Interactive Transcript
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You'll need those general questions at the end
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and we'll move on to our next.
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So our fifth case is another patient
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to presented with chest pain in the
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component elevation after covid. So after Stars
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could we do infection? And again, they were
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suspicious the clinicians versus suspicious for macarvis and
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referred patients for cards. So start with
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a similar pattern. I'm going to show you the four chamber sending us
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with the on the left and hopefully even on here you
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can see that the left ventricle function is imperative. It's not
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normal the shotgun ventricle injection fraction with
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impaired here moderately impaired with ejection
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fraction of 40% Right ventricle also
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has an impaired machine with the
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injection fraction Quantified of 45%
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And so even ours in the ssfps before
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we've got to any of the tissue characterization sequences. This is
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a young patient. This is a certainly not
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normal. Let's look at our short access to try to
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get a clue of whether they're maybe a regional wall motion remaldi
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to kind of direct our attention on our tissue characterization sequences,
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and hopefully at that in for
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a lot of wall at the base you could see that it was thickened and hypokinetic.
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So not only is their Global left ventricular dysfunction.
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There's also a regional law motion of normality and
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that's kind of helpful. I find when I'm reviewing
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with my photos to when you see that on the short axis when you
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see those original While most genre you want to look very carefully at
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that area of my cardium on your tissue characterization. So
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I'll just pause here.
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That this area of ladderwall is thickened and
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we can maybe see that there's some subtle higher
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signal intensity. Again. This is required
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those contrast. You can also see that there is a small effusion mostly
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in Fairly and lateral.
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Now we'll move on to our edema Imaging
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teaching math on the right and I'll pull up our teaching with
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the image on.
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on the left
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This patient was imaged again. Not in the very acute
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phase when they presented. This was an outpatient.
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Had a moderate covid-19 illness
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and then came in with
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chest pain, but with images and outpatient. So
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the Imaging was done a few weeks after their chest pain started
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and we can see though that despite this
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there is some increased signal intensity.
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At that influater wall, but unlike some of the more few
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cases that we image. It's kind of more diffuse almost the whole
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wall has in that area has higher signal intensity
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and you might be able to see as well that the overlying
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pericardium is also Abner and what's also
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itemitous and has higher signal times.
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On our teaching map again, you can see that that
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area of myocardium thickened and values were
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slightly elevated above our normal reference range.
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Here's our lake at an intense Imaging
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on the right and I'm going to show you where it's T1 map.
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Again. This is a need of two and map on the right. I find
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that the fusion kind of really pops here. So
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if you missed it before this might be include it there is a small
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pair of cartilage Fusion as well. And on the
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LG images. I'm just gonna window them a bit and start
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to see that it into a lot of wall. There's mid wall and some
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epicardial energy and as we come down
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we can see that it's also involving in nature wall here the
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cardium as well as the answer or lateral wall in
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the intra wall and there's also enhancement of overlying
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pericardium at that infilateral wall. So we have a myoperative
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in a patient to presented with
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very classic symptoms of a cute concept of
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chest pain and troponine elevation after Stars could be
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to infection but again in a very similar pattern and
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Station compared to the other myocards of
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this cases that we saw before here. You
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can see that this is a little bit more extensive though. It's extending actually all
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the way here in the mid ventricular level up and
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actually to the apical segments as well. So more extensive
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involvement involving more segments and
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more area of The myocardium. But again meeting
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both T2, based criteria and a T1 based criteria here
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with more extensive abnormalities and
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more impaired left ventricular ejection
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fraction in a regional motion abnormality.
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little pots there
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Okay, and I will answer questions the more
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general questions about treatment and stuff afterwards. But again,
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hopefully you're starting to see the pattern kind of
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how I go through cases and some of the similarities in
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these cases despite the fact that they actually
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trigger for the acute myocarditis.
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Okay, now our next case I need to pull up is our tax
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did not let me load all of the cases in advance.
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fun
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So we're gonna move on to our sixth case.
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This is also a patient to presented with chest
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pain after SARS copy to infection this patient with
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hospitalized during their covid infection, and I'm
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going to pull up the four chamber view on the left and
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You can see here that the left ventricle has impaired
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objection directions with this lap ventricle is not Contracting
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normally actually are left. Atria is
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dilated here. This is the patient you presented several
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weeks before kind of had a more indolent course
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and had again. Oh patient Imaging they have been very
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more sick the moderate infection during their
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acute SARS Kobe 2 infectious phase
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and then came in with persistent and
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worsening chest pain and I'm just
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going to skip I'm going to show you the tutu map on
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the right and I'll show you the
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resource images
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My teaching black way to do it teaching mapping
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images and we can see here that the pericardium is
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kind of quite hyper intense. There's some
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patchy areas of high signal intensity in The myocardium, but
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nothing vocal like we saw before our teaching mapping
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values, we're also wildly elevated but more diffusely and
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on our Lake and our enhanced image all
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the short axle Stack Up on the right. You can see here as a
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school through that. There's actually quite extends about normalities mostly
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stop that the cardio here at the basil in for
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a wall in for a lateral Angel Walling pericardium
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is enhancing and Molly thick
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and we also have an abnormality that extends to
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involve the right particular side. So and I
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will come down towards the midventricle we can see that this is much more
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extensive like and enhancement. But again,
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mostly step up a card you'll also involve me to stop
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them in this patient to presented with just
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pain and elevated Japan and level here after a
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more severe.
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Kobe 2 infection again would meet both teach you
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and T1 based criteria in this case in this
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patient who had
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a carditis after
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their covid itself very quickly. I'm going to show you
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a companion case and then we'll move on to the slides.
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A patient who is referred for MRI for suspected
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myocarditis after covid and
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I'm good quickly. I'm just gonna skip to
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the key findings in the interest of time unlike the
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last cases where the predominant abnormality
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on LG Imaging with epicardial or
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midwall in this patient. We did see that there
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was abnormal LG and pointing it out here. It's actually any some
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endocardial pattern here at the
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intro wall extending into the involateral wall. There was
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some corresponding and demo which I'll show you
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but it was quite subtle.
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And I wanted to show this with the companion
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case because this patient did have so there is some edema
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in that area of myocardium, but perhaps not
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as striking in some of the other cases this patient
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presented very similarly acute onset of
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chest pain and elevated drone levels after Cyrus Kobe
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to infection this pattern of abnormality,
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which is currently stubborn to Cardinal has been
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described in myocarditis. There was actually a paper
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in Radiology last year that described this subunit cardial
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pattern as a infrequent pattern of acute myocarditis,
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but of course the other thing you want
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to think about when you see this pattern particularly corresponding to
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Corner artery distribution is a cute monocardial infarction.
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And so that's what we suggested here at patient
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had a calf and did actually have a cute RCA occlusion
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proximately and underwent PCI and
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we'll talk a little bit about why that was potentially precipitated
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by the Stars Kobe to
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infection as patient, but I wanted to show it at the companion case.
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Just a reminder that other etiologies and other
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disease processes can present clinically quite similarly and
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just to have the differential in the back of your mind.