Interactive Transcript
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Okay, welcome to everybody. Um,
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I'm gonna start out with the first case today and, um,
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it is an 11 year old. He's a male,
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complains of medial pain in the right knee,
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and, um, he's got no known injury,
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as some of you may be noticing that, uh,
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this image on the left is actually a left knee and there is
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also a right knee. So we have both knees available to us.
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And I have two Coronas,
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two paired coronals for you that show a similar finding.
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Just to make it a little easier. Um,
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I might move this one over to the opposite side and then move this one
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over here so it's a little more intuitive. So this is right, this is left.
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And let's do a little scrolling in the coronal objection.
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Now,
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when I look at a pediatric case and we see well over a hundred pediatric MSK
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cases a day in our practice, um,
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one of the first things I do besides look at the history is I try and look at
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the growth plate and decide, you know, is it,
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is the growth played appropriate for the patient's age? Is it open or closed?
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That's very relevant, not only in terms of the diagnosis,
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but in terms of the prognosis of the abnormality.
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Many abnormalities do better when they present in a younger age group.
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Your,
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your decision as to the severity of an abnormality will
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also be based in many cases on the patient's age.
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And as I scroll back and forth on each of these,
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and I am focusing for now on the Corona projection using two
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symmetrical proton density fat suppression images,
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some of you might have noticed that
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the condos don't really kind of equal each other.
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So let's start out with our first polling question because I,
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I think I've given you the salient finding in the, in the salient projection.
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So let's go with the first polling question and put that up.
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Well, while you're doing that, I'll show some other
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Projections. Here's the left side, a T2 weighted image,
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and here's the left side, a T1 weighted image. Let's go to the area of interest,
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which is right here,
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and let's pull the T1 weighted image from the opposite side.
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Take a look at that. It's even broader.
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And now let's look at the water weighted images from both sides as well.
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There's the water weighted image from the right side and the sagittal
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projection, and here's the water weighted image from the,
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the left side in the sagittal projection.
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So rather than wait for the question, let me,
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let me ask you all to self-reflect,
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and I'm gonna put the Corona images back up from both sides.
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I'm make it so it's a little intuitive for you.
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So I got the fibular on the right here.
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This is the right side fibula on the left, left side.
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So do you think that these lesions are stable or unstable?
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I think that's the first question that you should answer because the diagnosis
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should be pretty obvious to you.
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There is an osteochondral lesion involving the lateral
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aspect of the medial femoral conal on both sides, an 11 year old.
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So that's a plain radiographic diagnosis. And let's,
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let's put up our, our question now and see if it's ready. Let's give it a shot.
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All right. Which of the following is true regarding these two knees?
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Now, I'm, I'm not here to be easy on you either.
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Some of these questions will be a little tricky. Some will be easy. A,
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the patient has bilateral osteo andr, decans B,
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the patient has bilateral femoral dysplasia. C,
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one of the A osteocondral defects is unstable and the other is stable D.
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The lateral aspect of medial femoral condi is a typical location. E,
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all of the above. See how you answer this one?
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I'm not allowed to vote, but you are.
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I've already given you clues to the correct answer on this one.
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Let's see what our panel of participants said.
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So most of you said all of the above, which is the correct answer. Yes,
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the patient does have bilateral osteochondritis decans, but yes,
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they also have bilateral femoral dysplasia.
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And I'll show that to you in a moment.
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One of the osteocondral defects is unstable and the other is stable.
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Co lateral aspect of medial femoral condal is the typical location.
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So let's talk about the dysplasia.
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Look at how tall it's taller rather than wide on both
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sides. In fact, most cases of osteochondritis decans, if not all,
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are related to a friction delamination of the
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subarticular blood supply due to repetitive mechanical trauma
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from a dysplasia.
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That's true in any joint where you see Oticon decans.
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I also like the four thirds rule.
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I'd like the lateral femoral con to be no more,
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more than four to three on the ratio between the lateral
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condi and the medial condi. In terms of transverse dimension,
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you can see this one's tall and narrow. This one's short and fat. In fact,
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this one's taller and proximal to distal extent and
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reaches more distally than the lateral femoral conal itself.
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So there's undue force being applied to the medial femoral canes
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on both sides from the simple act of weightbearing.
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So indeed there is bilateral femoral dysplasia.
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A fact that perhaps in the past you might have overlooked
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the lateral aspect of medial femoral condal is the most common location.
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But what's the second most common location?
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I've not posited that to you as a question, but I'm asking you to ask yourself.
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And the answer is the a**l lateral femoral troia,
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which in days gone by of plain radiography, was thought to be a rare location.
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In fact, it's not rare at all.
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It is by far the second most common location of osteochondritis decans after
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this one.
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Now another question that's posited to you is stable versus unstable.
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There is some debate minor,
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but some debate about how to approach stability in children.
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It is said that cysts greater than five millimeters in size are a sign of
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instability.
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I would suggest to you that this sign by itself is a sign of micro
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instability,
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but not necessarily macro instability because I've seen many children
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with open growth plates that have cysts,
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small cysts only even those that approach or exceed five millimeters in size,
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as long as they don't have fluid signal between the lesion and the bone,
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they have the potentially heal. Now,
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the ones that are more likely to heal are the ones that are further and further
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away from the weightbearing portion of the condo.
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So when they're in the antra lateral trochlear ridge of the femur
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non-weightbearing, they heal 95 plus percent of the time.
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So we treat those uber conservatively. Now, if we look at these two,
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look at the fluid like signal confluent,
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very bright and through and through the cortex all the way to the knee
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notch. Much different looking when you really drill into it than this other one,
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which has high signal intensity.
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Not nearly as many cystic foci doesn't have that free edge
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communication and doesn't have that smooth,
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well-defined confluence signal intensity with cortex on either side.
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So those are the criteria I use for stability in a child.
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I use cysts as a secondary sign.
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Some of my colleagues use it as a primary sign. In adults,
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I also use it as a secondary sign. The greater the number assists,
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the greater the size,
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the more likely the patient is to have severe micro or macro instability.
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I use fluid like signal, not edema, but fluid signal.
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I use the morphologic sharpness of the cortical edges and the lack of bridging
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thereof as another sign of instability.
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And then there are secondary signs such as a joint effusion.
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And if there's displaced chondro or chondro osseous bodies,
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then the diagnosis is made easy.
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So this is an example of bilateral osteochondritis decans.
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The patient does have some subchondral edema that's worse on the right than
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on the left. The patient has cyst from instability, more of them,
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and bigger on the right than on the left.
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The patient has a clear cut water signal cleft between the fragment
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on the underlying bone, right,
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rather than left and the patient has bilateral femoral
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dysplasia.
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How do you differentiate osteochondritis decans from an osteochondral defect?
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So osteochondritis decans is a subset of defects
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in the bone and cartilage.
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So all osteochondral defect means is that you have a defect in the bone,
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in the cartilage. So what are some of those subsets? Well,
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in a patient with osteoarthritis, Kellgren Lawrence, stage two or three,
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it could be a condral LASIK erosion in a patient who's had acute trauma,
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it could be a defect from an acute osteochondral fracture.
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It could be an unhealed fracture such as a chronic non-heated
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osteochondral fracture. It could be osteochondritis decans.
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These are all subsets of osteocondral defects.
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So you might introduce this case by saying there's an osteocondral defect of
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the osteochondritis decans type in a juvenile.
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Now I wanna point out something to you that, uh, Dr.
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Thapa has pointed out in some of his pediatric lectures.
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And that is it's very common to see
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stippled irregular ossification centers in the, in the medial femoral conduct.
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And they're usually seen in the back, in the non-weight-bearing area.
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And they can produce some confusion with osteochondritis decans.
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They may have a little bit of high signal in the cartilage associated with 'em
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and a little bit of high signal in the bone.
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They're usually stippled and multiple,
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they're usually non-weight bearing. They're usually not associated,
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they are not associated with an effusion.
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And they occur when the growth plates are grossly open.
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So the more close the growth plates appear,
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the more likely such a lesion or pseudo lesion would be a real
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lesion. But beware in the posterioral medial femoral condo,
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you'll see multiple ossification centers and what looks like a little bit of
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blistering in the back.
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This is way too low and in too classic or typical location,
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lateral aspect of the medial femoral con to represent the normal developmental
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ossification center of the juvenile. So hopefully I've answered your question.
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Odessan, a subset of osteochondral lesions or defects.