Interactive Transcript
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What would be the best advice when describing the images
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used to evaluate patients
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after biological treatment of tendinopathies?
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Mainly partial ruptures.
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Taking into account
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that many times no major changes are seen on the images.
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What are the findings that make you think of improvement?
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That's a very challenging question to answer,
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and I, I don't know that I can answer it.
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Um, I think the best answer to it is how the,
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how the patient feels
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because, you know, you're introducing blood
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and, uh, spin down platelet rich plasma, uh,
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sometimes you're taking as much as 90 ccs
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and spinning it down and putting, putting it into, say,
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a tenderness object.
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Um, so I would say to you this, if you've had a focal defect
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and now you're at least 90 days out
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and that focal defect has filled in, that suggests
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that things are going well.
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But I, but I'd much prefer that, uh, you, you use
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how the patient is doing rather than the
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imaging, uh, in a vacuum. Okay.
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The question here, uh, can you please show us
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with a practical example?
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I can't, 'cause I don't know if cases here, how
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to evaluate ERUs fibrosis.
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I don't know if you have a case or you wanna show that.
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I just would make a couple points about it
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before, maybe you have some images on that,
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but, um, ERUs fibrosis is a strand of tissue
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as we see it on mr.
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But if you know, it's anatomy, it doesn't stop there.
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It has medial and lateral limbs that go down
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and attach to the surface of the ulnar.
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And I, I learned that a number of years ago,
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and since then I've been trying to find those limbs
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and I have never been able to do so.
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So for me, the lacerda fibrosis, I try
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to find the val linear strand of tissue that blends
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with the surface of the pronator flexor group.
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And with injury, I look for the,
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the most helpful finding is the edema usually superficial
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and deep, but sometimes you can see actually disruption
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of the fibers itself.
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But I've never seen a case where I could follow
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the injury down to where it attaches to the oma.
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Yeah, I'm not sure this one will do it
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because this is a, this is a very abnormal one.
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Um, there's extensive interstitial signal in the,
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in the erti and the erti is a, you know,
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it's a fibroelastic membrane.
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So here you're at the myotendinous junction
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with, with the tendon.
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And then as you leave the muscle and things flatten out.
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Now this one doesn't flatten out
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because it is involved, it's actually infiltrated
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by this tissue that has involved the operative site.
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So what I might do tomorrow for those of you that are here,
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is maybe give you a quick snippet.
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I'll find a normal one
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and show you, show you the anatomy of a normal one.
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And I, I will remember to do that
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'cause I know they'll remind me.
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Good.
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Any other questions?
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Okay. How do you describe a distal triceps injury
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that involves a tendon portion, long
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and lateral, the common tendon,
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but not the medial muscular portion?
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That's, do you consider them as complete ruptures?
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It is the most it is, is it?
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Yes, it is the most frequently observed pattern.
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And in fact, I only showed you,
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or I showed you a couple of cases,
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but typically the muscular attachment
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of the medial head is intact.
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I do have a few cases where that was not the case.
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Uh, I have examples of the central
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or common tendon combined with injury to the medial tendon,
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uh, as being involved together.
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Uh, now complete
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and incomplete tears is very, uh, interesting.
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Remember when I talked about the rotator cuff,
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I did not talk about complete tears
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or, uh, incomplete tears of the rotator cuff.
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I used the terminology full thickness or full width.
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Uh, i, with the triceps and with the biceps.
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I will often use the terms complete or incomplete.
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So as you described this as a distal triceps injury
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that involves a certain portion.
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I might say that this is a complete tear of the tendon
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of the long and lateral, uh, heads of the triceps muscle,
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but it is not involving the tendon
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or muscular attachment of the medial head.
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But I wouldn't say that it's a complete tear
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of the entire tendonous apparatus.
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So that's how I would describe it.
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And I, I handle it the exact same way.
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And I will, uh, specifically tell them that the
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muscular head, which rarely tears the medial head,
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which rarely tears, it's got a very, very short tendon
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by the way, is intact.
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I'll add add that into the descriptor that you just gave.
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Right. And is it incorrect
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that the common extensive tendon is not really tendonous?
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As far as I know, in younger people it is purely tenderness.
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But as you go around the human body, as we get older
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in some tendons, as they attach to bone, you get fibro,
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cartilaginous, uh, changes.
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So I think it would depend upon the age of the person
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that you're talking about.