient non-contrast. They did put contrast into this joint.
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Did not need it. Highly suspicious for, uh,
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scapholunate ligament insult because the space is too wide,
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it's a little too swollen. Hey, you're all radiologist.
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Compare that with the all no TriCal interval.
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They look very different from one another.
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There's a little brighter signal right here,
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and then the secondary sign of invagination,
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of capsular synovial tissue into the lunate. Best seen here.
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When we ulnar deviate the fluid kind of comes in and shows you this big gap,
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but you really didn't need it.
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You could have gotten it with ulnar deviation without the contrast inserted into
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the wrist. Why did we do it? The clinician just wanted it projections.
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This also is a rule of thumb that applies throughout the body
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axial. That's, you know, that's our comfort zone. You know,
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we're used to looking at axials. So we use this for masses,
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for tunnel syndromes. I also use it for loose bodies, by the way,
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the sagittal view, anything that's running long, so tendons,
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vessels. And also I use it to look at various complex instabilities.
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The coronal, the workhorse sequence for the risk. I use it for all else,
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especially the triangular fibrocartilage.
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Let's start out with a little bit of axial imaging.
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Here we are at the tubercle of the trapezium,
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the proximal carpal tunnel space. There is the flexor retin aum.
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Here is the canal in which the ulnar nerve subsist.
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This is Ian's canal. Here's the carpal tunnel space. The median nerve,
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often round or triangular surrounded by the sublimes tendons deep
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are the profundus tendons. Here's the flexor lysis longus.
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Here's the flexor carp radialis, which goes towards the,
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the greater angular. And then as we get a little more distal,
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we're at the distal carpal tunnel level.
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We know that 'cause hook of the hamming.
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So tubical of the trapezium proximal with the pisiform hook of the hamate
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distal, although not drawn that way.
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The median nerve tends to be flatter at this level with a lot of the same
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anatomic structures you saw before.
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And the ulnar nerve is divided into a deep motor and a superficial radial,
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uh, a, a superficial, uh, uh, sensory branch.
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So here is the extensor compartment, and we have, uh,
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any one of six compartments here. We've got the first,
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the abductor lysis longest, the extensor lysis brevis.
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One way to remember this is longest brevis longest, brevis longest.
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And then you're off and running. So abductor lysis, longest extensor lysis,
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brevis extensor carpi radiologist, longest and brevis,
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longest brevis extensor lysis longest back to longest.
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Then we're at the communal tendons, then we're at the extensor digit mini.
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Easy to remember 'cause of the pinky finger.
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And the extensor carpi narrows with its sub sheath to be differentiated
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from the superficial Retin aum.
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And then here are our flexor tendons that we described earlier.
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I don't think we need a second description for them. The sagittal projection,
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using this for instabilities alignment, tendons, vessels,
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anything that's long and straight.
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And also for the pizzo triquetral articulation.
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Here are those tendons. Here are the profundus tendons right here.
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Profundus tendon. Sublimes tendon. We're at the midline.
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We've got the capitate, we've got the lunate, we've got the radius.
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They're all lining up relatively straight.
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So we have what we call our mid middle column alignment. Unfortunately,
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my, my pen is not working today. Okay,
15:15
we've got our middle column alignment that goes from distal to proximal and
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goes right through by sex, decapitate by sex, de illuminate by sex. The radius.
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And then here with a yellow arrow is the attachment of the radio
15:30
scavo capitate ligament, which prevents not shown yet.
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Stay tuned. Rotatory subluxation of the scaphoid.
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We're gonna see one detached a little bit later.
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And here's the short radio lunate ligament drawn in.
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Little hard to see on the m r I.
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Here's a series of sagittal views showing the normal architecture and
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alignment of the metacarpals, the, uh,
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the lunate and the scaphoid.
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And if we look at the angle of the scaphoid and goes straight distal to proximal
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in an orthogonal fashion,
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this angle right here along the long axis of the scaphoid between these two
16:11
should be around 45 to 60 degrees. So if the lunate, uh,
16:15
sorry if the scaphoid starts to rotate and sag downwards so that it's almost
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horizontal, we know that we have a rotatory instability problem.
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It's that simple. And I try not to do, you know,
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too much unnecessary measuring and I use my,
16:31
my eye a lot for expediency.
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So back to this high resolution coronal, uh, T one weighted image.
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Uh, this is the workhorse for the wrist.
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We see the scapholunate and lu NATO triquetral ligaments.
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Here is the rather, um,
16:49
trapezoidal shape of the triangular fibrocartilage.
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It looks triangular in the axial projection. Here are the radial attachments.
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No, that's not a tear. That is radial highline cartilage.
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That is ulnar hiim cartilage and synovium.
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There is the foveal attachment right there. There's the styloid attachment.
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And right there is one of the distal attachments known as the ulnar carpal
17:13
attachment. These wispy structures represent the ulnar collateral ligament,
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not a true ligament, but a condensation of the ulnar capsule.
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Let's move on now and discuss in greater detail.
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One of the most important structures. The triangular fibrocartilage complex.
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The complex consists of the fibrocartilage like
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structure. Its radial attachment.
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The distal radial ulnar articulation,
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the tissues underneath between it and the hylan cartilage of the ulna.
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The hylan cartilage of the lunate, the cortex of the lunate.
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Here's the meniscus hoal analog. It's an artifact of preparation,
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but here's the small triangular shape lu NATO triquetral ligament and
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this vascular pedicle here right in the middle is known as the ligamentum crewe.
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And just distal to the ulnar styloid is the pre styloid recess.
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Here's the mr that matches that T one on the left. Um,
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water weighted on the right there is the radial highline cartilage.
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There is its attachment. There's the distal radial ulnar articulation,
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mostly collapsed. Tiny bit of fluid inside it.
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There is the synovium and hylan cartilage of the ulna that of
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the lunate, the lu NATO triquetral ligament.
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The meniscus holo styloid attachments,
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which are pretty broad foveal attachments, which are a little bit more narrow.
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And there's the hyperintense ligamentum tum with the barely seen,
18:48
uh, pre styloid recess,
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still even higher resolution. Uh, at the level of the radius.
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There's the radial attachment. Again,
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do not confuse the radial cartilage with a vertical tear.
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Most tears are gonna be in the central or inner third of the triangular
19:06
fibrocartilage. There's a little bit of synovium and cartilage of the ulna.
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And the same for the lunate.
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Headed over towards the ulna meniscus hoog peripherally.
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Then here we are, uh, again,
19:20
higher resolution drilling down into the ulnar styloid.
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There's the pre styloid recess that has a few different variations that are a
19:29
bit beyond the scope of our discussion today here,
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highlighted by our white arrow anatomy. On the left, Mr.
19:36
On the right there is the foveal attachment.
19:39
And here's the broad styloid attachment with a hyperintense
19:43
central vascular pedicle. The ligamentum crew attom.
19:51
Here again, high resolution still.
19:53
We once again see the triangular fibrocartilage radial
19:58
attachment, which is pretty broad peripheral attachment to the meniscus
20:03
hoog. Here's a foveal attachment,
20:06
not showing you the the styloid attachment in this case.
20:09
But here's another really interesting structure right here from the triangular
20:14
fibro cartilage.
20:15
There's this subtle structure going to the lu NATO triquetral ligament that is
20:20
known as the ulnocarpal ligament.
20:22
There's a similar ligament that goes to the lunate, the ulnolunate ligament.
20:26
And there's one that goes to the triquetrum, the ul, no triquetral ligament.
20:30
Neither of those latter two are shown at this juncture.
20:34
But here is a more peripheral slice.
20:36
So here's a sagittal slice done out here where
20:41
we're more aptt to see structures that attach the T F
20:46
C to the triquetrum. Let's have a look.
20:48
We're all the way out to the ulnar side of the wrist. We've got some basic,
20:53
basic anatomic drawings here,
20:57
showing you the vola aspect of the wrist right here in the sagittal.
21:01
Let's focus on this. Here's the triquetrum. Here's the triquetrum.
21:06
Here is the triangular fibrocartilage with its attachment to the Palmer radio
21:11
ulnar ligament.
21:12
And its distal attachment to the ul no triquetral attachment
21:17
right here. There's one dorsally,
21:20
a dorsal ulnar triquetral attachment.
21:23
And it blends with the dorsal radial ulnar ligament.
21:27
So you see the triangular fibrocartilage is tethered anterior.
21:31
It's tethered posterior. It's tethered medially. It's tethered laterally.
21:35
It's tethered distally and it's also tethered proximally.
21:39
So it's a rather complex structure that is kind of floating
21:44
like a trampoline supported by any one of a number of of structures.
21:50
Now let's talk about the, let's talk about the uh,
21:55
triangular fibrocartilage classification of injuries.
21:59
Now I'm not a big classification person.
22:01
I mean there's so many classifications for fractures.
22:04
You could go absolutely bonkers trying to learn them all.
22:06
But there are certain classification systems that our colleagues like they rely
22:11
on it, it's in their comfort zone, therefore you should use it.
22:15
And who are those clinicians? Hand surgeons.
22:19
So if you're playing to a general orthopedic surgeon, probably not necessary,
22:24
certainly not to a family doctor, but to a hand surgeon. Gotta know this.
22:28
So class one a central perforation,
22:31
B peripheral tear, and with or without a styloid fracture.
22:36
C involvement of the distal and sometimes proximal attachments.
22:41
And D radial uls, which is quite rare.
22:44
Class one refers to traumatic injuries of the triangular fibrocartilage.
22:49
You'll see that class two refers to all no lunate abutment or chronic injuries
22:54
of the triangular fibrocartilage complex.
22:58
So here is a central third perforation,
23:01
the most common type of traumatic injury of the T F C.
23:05
These are treated conservatively.
23:06
They're too small to put a stitch in so you don't operate on them. Um,
23:11
you rest them a little bit and they usually will granulate in a little fluid in
23:14
the distal radial ulnar articulation.
23:17
We would call this a polymer one A, a central perforation.
23:22
And the clinician would immediately understand what you're talking about.
23:25
Here is our polymer one A. In the sagittal projection,
23:28
there are dorsal attachments to the dorsal radial ulnar ligament.
23:33
Our volar attachment is right here. We don't see our uh uh, vola all,
23:38
no triquetral attachment very well.
23:39
But we will later in another case there is our hourglass shape.
23:44
T F C. There's our tear, our vertical tear not so vertical.
23:48
It's a little bit oblique in the sagittal.
23:49
It's kind of like looking at menisci of the knee.
23:52
And here is our triangular shape right there of our T F C.
23:57
We're missing this part of the triangle right here 'cause it's torn,
24:00
allowing fluid to exit from the distal radio ulnar joint into the
24:05
vola recess.
24:08
Here's another example of a traumatic T F C tear.
24:12
This time we are not involving the central third of the T F C.
24:16
We're involving the periphery. Let's have a look for,
24:19
if he doesn't look too bad,
24:20
there's the meniscus holo and it is blending with the ulnar
24:25
triquetral ligament. There's the LT ligament,
24:29
the attachment of the T F C to the LT ligament.
24:33
And as we keep going, look at what happens. The periphery turns to mush.
24:38
We don't see a strong dark band like or
24:43
fan like attachment to the ulnar styloid nor to the fovea.
24:47
It is detached. This high signal intensity material is edema.
24:52
In the ligamentum tum,
24:53
there's swelling of the presty recess and you can see some of the
24:58
attachments dorsally right here in the sagittal projection.
25:02
And there is a vola attachment right there.
25:05
So this is a peripheral T F C tear one B,
25:10
there's another one. This is, this one is a bit more central.
25:13
I promised I would show you the,
25:16
the uh o no triquetral ligament. There is a central tear,
25:20
but there's also a peripheral tear too. It's a little bit swollen out here.
25:25
Now if you go back and remember the,
25:26
the one that I showed you earlier on was a bit thinner.
25:30
It wasn't so blurry looking. Here's your T F C,
25:33
here's some intrinsic tearing of the T F C,
25:36
but there is your fat chubby uh, uh,
25:40
ligament that goes to the triquetrum, uh,
25:43
as part of the T F C peripheral distal stabilization. And here it is again.
25:48
Look how fat that thing is.
25:50
So the patient has both a central injury and a
25:54
peripheral slash distal injury. Let's talk about variance.
25:59
Now,
26:02
variance is the relationship between the ulnar platform and the
26:06
radial platform.
26:08
So if you take this crux right here between the styloid and the body
26:13
and you compare it to the free edge of the radius,
26:16
it should be about eight millimeters. Either way within that line,
26:20
if it's too far forward, positive ulnar variance, if it's too far back,
26:24
negative ulnar variance.
26:26
Here's an example of somebody with negative ulnar variance.
26:30
You're more than a centimeter proximal to the free edge of the
26:35
radius. What happens to these people? Unbeknownst to many of you,
26:40
they have a high incidence of extensor carpe narrows injuries.
26:44
They have a high incidence of peripheral,
26:47
not central peripheral T F C C injuries.
26:51
And they also have this a board question.
26:53
An increased incidence of keen box disease or lunate necrosis positive
26:58
impaction syndrome may impact the lunate, may impact the T F C,
27:03
the ate triquetral ligament.
27:04
And you can even get styloid impaction on the triquetrum.
27:10
Here is an example of the classification system for these types of
27:15
impaction.
27:17
This is the Palmar class two system thinning of the T F C due to
27:22
wear a b ludo or UL chondromalacia
27:27
C perforation of the central T F C D
27:31
dreaded tearing of the ludo triquetral ligament and E generalized
27:36
carpal arthritis. Let's have a look. This is an easy one.
27:40
The patient has had a very serious complex bridged fracture
27:44
of the radius. There is for shortening.
27:48
The fovea of the scaphoid right here is destroyed.
27:52
There's some arthritis developing in the lunate, but,
27:55
but the money is over here where the ulna is jutting way
27:59
forward relative to the free edge of the radius.
28:02
It has just destroyed the triangular fibrocartilage.
28:07
It's hard to see a triangular shaped LT ligament. It's torn.
28:11
And there's extensive irregularity in the periphery of the T F
28:16
C C severe end stage abutment in acquired
28:21
positive ulnar variants from a prior radial fracture. Here's another one.
28:26
This time we are not impacting the central third so much
28:31
of the T F C, even though it is a bit attenuated. So there's some disease here,
28:36
but I am showing it for this.
28:38
This is a piece of the ulnar styloid that has broken off that in ulnar
28:42
deviation is getting slammed into the triquetrum.
28:46
Now normally there is a little indentation here of the trium,
28:49
so you don't want to confuse that with an O C D.
28:53
But when you see this object fitting in the indentation and then you have edema
28:58
deep to the indentation,
28:59
you know that you are impacting the structure against the triquetrum.
29:03
And the patient has also torn their ulnar capsule.
29:08
There's another example of a TF CCC injury this time.
29:13
Not positive ulnar variants, but negative ulnar variants. Now, you know,
29:18
kBox disease, we don't have that in this case,
29:21
but what do we have an increased risk for extensor cario narrows injuries and
29:27
peripheral T F C C terrace. Let's have a look.
29:31
Negative ulnar variance. There's our ulnar styloid,
29:34
there's our body and the crotch or crux between the styloid and the
29:39
body way proximal to to the radius. There's our T F C.
29:43
It does not land on the styloid,
29:45
it does not land on the fovea.
29:49
It does not have a clean attachment to the distal lu NATO triquetral
29:54
ligament. And we never really identify a distal,
29:58
uh, attachment to the triquetrum. Right here we do see a proximal one,
30:02
but not a distal one. And look posteriorly, posteriorly,
30:06
there's no attachments to the dorsal radial, the ligament.
30:09
It's just mashed potato swelling of the posterior aspect of the wrist.
30:14
So we have a dorsal attachment problem, a peripheral attachment problem,
30:19
all associated with negative ulnar variants. And we better check,
30:22
we're not doing it yet, but we better check the E C U,
30:25
which we would do as the next step.
30:28
Let's turn our attention back to the extensor tendons. Remember,
30:31
we've got longest brevis longest brevis longest, and then the digitorum,
30:35
the digit mini and the e c u.
30:38
Another important structure in this projection is lister's tubercle for
30:42
fractures, irregularities, and deformities of lister's.
30:45
Tubercle may lead to contraction of the extensor reticulum.
30:50
And as the E P L crosses over here to meet
30:55
the thumb, these two structures,
30:57
compartment two and three may friction over one another may rub
31:02
together and give you what's known as distal intersection syndrome.
31:07
So let's talk about intersection syndrome.
31:10
I'm not gonna show you that intersection syndrome.
31:13
I'm gonna show you a couple of others. And, um,
31:17
but let's start out with the,
31:18
the most common intersection syndrome in younger individuals. Sorry,
31:23
the most common extensor tendon that is affected in,
31:27
in in younger individuals, not intersection syndrome.
31:31
And that is below age 80.
31:32
And that is the E C U that is affected more frequently in people with ulnar
31:37
variants. Compartment number one,
31:40
the abductor lysis longus and extensor lysis. Previs, which you know,
31:43
as the Devan compartment, uh, has a crossing of the,
31:48
the two structures. And so that is a form of intersection syndrome,
31:52
namely devans disease.
31:55
And you do get contraction of the reticulum.
31:58
That leads to s stenosing, uh, teno synovitis.
32:02
And then there's another intersection syndrome that I'll show you a little bit
32:05
later very briefly,
32:07
that occurs in the arm between the extensor lysis long longest and
32:12
the extensor carp radialis, similar to that in the hand.
32:15
So three intersection syndromes, one in the forearm,
32:19
one in the dequeant compartment,
32:21
and one in the hand between compartments two and three. And as we said,
32:26
fractures of lister's, tubercle, and deformities. As such,
32:30
put the patient at risk for intersection syndrome.
32:33
So here's an example of dequeant compartment number one
32:38
intersection syndrome where there is marked
32:42
hypertrophic irregularity and proliferation of soft tissue and even
32:48
synovial tissue. And remember,
32:50
these structures have multiple tendon slips,
32:54
so it's very common to over-diagnose interstitial laminar tears
32:59
of the first compartment.
33:00
So you have to be very careful about that diagnosis and use the axial projection
33:05
with, with a, a very discreet,
33:08
near full depth area of signal to make the diagnosis of a tear as opposed to
33:12
hypertrophic deforming S stenosing 10 synovitis,
33:17
which is a subset of intersection syndrome.
33:20
There's also an erosion of the radial syl more on that a bit later.
33:24
Here we are in the short axis view.
33:26
There is so much inflammatory tissue from this crossover
33:30
intersection,
33:32
S stenosing 10 synovitis that you can hardly see the tendons.
33:36
You see them very as very, very tiny dark little structures.
33:39
But remember there are multiple tendon slips here and this seeming
33:44
discontinuity does not in itself mean that there is a tear.
33:48
Now this is describing the anatomy of the the crossover syndrome.
33:53
This is gonna take a little too much time.
33:55
I just wanna point out to you that in the forearm there is a third intersection
34:00
syndrome, the proximal type. And this is what it looks like. And,
34:05
and I'm not gonna talk too much about that today.
34:07
I just wanted you to see that there are multiple intersection syndromes and as
34:11
you get more distal into the wrist, it persists.
34:14
So not only does this patient have it in the upper arm,
34:17
they also have it in the hand right there.
34:20
Look at the swelling around the E P L and the extensor
34:24
Carpe radialis longest and brevis.
34:27
Those three together are showing you that this patient has both proximal and
34:32
distal crossover or intersection syndrome. Three types.
34:37
And you've seen all three. The extensor carpi narrows,
34:40
we said this is the most common extensor tendon to be affected in young
34:45
individuals. The most common in elderly individuals is the first,
34:49
the Devan compartment, the E C U inserts on the base of the fifth.
34:53
It's held in place by a sub sheath, not a Rett aum.
34:56
A sub sheath over top of that is the extensor reticulum.
35:01
And there are stabilizing fi fibers of the lineage.
35:03
Ag got which extend from the base of the ulnar styloid to the
35:08
extensor reac subs sheath injuries,
35:11
even with an intact extensor reticulum often lead to subluxation.
35:16
So here's a diagram showing you the E C U in its groove.
35:19
I allow a fair amount of latitude to the E C U as long as I don't see swelling
35:24
there,
35:24
I don't mind if it pers on the YL as long as I can see this or I have
35:29
no swelling and no focal high signal in the
35:34
tendon itself.
35:36
Now don't confuse the extensor Retin ulu with the sub sheath.
35:40
If it's perched and I have swelling and I have a sub sheath that's interrupted,
35:45
I get worried and I'm going to call it out.
35:49
So here we are with four consecutive views.
35:52
This is the E C U part one, there's part two,
35:56
there's no two parts to the E C U.
35:58
It is split much like you would see a split of the perineal brevis
36:04
in the foot. There is the subs sheath right there.
36:08
That's the reac on top of that. Yes, very, very subtle teased out findings.
36:13
There is the interruption of the sub sheet that has allowed the E
36:18
C U to split and portions of it to dislocate over top of the ulnar
36:23
styloid. Here's some other examples of E C U disease.
36:27
Now I'm not gonna get into tendons per se 'cause we're gonna talk a lot about
36:31
that in the September course with Dr Doctors Resnick and Chang and colleagues.
36:36
But you know, there's peritendinitis, there's peritonitis, there's tendinopathy,
36:41
there's tendinosis, there's tendonitis,
36:43
and we're gonna winnow those out for you at at a later date.
36:47
But right now I wanna show you an example of teno synovitis to make that
36:51
diagnosis. Your structure has to have synovium. For instance,
36:55
the Achilles know synovium.
36:58
So you don't use the term teno synovitis.
37:01
You might in the proper setting use the term para tendonitis more about that on
37:05
another day.
37:06
But here we have tenino synovitis with tissue that is not simple fluid
37:11
that is synovial hypertrophy and fluid.
37:15
That is proac surrounding the E C U in this patient with
37:19
ra. There's another example of an E C U problem. Uh,
37:24
this patient has ruptured the E C U at the base of the fifth.
37:27
That was a low field image.
37:28
As a higher field image is a patient that is a pitcher that is complaining
37:33
vehemently about his on the wrist. And he is got this tiny little tear,
37:37
which probably wouldn't bother me because I'm not throwing a ball 90 miles an
37:41
hour. But this person is,
37:42
it's a small interstitial tear of the triangular fibrocartilage.
37:46
Now let's turn our attention to instability and look at a hand surgeon's,
37:51
uh, classification of instability.
37:54
We've got acute less than a week,
37:56
subacute one to six weeks and chronic greater than six weeks.
38:02
Then we've also got what's known as constancy. Pre dynamic.
38:05
There's no instability from mal alignment on imaging only
38:10
symptoms. Dynamic mal alignment is only demonstrated with stress views.
38:16
Radial ulnar, radial deviation, ulnar deviation, pronation, supination,
38:20
clenched fist view, and then static.
38:22
There's permanent mal alignment seen in the neutral position on a standard
38:27
M R I. Let's look at some anatomy here. If we can get our uh,
38:32
video working. I don't think we're gonna get it working. Click, click it.
38:36
All right, there we go. Thank you. So this is a high resolution image.
38:40
I wanna just let you toggle through it and I'm gonna stop it right here.
38:45
Let's see if I can back it up. There is your radial collateral ligament.
38:49
Let's back it up a little bit more.
38:53
And there are some of your extrinsic ligaments.
38:57
Look at this extrinsic ligament right here. The radio scavo capitate ligament.
39:02
It's a long lar ligament. Here's a short lar ligament.
39:07
The short lar ligament is known as the arcuate ligament.
39:12
That's gonna be important. Uh,
39:14
along with this other ligament here in patients with vola and turchary segmental
39:18
instability.
39:19
This one is important in rotatory subluxation of scaphoid.
39:23
The one right underneath it, let's see it right here.
39:27
The one right underneath it is known as the long radial lunate
39:32
or radio ludo triquetral ligament.
39:35
Now I'm not gonna get you too involved in the extrinsic,
39:37
so you don't pull your hair outta your head.
39:39
I wanna show you one dorsal extrinsic, but before I do the vola extrinsics,
39:44
make an inverted V. You've got some long vs and some short vs.
39:48
I'm gonna break that down for you in a few moments.
39:51
But just remember inverted V,
39:54
there's also a weak spot right here called Corona space where none of the
39:58
vs really provide a lot of support.
40:01
So the carpus can come at you and can say proximally.
40:05
Very important biomechanical concept because my thing as opposed
40:10
to anatomy is biomechanics.
40:12
Some of my colleagues are more anatomically oriented.
40:15
I'm more biomechanically oriented.
40:18
So now let's keep going and go to the dorsal aspect of the wrist. Here we come.
40:24
I'm just gonna show you one ligament, so don't get scared. Here it comes
40:31
right here. Whoops. Oh no, shouldn't have clicked it. Hold on.
40:35
I got it right here.
40:41
Right there. This is the dorsal intercarpal ligament. Yeah,
40:45
there's some other ligaments proximal to it too,
40:47
but this is one that gets injured when you fall on an outstretched hand.
40:54
Now let's look at some of the intrinsics. Those are some extrinsics.
40:58
Let's look at the intrinsics. The intrinsics include the scapholunate ligament,
41:03
which we see here. I have a little mnemonic.
41:06
L T V Ss L V ss l v.
41:09
The sca scapholunate, uh, sorry, L T V.
41:13
The lu NATO triquetral ligament is stronger on the volar side. S L D,
41:19
the scapholunate ligament is stronger on the dorsal side.
41:23
So the dorsal portion of the s l ligament is more band like.
41:27
The middle is triangular and sometimes it'll have a little cleft in it.
41:31
That's okay.
41:32
I also don't mind this little cleft in the lu NATO triquetral ligament as long
41:36
as this space looks proper. And then as we get into the vola aspect,
41:41
this is a bit weaker.
41:43
The SL ligament is kind of trapezoidal in shape and here are some diagrams
41:47
to reflect just that.
41:51
Now let's look at the Ludo triquetral ligament that likes to honor the
41:55
great artist Salvador Dolly with Salvador Dolly's bar shaped
42:01
mustache.
42:02
Remember we said the triangular fibrocartilage has carpal
42:07
attachments, some to the lunate.
42:11
We can barely see one right here.
42:13
Some to the Ludo triquetral ligament known as the Ulnocarpal
42:18
attachments. There's one right there.
42:20
And then we also showed you earlier some to the trium.
42:24
There's the base of one right there.
42:27
All ulnocarpal attach. But look at our LT ligament.
42:32
It's a triangular nubbin, it's a broad triangle.
42:36
It's a crisp triangle with a little bit of a mustache. It's eccentric.
42:40
It's now got a bilateral bar shaped mustache. This is all, uh,
42:45
imaging taken from the same patient. So look at the variability.
42:50
You can see why some people are uncomfortable diagnosing LT
42:55
ligament tears without an arthrogram. I however am not,
42:59
I'm used to all these variations and I use the secondary signs,
43:03
the absence of arthritis, no fluid, a collapsed capsule.
43:07
Perfect hi in cartilage.
43:09
I am totally comfortable with this LT ligament without putting contrast in the
43:14
joint. Let's look at some intrinsic failure.
43:18
Let's look at SS scfo, lunate degeneration and tears. Widening synovitis,
43:22
pseudocyst erosions. You may get rotation of the lunate.
43:26
You may get dynamic changes on radial deviation and ulnar deviation,
43:30
which you've seen already.
43:32
And you might get the capitate migrating approximately.
43:36
And lar, if the patient develops a more complex pattern of instability,
43:40
this one's easy. You didn't need an arthrogram,
43:43
we did it to appease the clinician.
43:46
You could drive a a Buick through this giant hole between
43:51
the scaphoid and the lunate. There's the floating scapholunate ligament.
43:56
This is the membranous mid triangular portion,
43:59
but the whole thing was torn from front to back,
44:03
yet there is no rotation or displacement of the lunate.
44:07
And there wasn't any rotatory displacement of the scaphoid either.
44:11
Here's one that's a bit more subtle.
44:14
I do not need to give contrast in a case like this. Uh, I already know that the,
44:18
the s sl ligament is torn. I might do radial and lar deviation,
44:23
but look at the difference between this and this.
44:26
Now if I have to know the percent tear, then I might go for the arthrogram.
44:31
But that is indeed a rare event.
44:33
And if you look at all the slices at very high resolution,
44:37
especially with radiant and ulnar deviation, you will be able to tease this out.
44:41
But it is the secondary sign right here of widening and swelling compared to the
44:46
LT interval that makes the case for you very easily. Here's another one.
44:51
We do see a sick looking but present SL ligament.
44:55
It's irregularly shaped on the T one.
44:57
It's a little better shaped on the PD spur,
45:00
but we know something nasty is going on here because we have arthritis,
45:04
arthritis there and a little bit of arthritis here.
45:06
So right at that intersection we've got micro instability.
45:10
So in a case like this,
45:11
if my radial and ulnar deviation fails to show the widening that I'm
45:16
anticipating,
45:17
then this might be one of those cases where we would inject and perform an
45:22
MR arthrogram case like this. Not by the way, in that last case we did,
45:27
this was not a full thickness tear. There was no communication.
45:31
It was just a stretched,
45:33
insufficient ligament that allowed for micro instability.
45:37
Here's one that's obvious. Uh, there's a huge defect here.
45:41
The so-called Terry Thomas sign with a space in the
45:45
incisor tooths, uh, of the front of the mouth right there. There's the lunate,
45:49
there's the scaphoid, and then the axial,
45:52
which shows you the scapholunate ligament,
45:55
which was here and is now fallen into this hole. There it is.
46:00
It's trapped inside between the scaphoid and the
46:05
lunate. So this is one that's going to need a surgical extraction.
46:09
Radial ulnar failure.
46:11
This happens when you have insufficiency of the volar,
46:14
less commonly the dorsal radial ulnar uh, ligament.
46:18
It allows for excessive excursion of the ulna related to the
46:23
radius. You put a a.in the middle of the ulna, a.in the middle of the radius,
46:28
and you should stay within about five to eight millimeters of the central dots
46:32
of both when you go into steep croation and, and supination.
46:37
But there's going to be some movement and comparison with the other side is
46:41
important. Now, when you're looking at these spaces,
46:45
a knowledge of their communication is important.
46:48
And we will drill into that at our combined course in September.
46:53
Um, the radio ulnar bursa communicates with a horse shoe shaped hand bursa
46:58
about 70% of the time. There are other bursa in the wrist. There's Athena bursa,
47:02
there's a mid carpal bursa, there's a flexor lysis longus bursa.
47:06
And those will be stories four another day.
47:10
But here's an axial diagram and an axial M r I look at that dorsal
47:15
floating ulna.
47:18
There's a little bit of the triangular fibrocartilage.
47:21
We can't see the volar ligament because it's a T one weighted image.
47:24
You'll see it in a minute.
47:26
But look at how dorsally displaced the ulna is and there's a stubby
47:31
stump of the dorsal attachment of the T F C to the
47:36
triquetrum right there. It just ends pretty suddenly.
47:39
So it's a rather complex case,
47:40
but I'm showing it for the vola radial ulnar ligament tear.
47:44
There's one end of the vola radial ligament.
47:46
There's the fray destroyed ola radio ulnar ligament.
47:50
There's the dorsal floating ulna.
47:53
And because of the stresses it puts and the stretch on the
47:58
extensor support of the E C U,
48:00
the E C U is now starting to plow its way through the sub sheath and
48:05
ret. So everything is really a, is connected to B, connected to C.
48:10
And if you know what you're looking for, you're more likely to to find it.
48:14
Here's a patient with radio ulnar instability for years.
48:18
Look at the widening of the radio ulnar articulation and then the sagittal
48:22
projection. You'd make this diagnosis on a plane film,
48:25
but many people do miss it.
48:26
Look at the dorsal displacement of the ulna and look at the very
48:31
irregular chopped up appearance of the dorsal aspect of the T F
48:36
C. Here. It looks pretty good other than being thinned,
48:38
it's very thin here it looks a little irregular,
48:40
but here it's just attenuated dorsally.
48:44
So the dorsal attachments are gone. Here's yet another one.
48:48
This is a patient without a lot of displacement of the ulna,
48:51
but with severe chronic longstanding wrist pain.
48:54
It is an athlete look at the fluid and the distal radial ulnar articulation.
48:58
And there's one end, one end,
49:01
and there's the other end of the normally connected volar radial ulnar
49:06
ligament. There's the defect right there. So volar, radial ulnar ligament, uh,
49:11
rupture with, uh, dynamic radial ulnar instability.
49:16
So on the static it didn't look unstable,
49:18
but on the dynamic that thing floated all over the place. Extrinsics,
49:23
we've already talked about some of the key extrinsics.
49:26
One is the radios scfo capitate ligament.
49:30
The other is the long radio NATO triquetral ligament.
49:34
We're just gonna focus on this one today.
49:36
We look at it all the time when we have complex instabilities.
49:41
And those are really the two. You should concentrate on these short ligaments.
49:45
I'm not so interested in this short ligament from the hammit to the
49:50
capitate. I'm not so interested in you learning about it,
49:53
but just know that this is one of the divisions of the arcuate ligament that
49:58
helps support the center of the mid carpal space and prevents the capitate from
50:03
coming at you and migrating proximally,
50:06
especially in especially in vola, in ary segmental instability.
50:11
So this is your big one here. This is primary. This one is your,
50:14
your secondary area of interest. And in the volar wrist,
50:19
the ligaments make an inverted v. So here's a little bit, uh,
50:23
of drilling down into the extrinsics. Again,
50:27
here is our radios scfo capitate uh, ligament.
50:31
Here's another long ligament that also supports the ulnar aspect of the wrist.
50:36
Here's that short amato capitate ligament.
50:40
And together these will be disrupted in patients with visi and
50:45
in ulnar sighted wrist clunk in dorsal ary segmental
50:49
instability. Uh,
50:50
these may be compromised along with the scapholunate ligament.
50:57
And here are two short ligaments known as the arcuate ligaments.
51:00
When these tear, this is going to allow for a complex instability. Again,
51:05
the capitate is gonna migrate proximal and it's gonna come at you and may
51:09
lead to volar and intercalary segmental instability.
51:12
Here are these short arcuate ligaments in the mid carpal space,
51:17
deep to the carpal tunnel space.
51:19
And when these structures start to sag anteriorly,
51:22
they can compromise the median nerve. Here, uh,
51:26
here are the dorsal extrinsics. They make a sideways V.
51:30
And the one I'm most interested in is the dorsal intercarpal ligament.
51:35
The upper limb of the V or that one gets injured when you fall on an
51:39
outstretched hand. And here it is fall on an outstretched hand.
51:43
The patient has bled into the dorsal intercarpal ligament.
51:47
The treatment is completely conservative, violent, complex,
51:51
carpal instabilities.
51:53
Let's start out with dorsal and ary segmental instability.
51:57
While my pen isn't working,
51:59
you can see that there is a straight alignment between the metacarpal,
52:03
the capitate, the lunate, and the radius.
52:06
If the lunate starts to turn dorsally facing,
52:10
we call that dorsal and ary segmental instability.
52:15
It may or may not be associated with rupture of the radios
52:20
scfo capitate ligament. Here's a normal radios scfo capitate ligament.
52:25
Here's a patient with dorsal and turchary segmental instability.
52:29
Know the radios scfo capitate ligament is not obviously torn on this image,
52:34
but I just wanna show you the dorsal facing lunate.
52:37
It is highly unlikely that this patient is going to have an intact
52:42
scapholunate ligament even before looking.
52:45
There's another example of a normal reference.
52:48
Here's our straight alignment between metacarpal, capitate, lunate and radius.
52:53
And our scaphoid is gonna be at about a 60 degree angle.
52:58
I'll show you what I mean in a moment.
53:00
But let's turn our attention not to dorsal and intercalary segmental
53:05
instability. A dorsal facing lunate, but a vola facing lunate. Yeah,
53:10
we're vola because there are the flexor digitorum sublimes and
53:15
profundus tendons.
53:16
There is your median nerve volume average you are facing Palmer.
53:21
And look at the mid carpal space, it is destroyed.
53:24
Looking at a capitate is starting to work its way proximal and anterior
53:30
on its way to producing secondary carpal tunnel syndrome in a
53:35
patient with visi position or visi posture and severe
53:39
injury of the lu NATO triquetral ligament and other intrinsics.
53:44
There's another complex instability SL ligament. No problem.
53:49
You don't need contrast for this. There's a giant hole here.
53:53
There are the two limbs of the ligament.
53:54
The LT ligament is absolutely positively intact
54:00
and the space between the trium and lu lunate is perfect.
54:05
Here's the sagittal of this patient.
54:07
The lunate is starting to face dorsally and the radios
54:12
cafo palpitate ligament is starting to de-laminate right there.
54:16
It is not allowed the scaphoid to rotator sag yet,
54:20
but it will coming to a theater near you very shortly.
54:24
So a little bit of extrinsic delamination, some dizzy,
54:29
a big sl ligament tear, but no rotatory subluxation of escape for.
54:34
Finally we finished with scapholunate advanced collapse and some more advanced
54:39
stability. This is also known as slack wrist.
54:43
You are looking for proximal capitate migration. The,
54:47
the lunate may migrate to the ulnar side,
54:49
so-called ulnar translocation of the lunate. As you've seen,
54:52
there is extensive arthritis,
54:55
one of the earliest signs and a stage one of slack wrist radial
54:59
styloid hypertrophy.
55:02
You can get secondary av n of the lunate scaphoid rotation,
55:07
destruction of the scaphoid, and then lunate fossa of the radius.
55:11
And then finally, carpal tunnel syndrome.
55:14
Here is the Watson classification or a modification of it
55:19
showing the four stages of slack wrist degenerative changes only in the scap
55:24
styloid tip easy two involvement of the scaphoid phos
55:29
of the radius.
55:30
Three involvement of the lunate phos of the radius and four involvement of
55:35
the wrist in its entirety.
55:38
Let's have a look at a slack one. Sorry, slack two wrist.
55:44
Why is it a two?
55:45
It's a two because there is some styloid involvement right there.
55:49
Look at how pointy it is. That's one of the earliest signs of slack wrist.
55:53
A pointed radial styloid, yes, an obvious SL ligament tear.
55:58
Yes, there is some osteoarthritis and some erosions, but why is it not a one?
56:03
Why is it a two?
56:04
Because there is marked narrowing of the scaphoid fossa cartilage
56:09
where the scaphoid sits in the radius. Look at the gradient echo,
56:12
almost bone to bone. There's still some cartilage here,
56:15
but there's no cartilage here.
56:17
So styloid plus radial fossa,
56:21
stage two slack wrist with scapholunate ligament rupture.
56:26
And to make matters worse,
56:28
the radio scavo capitate ligament ruptured. There it is right there horn.
56:33
And the, the scaphoid is now rotating in a clockwise fashion.
56:38
No longer do you have, uh,
56:41
60 degrees of angulation between the scaphoid and a vertical
56:46
line drawn in this fashion. It's almost horizontally oriented.
56:50
So this patient has a complex pattern of instability that involved
56:55
rupture of the radios scfo capitate ligament.
56:58
Here's another one that's very complex. The SL ligament is destroyed.
57:03
The lunate is now translocating to the ulnar side, just as we said it would.
57:08
In late stage slack wrist,
57:10
the hamate and the capitate are migrating proximally and getting destroyed
57:15
at their base. They're also migrating into a ventral position,
57:20
likely to encroach on the median nerve.
57:24
The T f C has been destroyed.
57:27
The lt ligament has been destroyed and there is rotatory
57:31
malalignment between the lunate and the trium.
57:35
So the intrinsics are completely wiped out.
57:39
If we look at the lunate, it is lar facing, it's facing this way.
57:44
So the patient also has visi. Now this time we have a stage three slack wrist.
57:49
Why is it stage three? Because we have the radial tip involved, not shown.
57:54
We have the radial fossa involved. I didn't show it quite as well as I,
57:59
I might've liked, but now the lunate fossa is involved right there.
58:03
Extensive erosive change of the lunate fossa with a small
58:07
erosion and cyst or pseudo cyst that that is proliferating.
58:12
So now with lunate fossa involvement,
58:14
where at stage three the whole wrist takes you to stage four.
58:17
And here we are at stage four in this patient with
58:22
a p****n cousin of slack wrist. The snack wrist,
58:27
no, it's not Frito lace snacks,
58:29
it is scaphoid non-union advanced collapse.
58:33
This is one proximal fragment of the scaphoid. There's the other fragment.
58:38
So this joint is now serving as a ligament where there is widening and
58:43
instability,
58:44
there is extensive erosion of the scaphoid fossa.
58:49
There is a pointed radial styloid not shown there was involvement of the
58:53
lunate fossa,
58:54
but look at that generalized degeneration of the entire carpus with a
58:59
dorsal facing lunate with osteoarthritic spurs.
59:03
Stage four slack wrist with dorsal and ary segmental
59:07
instability and with a fracture really making it a snack wrist
59:12
rather than a slack wrist. And here it is on the water weighted image.
59:17
Look at that massive proximal capitate migration.
59:21
Ulnar translocation o o of the lunate.
59:24
And now you cannot even see the median nerve. It's right over here.
59:29
It's this flat,
59:30
tiny little pancake that is compressed by the lar
59:35
displacement of the capitate and hammid.
59:39
And look at the thenar eminence. Hypo thenar looks fine.
59:42
The patient virtually has no thenar eminence.
59:45
So end stage carpal tunnel syndrome from end stage class four slack
59:50
wrist. So that concludes our,
59:52
our discussion today of taken you through some basic
59:57
anatomy, some basic tenets of, uh,
60:03
selective imaging sequences like radial and ulnar deviation, pronation,
60:08
supination compound, uh, scaphoid views.
60:11
I took you through the details of the triangular fibrocartilage,
60:15
the Palmer one traumatic classification system,
60:18
the polymer two abutment classification system.
60:21
Then we looked at some intrinsic and extrinsic ligaments and finished with
60:26
some very complex instabilities of the wrist. And with that,
60:29
I'll take some questions.
60:38
All right? Yes. Thank you, uh, for sharing your lecture today, Dr. P Um,
60:42
at this time we open the floor for any questions from our audience. Uh,
60:45
you can submit your questions to Dr. Pomerantz through the q and A feature.
60:50
Uh, Dr. Pomerance,
60:51
would you like me to do my best to redo the questions or can you see them?
60:55
I can see them.
60:56
So U C L is considered part of the T F C or not the TF
61:01
T F C C or not. T F C C is kind of a wastebasket, so everything goes in there.
61:07
So the ulnar collateral ligament is in most circles considered part of the the T
61:12
F C C.
61:13
It's not a critically important structure since it doesn't provide a lot of
61:16
instability. It's usually used as an indirect sign of other things,
61:21
uh, that are happening such as the case that I showed you. So the answer is,
61:25
it is how reliable is various vari variance assessment on M r I.
61:30
Um, doesn't patient positioning affect this? It absolutely does.
61:36
You'll notice in my slides slides, I didn't say ulnar variants positive.
61:41
I said positive variance posture because hand surgeons are like
61:45
neurosurgeons, detail oriented, thank God O C D,
61:50
not to a fault they're O C D, thank God they are. So,
61:54
they are very specific about how they want their variants measured on
61:58
conventional radiography. And that is why I use the term posture. However,
62:03
you have an obligation to use a little bit of common sense.
62:06
So let's say you're more than eight millimeters distal to the radius with your
62:10
ulna, look at what's happening around you. If the T F C is thinned,
62:14
if there's fluid in the radial nerve articulation, if there is luon malacia,
62:19
you have an obligation to call out that ulnar positive variance
62:24
posture to protect yourself and say that the patient has secondary signs
62:29
of ulnar lunate abutment syndrome.
62:31
So I absolutely use the secondary signs to put myself on
62:36
sound footing as it relates to variants when dealing with hand surgeons
62:41
who have very strict criteria for such. Um,
62:45
which protocol would you recommend when evaluating a vitality of bone on M R I?
62:49
For instance,
62:50
in the case of keen box disease or scavo fracture is T one fat sat
62:55
before and after contrast injection sufficient,
62:58
or the only examination that can get the optimal and realistic results?
63:02
When we use profusion sequences? Well, first of all,
63:05
I wouldn't do profusion imaging if I have a uniform or nearly uniform
63:10
black slightly collapsed or markedly collapsed, you know, lunate.
63:15
Now if somebody, you know,
63:16
has a normal size lunate and it it's an indeterminate keen box case
63:21
or they're trying to determine how much is viable and how much is not viable,
63:27
which wouldn't be in a uniform black lunate,
63:30
then I will do dynamic contrast imaging,
63:33
just as I might do with say a breast m r i,
63:37
I'll do very fast fat suppression, gradient echo imaging and um,
63:42
you know, maybe a slice every three seconds or so. You don't have to be too,
63:45
too quick with it. And look at how the lunate peruses, how often do I do that?
63:50
Maybe two to three times a year.
63:52
I've done it a few times in the scaphoid as well, but, but it isn't,
63:56
it isn't standard practice for me, but that's the best way to do it,
64:01
kind of mimicking the dynamic breast protocol. Next, please.
64:05
Any other question? There's more right there.
64:08
How much physiologic fluid is there in the distal radial ulnar joint?
64:13
I allow a slit. What's a slit a millimeter of fluid.
64:17
There's gonna be some subjectivity there,
64:19
but it's gonna be a very ti a very tiny amount.
64:23
And it's also gonna depend on patient age. For instance,
64:26
if I have a 15 year old, I don't wanna see any fluid there.
64:29
If I have a 50 year old, I'll allow a millimeter of, you know,
64:32
lubricating fluid and, you know, potential overuse and, and so on. You know,
64:37
if, if I'm on the fence, I'm looking at everything else. I'm looking at the,
64:42
the vola and dorsal radio ulnar ligaments. I'm looking at the intrinsics,
64:45
I'm looking at the adjacent, uh,
64:48
radio ulnar cartilage using indirect signs to make that decision.
64:53
Next question. What is the significance of the space of Poirier? Well,
64:57
the space of Poirier is this sort of weakness that occurs between those short
65:02
lar blue ligaments that I drew for you that is kind of right in the middle,
65:06
just volar to the capitate. It is important 'cause it's an area of weakness,
65:11
and when you have these more advanced complex instabilities,
65:15
it will allow the capitate to come forward.
65:18
It'll allow the capitate to drop down and it can contribute to what you saw
65:23
at the end end stage.
65:25
Carpal tunnel syndrome orthopod tells you to look for ulnar collateral
65:30
ligament injury. Where to look for it. And is there any significance? Well,
65:34
I'm not sure, um,
65:36
an experienced hand surgeon would ever order an M R I for that purpose.
65:41
We all know that.
65:41
Do a lot of wrist imaging that the U C L is a flimsy structure.
65:46
It is used by us as an indirect sign of other problems, ret, macular stripping,
65:50
E C U disease and so on.
65:52
But the best place to look for it is where I showed you on higher resolution
65:57
coronal uh, imaging. And it doesn't necessarily matter which sequence,
66:01
although I, I, I see it best on a one to two millimeter fiesta sequence.
66:06
How reliable is T F C C interpretation on films or on scans done in other
66:11
places? Do you end up repeating such scans at your place?
66:15
That's a loaded question. You know, we are a, a tertiary referrals facility,
66:20
so we do get to see and resolve these usually without contrast.
66:25
And, um, MR is extremely reliable, extremely reliable.
66:30
I I hardly ever inject a risk to diagnose A T F C or
66:35
A T F C C tear.
66:37
The most common use of contrast for me is in an equivocal
66:41
LT ligament injury. And that, that is not often a next question, um,
66:47
about DIS and vsi. Uh,
66:49
are there standard angles to measure the position of the lunate and scaphoid and
66:54
capitate bones? There are. If you email me, I'll send you those angles. My,
66:59
my pen is not working. But as a general rule of thumb,
67:02
I like the scaphoid to have about a 45 to 60 degree position
67:07
relative to the vertical. So if I start to see the sca,
67:11
I dip below 45 degrees and start approaching the horizontal,
67:15
then I know I have rotatory displacement regarding DSI and vsi.
67:21
That's a little more easy. However, if the technologist puts the,
67:25
the hand in the scanner and they do this, they owner deviate,
67:29
you are going to create a disci posture appearance,
67:33
so-called pseudo disse.
67:36
So make sure that your wrist is absolutely straight and if
67:40
it is,
67:41
your lunate should be pointed straight up towards the capitate and straight up
67:45
towards the base of the third metacarpal. Um,
67:51
let's see. Question about the E C U.
67:54
Is the E C U part of the T F C C?
67:57
It is as is its sub sheet.
68:02
Alright, are there any other questions? 1.5 versus three T,
68:05
which one is preferable? They're both fine. Absolutely.
68:10
And you can scan with low field in the wrist because you can get the hand in the
68:14
center of the magnet bore. So if you can do the right sequences, stern,
68:18
then section gradient echo imaging, Sarge, you know,
68:22
one two millimeter slices.
68:24
You absolutely can image the wrist at low field as low as 0.18 Tesla.
68:30
Okay, I think I have answered all the questions.
68:34
Doesn't patient positioning affect the ability to assess dsi?
68:37
I think I answered that one. You absolutely need to have the risks straight.
68:41
If you owner deviate, you're gonna create pseudo dsi. If you radial deviate,
68:45
you're gonna create pseudo vsi. Alright,
68:50
I think that's about it.
68:51
Thank you for your thoughtful questions and I hope to see you all
68:56
in September for the combined, uh, Resnick,
69:00
Pomerance Chung and Colleagues course. Uh, we're looking forward to seeing you.
69:05
Have a great day.
69:06
Thank you so much again, Dr. Pomerance. Uh,
69:09
and thank you to everyone for your questions and participating in our noon
69:13
conference.
69:14
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69:29
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