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Differential Considerations in Hindfoot Pain: Posterior Impingement Syndrome

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Moving now to posterior impingement syndromes.

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So when we're thinking about posterior impingement syndrome,

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this of course is impingement on the posterior

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or soft tissues, uh, at the posterior margin

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of the ankle articulation.

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And that can occur from a variety of different

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osseous abnormalities.

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The ADA process that's defined

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as a long posterior tailor process,

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a prominent posterior superior calcan,

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a prominent posterior tibia, or a prominent large O trigonum

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or perhaps even fractured sada process.

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So you can imagine that all

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of these osseous abnormalities could place mass effect on

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those posterior soft tissues.

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In this case, contrast placed in the posterior subtalar

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joint here, specimen photograph

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showing the posterior subtalar joint tibiotalar joint.

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And the point here is to show you

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that there are three sources

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of synovium back here at the posterior aspect

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of the ankle articulation.

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You've got the synovium of the tibiotalar articulation,

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you've got the synovium of the posterior subtalar joint,

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and you've got the synovium synovium

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of the intimately associated flexor lysis longus

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as it passes posterior to the ankle articulation.

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So you see that this area back here is really rich

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with potential areas of synovium that

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with mass effect could have synovial proliferation,

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irritation, and secondary pain.

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So in this case, you can see

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that we've got a long posterial process

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and possibly even a fracture through that.

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So as you consider really this position

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of the foot, it's been associated

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or called the nutcracker type phenomenon.

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So when you plantar flex the foot, you see

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that the poster aspect of the tibia

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and the superior aspect

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of the calcaneus come in approximation

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or approximate one another.

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And if you've got ossea structures here,

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even the synovium could be caught between the tibia

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and the calcaneus

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and ultimately could end up with synovial hypertrophy.

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When I'm looking on my ankle films, I always try

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to look closely to see if there are focal

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areas of increased density.

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Back here. In this case, you've got a thick Achilles,

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you've got enthesopathy at the Achilles.

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I always follow my flexor haliss longest.

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Remember, at the level of the ankle articulation,

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this muscle is the only one

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where we're seeing a significant amount of muscle belly.

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So you know that's your flexor hallis longest.

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Here you would find of course,

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NOL hypertrophy from the ankle articulation in this area,

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synovial hypertrophy from the poster subtalar joint

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and the flexor house.

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As long as that can kind

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of extend along the surface of both. Similarly,

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Of course I would look for anterior tibiotalar increased

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soft tissue densities as well.

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So let's look at this

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lateral ankle film sate a process long

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poster Taylor process.

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Assess the posterior soft tissues.

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You see a face of CAGR fat pattern in the posterior margin

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of the flexor lysis.

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To me, nothing here really looks like there's

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mass like synovial hypertrophy.

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When you look at the sagittal stir image,

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you can see a little bit of high signal intensity here.

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Fally reactive marrow change in

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that sate process, no fracture.

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But when you look at the axial images here, you can see

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that there's synovial hypertrophy along the poster.

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Talo fibular ligament.

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So you know, there's this idea

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that you can only identify synovial hypertrophy if you've

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got T one post contrast images.

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But take a look at the capsule.

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I mean here you're looking deep.

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This is the fibular concavity, right?

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The anatomic landmark that Don was describing to you.

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Not a normal looking a**l fibular ligament.

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Here, your poster talo fibular ligament.

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A little bit ill defined,

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but clearly you see this thickening back here.

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There's simple fluid. This is synovial hypertrophy.

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So don't feel that you're not gonna be able to make

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that diagnosis without post contrast enhanced images.

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Look for it. Look at the level of the joint line

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and where these capsule ligaments are,

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and you'll be able to identify these areas of synovitis.

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Another example here,

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and you can kind of get the fact that there's a plane

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of lucency, right?

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So a large trigonum, again, maybe a little bit

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of synovial hypertrophy here, increased density.

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But when you move into sagal images,

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you're seeing reactive marrow changes on both sides

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of that syn chondro.

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There's that little bit

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of synovial hypertrophy, pretty minimal.

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But when you see reactive marrow changes here,

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clearly those can be a good indicator that you've got pain

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or could have pain.

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When we think about structural abnormalities that correlate

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with pain in the vast majority of cases, um,

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reactive marrow change

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or bone edema usually has a high correlation.

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The other things often are kind of hit

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or miss synovial hypertrophy.

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Synovitis also associated with pain, fat,

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altered signal intensity, high association with pain.

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Other things may be a little bit less. So.

Report

Faculty

Donald Resnick, MD

Professor Emeritus, Department of Radiology

University of California, San Diego

Christine B. Chung, MD

Professor of Radiology, Executive Vice Chair, and Director of UCSD MSK Imaging Research Lab

UC San Diego

Karen Y. Cheng, MD

Assistant Professor of Clinical Radiology

University of California, San Diego

Tags

Musculoskeletal (MSK)

MRI

Foot & Ankle