Interactive Transcript
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Moving now to posterior impingement syndromes.
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So when we're thinking about posterior impingement syndrome,
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this of course is impingement on the posterior
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or soft tissues, uh, at the posterior margin
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of the ankle articulation.
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And that can occur from a variety of different
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osseous abnormalities.
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The ADA process that's defined
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as a long posterior tailor process,
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a prominent posterior superior calcan,
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a prominent posterior tibia, or a prominent large O trigonum
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or perhaps even fractured sada process.
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So you can imagine that all
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of these osseous abnormalities could place mass effect on
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those posterior soft tissues.
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In this case, contrast placed in the posterior subtalar
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joint here, specimen photograph
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showing the posterior subtalar joint tibiotalar joint.
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And the point here is to show you
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that there are three sources
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of synovium back here at the posterior aspect
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of the ankle articulation.
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You've got the synovium of the tibiotalar articulation,
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you've got the synovium of the posterior subtalar joint,
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and you've got the synovium synovium
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of the intimately associated flexor lysis longus
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as it passes posterior to the ankle articulation.
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So you see that this area back here is really rich
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with potential areas of synovium that
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with mass effect could have synovial proliferation,
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irritation, and secondary pain.
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So in this case, you can see
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that we've got a long posterial process
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and possibly even a fracture through that.
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So as you consider really this position
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of the foot, it's been associated
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or called the nutcracker type phenomenon.
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So when you plantar flex the foot, you see
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that the poster aspect of the tibia
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and the superior aspect
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of the calcaneus come in approximation
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or approximate one another.
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And if you've got ossea structures here,
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even the synovium could be caught between the tibia
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and the calcaneus
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and ultimately could end up with synovial hypertrophy.
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When I'm looking on my ankle films, I always try
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to look closely to see if there are focal
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areas of increased density.
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Back here. In this case, you've got a thick Achilles,
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you've got enthesopathy at the Achilles.
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I always follow my flexor haliss longest.
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Remember, at the level of the ankle articulation,
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this muscle is the only one
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where we're seeing a significant amount of muscle belly.
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So you know that's your flexor hallis longest.
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Here you would find of course,
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NOL hypertrophy from the ankle articulation in this area,
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synovial hypertrophy from the poster subtalar joint
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and the flexor house.
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As long as that can kind
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of extend along the surface of both. Similarly,
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Of course I would look for anterior tibiotalar increased
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soft tissue densities as well.
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So let's look at this
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lateral ankle film sate a process long
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poster Taylor process.
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Assess the posterior soft tissues.
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You see a face of CAGR fat pattern in the posterior margin
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of the flexor lysis.
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To me, nothing here really looks like there's
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mass like synovial hypertrophy.
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When you look at the sagittal stir image,
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you can see a little bit of high signal intensity here.
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Fally reactive marrow change in
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that sate process, no fracture.
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But when you look at the axial images here, you can see
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that there's synovial hypertrophy along the poster.
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Talo fibular ligament.
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So you know, there's this idea
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that you can only identify synovial hypertrophy if you've
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got T one post contrast images.
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But take a look at the capsule.
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I mean here you're looking deep.
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This is the fibular concavity, right?
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The anatomic landmark that Don was describing to you.
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Not a normal looking a**l fibular ligament.
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Here, your poster talo fibular ligament.
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A little bit ill defined,
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but clearly you see this thickening back here.
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There's simple fluid. This is synovial hypertrophy.
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So don't feel that you're not gonna be able to make
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that diagnosis without post contrast enhanced images.
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Look for it. Look at the level of the joint line
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and where these capsule ligaments are,
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and you'll be able to identify these areas of synovitis.
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Another example here,
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and you can kind of get the fact that there's a plane
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of lucency, right?
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So a large trigonum, again, maybe a little bit
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of synovial hypertrophy here, increased density.
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But when you move into sagal images,
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you're seeing reactive marrow changes on both sides
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of that syn chondro.
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There's that little bit
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of synovial hypertrophy, pretty minimal.
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But when you see reactive marrow changes here,
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clearly those can be a good indicator that you've got pain
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or could have pain.
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When we think about structural abnormalities that correlate
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with pain in the vast majority of cases, um,
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reactive marrow change
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or bone edema usually has a high correlation.
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The other things often are kind of hit
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or miss synovial hypertrophy.
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Synovitis also associated with pain, fat,
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altered signal intensity, high association with pain.
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Other things may be a little bit less. So.