Interactive Transcript
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of case-based micro-learning courses across all key
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radiology subspecialties.
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Today we are honored to welcome Dr.
0:43
Alka Singal for a lecture entitled Ultrasound, gallbladder
0:46
and Biliary Tree, common and Uncommon Diagnosis.
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Dr. Singal is currently an associate director
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of radiology at Madon Division of Radiology
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and nuclear medicine.
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She's a renowned expert in diagnostic whole body
1:01
ultrasound imaging.
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Her areas of focus include the abdomen, obste, obstetric
1:07
and fetal USG with 3D
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and four D imaging, elastography, Doppler, vascular studies,
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neck parathyroid thyroid, and MSK ultrasound.
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Dr. Singal, who is dedicated to quality, accuracy
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and patient satisfaction, has earned fame as one
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of the best radiologists in the area
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and has a great passion for work, both
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for diagnostics and academics.
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At the end of the lecture, please join Dr.
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Singal in a q and a session
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where she will address your questions you may
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have on today's topic.
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Please remember to use the q
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and a feature to submit your questions so we can get to
1:43
as many as we can before our time is up.
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With that, we are ready to begin today's lecture. Dr.
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Singal, please take it from here.
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Thank you so much. Thank you so much everyone
1:54
for joining in at this time.
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And today's topic is, uh,
1:59
pretty much the most basic ultrasound image
2:01
that we have been doing, ultrasound, gallbladder
2:04
and biliary tree, common and uncommon diagnosis.
2:07
However, it is also one of the areas
2:11
where you have a lot oft reporting.
2:14
Why is that then?
2:16
That is something which we really begin to have,
2:19
have having done when we started learning
2:22
ultrasound imaging.
2:24
However, even
2:25
after 2030 years of imaging that same organ in
2:29
that same area, why is there a discrepancy in the reporting
2:34
from tool often within the same operator, often
2:38
at a different time, or with a different machine
2:41
or with a different, uh, place?
2:44
Let's unravel that.
2:46
Okay, so the of course, as we all know,
2:48
there's a lot to do with the operator.
2:49
There's a lot to do with the technique.
2:51
There's lot to do with the patient preparation
2:54
and all those factors that go into
2:58
optimal imaging on ultrasound.
3:01
It's okay.
3:06
So the common and uncommon diagnosis, it's one
3:08
of the such a common areas that the first thing we, we,
3:13
we have actually narrowed down ourselves to think
3:16
that the gallbladder, whether is it there
3:18
or whether it's removed and whether does it have a calculus
3:22
or is there any mass obvious and that's it.
3:27
And we think that's, that's all for the gall bladder,
3:30
but sometimes we need to think beyond.
3:32
There's a lot more that ultrasound can unravel
3:36
and especially because ultrasound is the mortality of choice
3:41
and it is the first mortality
3:43
and it is the recommended mortality for the evaluation
3:46
of reta for the common inflammatory conditions right now.
3:51
So why the discrepancy?
3:53
Of course, is there any ion preparation needed
3:55
to evaluate the treat?
3:57
It's, of course there is.
3:58
However, in cases of acute abdomen, we bypass that.
4:03
So we have to evaluate whatever scenario
4:06
the patient has presented.
4:07
That doesn't remain as an hard and pass guideline as well.
4:11
However, optimally we do require the patient
4:14
to be four to six hours.
4:15
A asking why, because gallbladder is a peer shaped
4:19
structure, which is stored the bile juice.
4:22
Only when it's distended can we see the walls very clearly,
4:25
whether there is any lesion, any, any, uh,
4:29
any LAR in the sac and what is the thickness of the wall
4:33
and every area of the gallbladder,
4:36
whether there are any falls, any uh, junction falls
4:39
or any F cap or the walls of,
4:42
or the neck areas, everything,
4:44
because only when it's distant,
4:46
it just like the urinary bladder can you really unravel it.
4:49
Right? And of course the amount of fasting will vary, uh,
4:54
for infants and neonates and often two hours is good
4:58
and we do a postprandial scan in pediatric population
5:01
to see the and functionality when we are suspecting praz.
5:06
So that's another part of the dynamic scan.
5:08
And especially in patients in adult population,
5:12
sometimes even with four to six hours of fasting,
5:14
we are not able to see the gall bladder.
5:16
They could be thickened and inflamed
5:18
or contracted due to chronic chronic cystitis.
5:21
There you may require a longer fasting, which could be eight
5:25
to 12 hour fasting.
5:28
Okay? So with that now coming to the transer selection
5:33
and the patient position,
5:34
but transducer selection again goes to the crown root
5:38
of the ultrasound basics, the highest frequency transducer
5:42
that gives you the required depth of penetration.
5:45
So as simple as that.
5:46
So I would say if I would use a three
5:49
to five megahertz kby transducer for a, uh, for a patient
5:53
with a, with a large body habitus,
5:56
that would be correct appropriate.
5:58
But if I have a lean thin pediatric patient, right,
6:02
I wouldn't go with a 3.5 megahertz.
6:04
I would go with a linear transducer, maybe seven to 11
6:07
or maybe a linear, a higher frequency curve transducer
6:11
to get the better resolution.
6:13
So the transducer frequency selection then comes the
6:16
equipment, the both the harmonics and the compound imaging.
6:20
Now that part currently is actually taken care of
6:24
by the inbuilt applications within the machine.
6:26
So we do not do much changes to that.
6:29
However, going back to my, um, uh,
6:33
like few years ago, we, we would change it to harmonics
6:37
and compound and various other factors
6:40
to get the best imaging.
6:42
And of course we will be adjusting the focal, uh,
6:46
focal point at the level of the pathology
6:49
because that's where the beam width is the narrowest,
6:52
and that's where you get the best resolution
6:55
and the best acoustic shadowing of calories.
6:58
So that's about the patient position.
7:00
Now what's the patient position again, comes
7:03
to the same principle.
7:05
The, we want the, it's examining part to be as superficial
7:09
to the transfuser as we can.
7:12
So we are going to rotate, uh, so
7:14
that we have the examining parts superficial.
7:17
However, we also gonna use a solid organ such as a liver,
7:22
as a window through which we are going
7:24
to scan the gallbladder.
7:25
So we try and position the patient sine a left lateral
7:29
and we use a liver as a window to see through to the,
7:33
which works better because if you try
7:36
and go the other way, you might have bowel gases in the
7:39
middle, uh, in the central part of the abdomen,
7:42
they'll cause their own dirty or postage shadowing
7:44
and will limit visualization
7:46
of any pathology which may be there in the gall bladder
7:50
transducer selection,
7:51
we discuss the higher frequency image optimization modes.
7:54
We will discuss ATE scan.
7:56
So we will evaluate
7:58
as we evaluate any solid organ in all the three orientation
8:02
and planes, s lung, uh, oblique, such trans oblique.
8:06
And of course all bladder scan doesn't finish until an,
8:10
unless any examination is performed in the erect position
8:14
because that's when
8:16
the de the fundus gravity dependent part comes down.
8:20
So if there's any small subtle not uh, the uh, calculus,
8:25
it'll just trickle down in the fundus, it would be very easy
8:28
to demonstrate.
8:31
Moving on. So now what do we mean
8:34
by the goldbe and the ary tree?
8:36
So we see, uh, we all know it's a peer shaped structure,
8:41
thin wall, smooth wall,
8:42
and it's seen in the right upper quadrant.
8:45
It's goes by and it's fasting.
8:47
It can be approximately 10 centimeters long.
8:52
Okay?
8:58
Okay. Right, right.
9:00
So certain features that, that are normal
9:05
a**l variations in the normal, we must remember.
9:09
So there is a fundus, there is a net area.
9:12
So that, why is this important is when you're labeling
9:15
technology, the communication remains the same
9:19
throughout in the neck.
9:21
There is especially an area called in fund development,
9:23
hartman's pouch, which is an common area
9:26
for impaction of gallstone.
9:27
So evaluating of the gallbladder, just seeing the fundus
9:32
and not seeing the cystic duct
9:34
and its confluence with the gallbladder
9:36
is like you're evaluating half the orbit
9:39
and you're saying that it's normal and not no sizing.
9:42
That is not the correct strategy.
9:45
So the correct strategy should always, always demonstrate,
9:54
always, always demonstrate the make
9:57
and the distal part of the very well clearly so
10:01
that there is no theology left out,
10:03
including if you can see the balls of hazel,
10:06
that is also good.
10:07
Now the cystic duct, which comes out of the gallbladder, you
10:12
if you can, you would try and see it and try
10:14
and see the confluence and where it merges.
10:17
Then you come into the common hepatic duct.
10:20
So normally if the biliary tracts are dilated,
10:24
you would often see them
10:26
and uh, the diameter of the common, uh, uh,
10:29
common pal duct often is four
10:31
to six millimeters in diameter.
10:33
But however, it can have a normal slight sif form dilatation
10:38
in, uh, elderly patients or post cystectomy.
10:42
You must see the duct in total in longitudinal
10:46
and transfer section to exclude any focal calculus
10:50
or any focal subtle mass lesion.
10:53
There may not be any other sign,
10:56
and believe me, catching that pathology very early.
11:00
Only you who's holding the ultrasound prob has the,
11:05
has the privilege to actually alert
11:07
and advise for a further imaging or a CT scan
11:10
before the patient present with symptoms.
11:13
When the patient present with symptoms, often this area,
11:16
because it's already such a narrow structure,
11:19
it often goes into a, uh,
11:22
nonresectable stage.
11:24
So a lot of responsibility goes in evaluating the reta
11:29
and the CBD completely
11:32
by the person who's holding the ultrasound.
11:35
And of course the common B duct is named
11:37
as supra pancreatic, retro pancreatic.
11:39
And the ary component,
11:40
which all must be evaluated very well,
11:43
intrahepatic period guts when dilated,
11:46
that's when we see them.
11:48
And if you don't see them, that's good.
11:50
So of course anatomy radiations are there
11:53
that you must remember and accordingly scan.
11:56
Now the role of ultrasound.
11:59
So of course we have other modalities
12:01
because going from pediatric to pregnancy to any age,
12:07
the goal ultrasound is like suitable, but every age,
12:11
and it's easy, it's quick, it's, you can move it around
12:16
and you can get the information,
12:17
you get the information in everybody.
12:21
So with the use of technology,
12:23
ultrasound is the first mortality
12:26
and it is, uh, it is a goal, uh,
12:29
recommended option for the gallstones.
12:31
Stones beyond the G
12:33
and the CBD are often requiring high ends
12:37
of suspicion and often require other imaging
12:40
to further conclude.
12:42
Right Now coming a little word about
12:45
the pediatric population.
12:47
So the bases we bases are the same In cases
12:51
of emergency, often we have to ignore the preparation.
12:54
Preparation. There could be cases of acute cholecystitis
12:57
or perforation or gangrene or any other.
13:01
So then you take it as it is.
13:02
Now, why is the child sent bi atia
13:06
as a communist diagnosis that you're looking at?
13:09
So what are you looking at?
13:10
Often there is a streak gallbladder
13:12
or there's a tiny gallbladder.
13:14
So what kind of a transducer would you require?
13:17
You would require a high frequency linear transducer
13:20
to demonstrate the gallbladder
13:23
and that's what you have to see
13:25
and give a measurement to the size to the length
13:28
that you see, to the width that you see.
13:30
And if you can get a volume, that's also great.
13:33
So that is a little different way
13:36
of scanning the gallbladder.
13:37
So, so in REIA you may not see the gallbladder
13:40
or you may see a tiny gallbladder,
13:42
which is like a streak gallbladder.
13:44
And this may be uh, even
13:47
after two to three hours of fasting.
13:50
Okay, so another common area when you're uh, suspecting
13:54
razia is a triangular sign, which is a thickening
13:57
of the ligament that you see, uh,
13:59
of the triangular portal area.
14:02
So when you see this area, hyper coic interior to the fo
14:07
really, really suspect the abnormality and
14:11
accordingly, uh, classify and give the diagnosis.
14:17
So the most important uh, different uh,
14:20
difference in imaging in cases of pediatric
14:22
and versus adult population is uh, looking at the cues
14:27
and using the appropriate transducer frequency
14:30
to evaluate the detail.
14:34
Now, I mean just moving on with the age.
14:36
So what are poly cy in the pediatric population?
14:40
The commonest abnormality that you would see is either a,
14:43
like we discussed or often the child is presented
14:46
for al cyst, the con.
14:49
Now the al cyst, they will just come to you,
14:53
they'll actually present as cystic lesion at times
14:57
of various shape and various locations for you to be able
15:02
to establish the connection with the biliary tree.
15:04
And that's what's going to lead to a diagnosis
15:06
and help you differentiate whether you're dealing with a
15:11
cyst in the bary tree
15:12
or whether a cyst in the hepatic or the liver.
15:17
So the challenge will come in differentiating
15:20
between the two for which you really need to be able
15:23
to establish the connection of it with the beary tree.
15:26
So what are poly cyst represent?
15:28
Conjun cystic dilatations of biliary
15:31
and diagnosis relies on exclusion of other conditions such
15:34
as gall stone inflammation as a cause
15:36
of biliary tract dilatation.
15:38
And the 60% are diagnosed before the age of 10 years.
15:43
Clinically child presence with pain, jaice, mass
15:48
ultrasound, you often see assisted structure
15:52
and seeing that assisted tructure may be often in the
15:56
appropriate clinical se settings be an in enough indication
15:59
to better uh, further imaging back cross-sectional imaging
16:04
and often de cyst over a period
16:07
of time may collect sludge stone or even grow a new plasma.
16:12
So sometimes this is are so large that the connection
16:16
with the duct is not easily
16:18
recognized on ultrasound imaging.
16:20
And of course we have to do our best maneuvers,
16:22
various windows to demonstrate that relationship.
16:26
For example, here we have a case, now we see IHVR,
16:30
we see the dation data with radicals
16:34
and we see a cystic structure in the region
16:37
of the portal, right?
16:39
So it's a dilated probably this is a c dilated CBD
16:43
and there's a few form kind of a dilatation of the distal
16:47
portion of the mid and the distal portion of the V.
16:53
Now what are the types of al cyst? There are uh, five types.
16:57
First is the diffuse dation of the extra hepatic bile duct.
17:00
This which is the most common type.
17:02
Second is the true tubulu of the biotech very way.
17:06
The third is also called the choto seal,
17:08
which is a diffused dation
17:09
of the very distal indual common bile duct.
17:13
Type four is multifocal dations to the in hepatic
17:16
and extra hepatic bile duct type five ISIS disease is
17:20
omitted because it's not a true choto cyst.
17:23
So accordingly you can have the commonest is dilatation
17:27
of the extra hepatic bio duct seen in 80 to 90% of cases.
17:31
And depending upon the shape they could be cystic,
17:33
secular or fusic.
17:36
So you could have this kind of an appearance.
17:38
Now let's look at this case.
17:40
Now here we have a picture which is showing an ultrasound
17:44
image, which is an image of a right upper.
17:47
I see that's the dome of the diaphragm
17:50
and I see a large cystic region right
17:54
now I've got a video clip.
17:56
So with this case that I've done few days ago
18:00
and I see in this cystic area, I see some, some, uh, sludge
18:04
and debris here in this area and I try
18:07
and establish a connection.
18:08
So maybe, uh, it's connecting here with the V tree.
18:12
Now these are some still images of the same patient.
18:15
You see the V tract, you see the dilatation,
18:18
you see the cystic area
18:20
and you see some internal content that moves.
18:24
Obviously the diagnosis can be very challenging,
18:27
whether it's a hepatic or it's a affiliated tree.
18:31
However, this was a confirmed case of colorectal cyst
18:37
and the patient was also sent for a ela uh, assessment
18:41
of the, uh, parent chime to see if it was still functional
18:46
or it had, it had concourse on.
18:49
Okay, so that's, uh, the other part
18:52
of the imaging that you would do.
18:54
So this was a case of quality of cyst with the stenting.
18:58
So again, we look at the establish the connections.
19:00
So see we done stenting to release some pressure
19:04
and we can hear, establish some connection with the B three,
19:08
which is pretty convincing
19:10
and consistent with the diagnosis.
19:13
And this case child presented with vomiting
19:15
and pal thrive lump abdomen increasing in size, known case
19:19
of political is diagnosed at five months
19:21
and gene on antenatal, uh, uh, ultrasound five months
19:27
and which was uh, size of 18 and 14 to 14
19:31
after birth and followup ultrasound,
19:33
there was further dilatation and increase in size
19:37
and it was then MRCP, there was a cross dilatation.
19:41
So finally the patient was, uh, planned for hepatectomy
19:46
with cyst resection and HEPA anastomosis
19:49
and child was disturb.
19:53
So that's mainly where the pediatric population not coming
19:56
to the normal adult population
19:58
that we are scanning every day.
20:00
So of course like we discussed positioning,
20:03
we taken the lip window, why we are displacing the bowel
20:07
and we are getting a nice contrast to CD clear
20:10
and equally uht role of breathing,
20:16
role of breath holding in p it doesn't apply
20:18
because you won't be able to give that command
20:20
and role of dynamic scan in various patient positions.
20:23
Not all these factors.
20:25
How persistent, how passionate you're
20:27
to really see the verta in total would reduce your
20:33
uh, uh, redos, right?
20:35
So I'm like, I'm very, uh, like, you know, particular
20:39
for the positioning to do the gall often when we are trying
20:43
to do the lateral disposition.
20:46
So the most common pitfall
20:48
or the error that is made is when you're trying
20:50
to meet the patient is supine and we try
20:52
and meet the patient in decubitus.
20:54
So instead of a full decubitus,
20:56
often this patient is scanned in a slammed position In
20:59
that position, whatever you do, you will not be able
21:03
to scan the complete neck area.
21:06
That is one of the very commonest, um, positioning, uh,
21:11
pitfall because yes, we have to roll the patient this side
21:15
and that side and all those things, but
21:18
however, if you haven't put the patient completely
21:21
in the depth disposition, you wouldn't or,
21:24
or rather slightly forward faster the disposition
21:28
and then you really begins to be able
21:30
to see the complete neck area that is very, very important
21:33
to evaluate the area fully.
21:36
So the is best viewed in the left lateral disposition.
21:39
However, it can be viewed
21:41
with the patient's spine on right it are useful
21:45
to determine if the students are mobile
21:47
or impacted in the neck
21:48
or sometimes even if they're just uh, mobile
21:51
and it may not be seen on the routine scan,
21:54
but if we do the last finishing touch in then position,
21:57
he would just say that something just drop down, right?
22:00
Role of 3D, not really, I don't use it,
22:03
but just outta curiosity.
22:04
But an image of a a polyp, we are just trying
22:08
to see the old surfaces.
22:10
Whatever you can, if there are any IAC polarities
22:13
as we'll come to the topic polyps.
22:15
So small benign polyps are okay,
22:17
but where like 10 millimeters
22:20
or more come into the significant category.
22:24
So as we all know, GBA is a peer shape structure.
22:28
Now what are the dimensions? Do they really mean anything?
22:31
Do we really need to be worried about the dimension?
22:33
What do we mean by what dis tend, what do we mean
22:36
by understanding what do we mean by postprandial?
22:39
Like these are the things which are um, used
22:44
but were not with very defined criteria, right?
22:47
So we can only give a,
22:50
it's about seven centimeters long post centimes wide
22:53
and this is capacity
22:55
and when it's markedly distended it can up
23:00
to 300 ml F fluid.
23:01
Now coming to the size size we have pediatric
23:06
nomograms are charts that we have to follow by the age
23:09
because that's where we are trying to see uh, to exclude as
23:14
to what is the minimum size of the Colorado.
23:16
So we have at least a length of six, uh mm
23:20
and at least a two mm width, uh,
23:25
uh, two centimeters width
23:27
and a six centimeter length is what we uh, have at least.
23:32
So that's the gallbladder wall
23:35
and the wall is normally about two millimeters,
23:38
but not more than anything.
23:40
More than three is defined as abnormal.
23:43
So you should be able to see the make completely through
23:47
and through and establish a connect with the cystic duct.
23:50
That is very, very important.
23:53
Septe are seen in the ver sometimes they divide the ver
23:57
into compartments.
23:59
So septe are normally through
24:00
and through junction holes that we see are often half part.
24:05
So they is considered as a serial duplication
24:07
of the Colorado and may be associated with some complication
24:11
during poly system,
24:12
but I don't really think that's mainly a literature review.
24:18
Uh, water are dimensional folds.
24:19
These are the holes that just fold onto himself in the part
24:23
just like uh, hydros we have the folds, right?
24:27
So these are usually taken incomplete
24:29
and the CEP are maybe complete
24:32
and they may be associated with cases of small polyps
24:36
or multiple coele artifacts in case sub ayo
24:41
coming to the ian cap.
24:43
That is interesting because ian cap often lodges some,
24:48
it can lodge a calculus
24:49
and it can become so dense
24:51
that the whole area can become dense
24:53
and sometimes it can be just missed pathology of a uh, of a
24:58
calculi or any lesion or any mass lesion in that area.
25:02
So when the fi cap is there, you have
25:05
to look at it very carefully that it is distended
25:09
and you can demonstrate the pathology.
25:15
We all edema is thickening of the edema,
25:18
which could be non-specific due to any inflammation
25:21
or cystitis.
25:23
We will come to that. So the commonly list, uh, appearance
25:28
that we see is hepatitis.
25:30
We see the GB wall ine cases of uh,
25:33
typhoid hepatitis and placebo.
25:36
Now what is gall bladder sludge?
25:38
We use that word very commonly called bladders shows hyper
25:42
sl or hyper slage or genic slage
25:45
or soft with no shadowing.
25:49
So what is sludge?
25:51
So gall liver produces bile stored in the gall bladder.
25:55
So when it's not draining well when it's
25:57
staying for a long time.
25:59
So the green pile can often be collected there, it can be,
26:03
it can form a sludge, it can also convert to mucus
26:08
and it can be a mucus seal as well.
26:10
So the mucus from the reta can mix with the cholesterol
26:13
and the calcium combining to create the sludge, okay?
26:19
And over a period of time, sometimes it may be so um,
26:24
thickened and dense that it's actually giving an appearance
26:27
of almost the same equ
26:29
and echogenicity as a it HEPA paradigm.
26:33
Now this word called he of
26:37
could happen due to various factors.
26:39
One of them is SL collecting over a period
26:41
of time sometimes there could be an actual lesion which is
26:45
hiding behind this kind of an appearance.
26:48
So do you when you see he of re
26:51
so really you cannot comment on ultrasound imaging whether
26:55
there is a pathology or mass lesion in the GB
26:58
or there is not a a lesion there.
27:01
You would need a contrast enhanced CD scan
27:04
for further evaluation, right?
27:08
So uh, of course yes we see longstanding cases with stasis.
27:12
So when there is inadequate drainage,
27:14
that's when it collects and it pop.
27:17
Another common finding that we see is adeno
27:21
omes are called that.
27:22
Now what is that? Basically your ultrasound,
27:25
what we see is tiny echogenic shadowing from the wall.
27:28
Now what it is, it is an acquired,
27:31
it is a benign acquired anomaly characterized atrophy
27:34
of the mucosal epithelium
27:35
and invaginates into the interes of the
27:38
thickened muscularis forming the OENs of sinuses.
27:43
So this is basically hyperplastic polys of the gall
27:47
and on ultrasound what we see is genic, which are giving it
27:53
atail is like a tapering artifact.
27:56
So a thicker at the bottom and lower narrower
28:00
and usually suggest benign pathology.
28:04
Tiny polyps, tiny hyper
28:07
lesions which are attached to the wall.
28:10
So when you move around the patient you do your maneuvers in
28:13
various position breath.
28:15
When you see they're not moving,
28:16
that's when you label them as polyps.
28:18
And of course, uh,
28:20
less than once in media we would just follow up.
28:24
Adults do cross-sectional imaging.
28:27
The GB polyps are often asymptomatic may cause IC colleagues
28:33
and they're often incidental findings
28:35
and primary limitation to distinguish benign
28:38
and malignant is always there.
28:40
So there is no way we can tell about a histopathology
28:43
on basis of ultrasound fluid, any polyp that we see,
28:49
whatever if their patients base is not of ease and the skin
28:52
or if you think you can go
28:54
for a higher frequency transducer, try
28:57
and evaluate the detail
28:59
and get an assessment whether there is a flow or not.
29:03
That is very important.
29:09
Okay, so
29:13
what about uh, the next commonest finding
29:16
that we see every day,
29:17
which is the calcula in the gall bladder poly lithia.
29:22
Now, now sounds simple again is a an area
29:27
of a lot of discrepant reports
29:28
because patient comes, okay I got an ultrasound done at so
29:32
and so place, they said there is a gold stone.
29:35
I got an ultrasound set at another place.
29:37
They said there is not a goldstone,
29:39
I got an ultrasound from so and so.
29:41
They said the size of my stones is this.
29:43
I got done from another piece.
29:45
They said the size of the stone is this. Why this variance?
29:49
Okay, so the positioning has to be important
29:52
and for the size factor we have to understand
29:55
that the ball rather is like a peer shaped,
29:59
it's like a balloon
30:00
or a bag that you are holding if there are stones in it.
30:04
Now it depends upon which stone
30:06
and which angle of the beam are you
30:09
or uh, insulating how you would measure it.
30:13
So there would be a variance in the report
30:18
provided you try
30:19
and really do a four three dimensional orientation
30:23
of the available stone.
30:25
It is one and if they're not adherent to one another
30:28
and get the longest measurement in play
30:31
and then you freeze it and then you record it
30:33
and then you document it.
30:35
So that way you might have a reduced discrepancy in the
30:39
ultrasound imaging group coming to the gallstones.
30:42
They may be symptomatic in about 25%.
30:45
The most common presentation is with biliary colic,
30:47
right upper quadrant or epi pain, especially
30:51
after a fat rich meal.
30:54
Other symptoms include belching including ulence and uh,
31:02
and abdominal pain is often referred to the right shoulder
31:05
because of the nerve supply which is combined
31:09
uh, for that area.
31:11
And now ultrasound is a gold standard.
31:13
Now what is the pitfall telephone?
31:18
Now what is the pitfall?
31:21
There is a dirty acoustic gas shadowing
31:24
that is the commonest uh,
31:27
thank you during the time.
31:30
So it's, so we have
31:34
to differentiate the gall bladder shadowing from the uh,
31:37
gas shadowing in the area
31:39
because there's also ball in the area not to mis
31:44
misinterpret bowel shadowing with a GB shadowing
31:48
often the GP is removed
31:51
and then the shadowing in the fossa could be mis as
31:57
BCO shadow complex and a GB could be reported there.
32:01
So we have to prevent these pitfall by thorough scanning
32:06
and understand calculi is a dense shadowing
32:10
and the gas shadowing is a ring, uh, is a dirty shadowing,
32:15
meaning it's not completely dense.
32:17
So highly reproductive calculi will give highly re uh,
32:21
cal highly reflective echogenic ProCard within the Colorado
32:26
and with the normally prominent posterior caustic shadowing
32:30
regardless of the pathological type postage shadowing is
32:33
independent of the composition
32:34
and the calcium gravity dependent movement is often seen
32:39
with a change in the position.
32:41
The rolling stone side
32:43
and color doctor, as you all know when we evaluate stones in
32:47
the kidneys or anywhere demonstrates a twinkling art with
32:52
small stones on big stones, right?
32:54
So shadowing is more with more larger stones
32:59
with the higher frequency
33:01
and with the focal zone at the level of the gallstone.
33:05
So a gallstone may paradoxically be hard
33:07
to demonstrate when you have a ball eco complex
33:10
you come to that later.
33:12
So call stone will common in females.
33:16
However, when you see one pathology, what we learned,
33:20
we do not stop there.
33:22
We have to still look at more.
33:25
You see one stone, you do not stop there,
33:27
but there is another stone down in the tuck,
33:30
down in the CPD down in the PLE elevator.
33:33
So you really have to go,
33:35
especially when there are small gravel light
33:37
stones that's very important.
33:39
And the stone could be hiding a poly behind.
33:43
So that's another thing that you have to be aware of.
33:45
So this is an example of a case.
33:48
These both images belong to the same patient.
33:51
So there is a calcula with a dense postage tissue
33:54
and there are tiny little polys which are
33:57
attached to the wall as well.
33:59
So that meets the diagnosis a little bit different than just
34:03
quoting one pathology as the poly.
34:08
Of course you can sometimes have fine rebel like lar.
34:11
So it's a good idea to give some inclination to the surgeon
34:16
as to what is the approximate size of the scalp,
34:19
the scalp line that you're seeing
34:21
and where are they located funders or in the next region.
34:26
So which is very important to plan the next management,
34:32
the gas shadowing because cash shadowing is a ring down
34:35
and it's a dirty shadowing.
34:36
So you have to differentiate that from there.
34:39
You can have multiple lar you can have tiny multiple
34:42
shadowing lar you can have wall equal shadow complex,
34:46
you can have calculus impacted in the nitrogen
34:49
where you see an over distant wall bladder
34:53
and a nice large Lina.
35:00
So whenever the drainage to the gall bat is stopped, so the
35:05
the uh, the collections, the sta will lead to collection
35:09
and creation of the sludge we sl may be there.
35:13
These may include micro lids and protein
35:18
and lys cells and mucins.
35:21
You can have sludge, you can also have a polyp
35:24
or a mass, you can have small polyps.
35:26
So a thick and wall.
35:28
So that is another thing that you have to evaluate.
35:33
Now coming to the shadow complex absence
35:37
of gall bladder and just the TB fossa bowel shadowing
35:41
that is misinterpreted as a shadow complex
35:46
and of course Celine gallbladder.
35:48
Now these are three different scenarios.
35:51
These three have their own characteristic features and
35:54
and overlap of this.
35:56
This is disaster, right?
35:59
So let's understand wall equal shadow complex is a
36:03
double arc shadow sign.
36:05
The proximal hyper equ arc represents the wall of the gall.
36:09
So this hyper equate arc represents the wall of the gall
36:13
and the distal hyper equate art
36:16
represents the reflections from the gallstones, right?
36:21
And the hyper equate space in between the two.
36:25
It's a small pin sliver of fluid between of the vi
36:29
between the wall of the gall and the hyper, uh,
36:33
and the the gallstones, right?
36:36
So when error calcification is present,
36:38
the normal cul wall is not seen and only a bright eco
36:42
and the wall shadowing is that's
36:45
how we see this kind of an appearance.
36:47
So this is a classic wall eco shadow
36:50
and we'll look at the image a postal gall pattern.
36:53
Understand what's so understand here the e echogenicity was
36:58
inside the wall, it was not in the wall right now
37:02
of course scanning tears you have to go
37:04
through all the PO positions
37:06
and ask the patient to breath forward.
37:08
Now coming to this different uh, this thing uh,
37:11
call the postal gall pattern where we have
37:14
a dense calcification in the wall of the gall bladder
37:18
and we also see e echogenic contains, right?
37:22
So oline gallbladder refers
37:24
to the condition in which the inner gallbladder wall
37:26
is entrusted with calcium.
37:28
So wall becomes Britain heart and often takes a virtue.
37:32
And other names of course are calcifying cystitis
37:36
and calcified poly bladder, right?
37:40
Causes are known occurs in association
37:42
with ball stone disease
37:43
and may represent a form of cystitis.
37:45
The entire wall apart may be calcified.
37:48
Patients often have symptoms not uh,
37:51
prophylac coly technique often advised
37:54
because of there is a high in of relation with CHEB
38:00
Oma DD includes of course gallstones
38:03
and five cters coly cystitis and poly complex as well.
38:09
So here that's
38:10
because the calcification is occurring in the wall.
38:15
Now role of standing in erection,
38:18
sometimes we see a very clear movement,
38:20
we do not see a stone and we sta scan an requisition
38:24
and a stone just drops down into the front.
38:26
That's fine. Sometimes you see some hypo egos
38:31
that you think are polyps
38:34
but when you actually do a dynamic
38:36
maneuver you see oh they're boom.
38:38
Oh so it's probably just in inspirated
38:41
or collected sludge which was really looking like
38:44
as if it's a fit at that one spot
38:46
because getting vascularity
38:48
and ultrasound imaging in a Colorado poly is very
38:51
challenging unless you have a really very nice transducer
38:54
frequency and a really very lean in patient, right?
38:58
So this is a case where the these,
39:02
what we thought was polyps were actually moved in the
39:06
gravity dependence scan
39:08
or requisitions scan right now
39:11
what are the complications called stones.
39:13
People have them, people stay on them with ears,
39:16
they just come for annual follow up and like goes on.
39:20
What are the complications?
39:22
Why should we have them need to we treat them?
39:25
That's up to the surgeon.
39:26
But let's just understand
39:28
of course sometime they can lead pain, they can lead
39:31
to poly cystitis, acute chronic cholangitis
39:34
and titis when they can go
39:36
and block the obstructive jaundice called
39:41
hydroxy muco and me syndrome
39:44
and increased risk of cholangiocarcinoma
39:48
identifying the presence of the peric coly cystic fluid
39:51
is useful because it's a highly specific for GB disease
39:55
and can be seen in cases of acute cholecystitis, peric,
39:59
cystic abscess and GB perforation right
40:03
now coming to the very common diagnosis which is acute
40:06
coly cystitis.
40:08
Now how do we diagnose that?
40:11
Now the this acute coly cystitis acute inflammation,
40:14
we called greater presence pain tenderness in the right
40:17
allic poa, positive sonography sign,
40:20
which means tenderness is seen when you put your probe in
40:22
the right, uh, in the right upper upper heart.
40:26
This could be ulu coly cystitis
40:30
or a calculus coly.
40:32
So the findings all are pretty much the same.
40:35
The only difference is in calculus cholecystitis the lead
40:39
point or the cause of the precipitate is a calculus that is
40:43
impacted in the neck.
40:46
Whereas in the a calculus it's just a mucosal swelling which
40:49
has caused obstruction and STAs and again collection
40:53
and lead into all the uh, symptoms.
40:57
Basically there is an obstruction
40:59
to the tree flowing drainage of the P.
41:02
Eventually it leads to gall bladder extension,
41:05
edema of the gall bladder.
41:06
Increased luminate lash again it leads to uh,
41:11
inflammation and acute oly cystitis, right?
41:16
So what will you see? You will see there's a blockage.
41:19
You'll see the stone which is lost the blockage.
41:22
You will see the collection hypo equate sludge
41:25
and you'll see the wall edema
41:27
and you can see perico cystic fluid which is ous
41:32
or you can also see some perforation
41:34
and other complications as well, right?
41:38
And in cases of a calculus, like I discussed,
41:41
the pathologist pathophysiology results from a gradual
41:44
increase in the bowel viscosity
41:46
because of prolonged sta that leads
41:47
to functional obstruction of the system.
41:51
I have to move on faster, right?
41:54
So chronic coly cystitis lead to wall thickening
41:57
and inflammation
41:58
and piers coly cystitis is a rare form
42:01
of acute cholecystitis
42:02
where the wall undergoes necrosis inflammation
42:06
and can lead to perforation as well.
42:09
So gang greenness, coly cystitis, again you see a lot
42:13
of heterogeneously looking bald bat with a lot
42:16
of air shadowing and there's hemorrhage necrosis
42:20
and micro abscess formation in over there
42:24
and it's a seen in about, uh, you you may be able
42:27
to see the mur sonographic murphy sign.
42:30
So what are the findings?
42:31
You have str of the wall, the intinate membranes
42:35
and marked asymmetry
42:37
of the GB wall focal irregularities mass like appearances SL
42:42
paralytic fluid collection.
42:44
First the se e that we don't do not wish
42:47
to see is the TB perforations.
42:50
That's why our role in early uh, diagnosis
42:54
and appropriate management is paramount.
42:57
So how would you see a perforation of the tv?
43:00
So remember the shape, it's a nice peer shape.
43:03
So if you see something,
43:04
but if you don't see any peer shaped structure,
43:07
the funders area or any other area,
43:10
'cause fundus is the most prone area inside of perforation
43:15
and uh, you can see a focal outpouching
43:19
and the collection over there in case the perforation
43:23
like this, you can have a focal collection out pouching and
43:28
because fundus commonly has a compromise
43:32
group, okay?
43:35
So you can have micro abscesses, thickness and LA right?
43:40
Coming to the next topic, polyposis
43:43
or calculi in the CPD.
43:46
Now as we all know, that's the portal vein,
43:49
that's the P artery and that is the CPD.
43:52
And obviously we can very well demo, uh,
43:55
very well see a large calculus
43:58
and that's the same calculus in transverse section.
44:01
So this we have to demonstrate
44:04
and look at the tree, tree longitudinally
44:09
and transversely pay attention to the very d part
44:13
of the common that as passes
44:14
through the pancreatic head 10 15.
44:18
So on ultrasound you'll see cal eye,
44:20
which could be two to 20 mm.
44:21
Shadowing is difficult to eit then
44:24
with cause stone within the call ladder
44:26
because of various positioning challenges and others.
44:29
So this is the dilated CBD, these are the
44:33
IC sizes for the surgeon.
44:35
So more images of the same case.
44:38
So we can just demonstrate the CBD calculus.
44:41
So here's a case with a ca calculus in the gall bladder
44:46
and another calculus in C.
44:49
So that's why even though this calculus is a big sized
44:53
calculus, we do not stop, we still have
44:57
to have a careful look at the entire period tree, especially
45:01
as we we know by experience.
45:03
If the are tiny and small ones it becomes very important.
45:07
But a large calculus would also have a
45:10
instance small calculus.
45:14
Okay, so dilated CPD by definition more than six
45:18
and more than 10 post fully
45:21
and could be there right dilated IHPR
45:26
more than three mm we can see them
45:29
and we have to understand that what is the root cause?
45:32
That's where we need to focus.
45:34
It could be a lesion in the, in the duct,
45:38
even a small lesion which is not passed.
45:41
But beyond the wall of the CBD we could still be resectable.
45:45
So early diagnosis is very good.
45:48
So whenever you are looking at scanning,
45:51
what all should you be looking for?
45:53
A GB mass, if you see, what do we look for?
45:57
We'll look, if we see any focal lesion in the verta, we have
46:00
to look at the size, shape, societal potentiated, ex extens,
46:04
ization spelling,
46:07
and the vascularity associated ized status
46:10
of cystic duct, C, BD.
46:11
And so masses can be projecting poly
46:16
foal masses
46:18
and they could be sometimes with ill-defined margins,
46:20
heterogene genicity.
46:24
And they could be hyper foci, they could be calcifications.
46:28
So here we have a typical, uh, uh, uh, solid
46:34
heterogeneous mass, um,
46:37
projecting in the fundal region into the cold movement.
46:41
And the ball appears maybe very thickened a bit.
46:45
And yes, of course we have vascularity
46:48
seen on colored doctors.
46:49
So findings are really, really suggestive
46:51
of suspicious etiology, right?
46:55
Coming to the next
47:01
we can have larger mass lesions as well.
47:04
Okay? Now the most important thing is
47:10
a cal life could be hiding mass lesions or sludge.
47:14
So you really need to be vigilant to look
47:17
for all the pathology.
47:18
Again, MAs and Cal life could be coexisting, so you have
47:22
to look at it very carefully.
47:24
Again, mass and caite could be coexisting.
47:27
You have to look at it very carefully.
47:30
Mass could be seen as large heterogeneous lobulated
47:34
and you could see you cannot appreciate any clear interface
47:39
between the liver and the mass region here.
47:43
Possibility of direct extension into the hip hs.
47:47
Hepatic parent is highly suggested in this case.
47:51
So again, you can have mass
47:53
with the heterogeneous appearance.
47:54
Again, we have another case with the CCPT mass.
47:58
Now over a period of time the vial is, could be replaced
48:02
with mucus and you could again have both
48:04
more than one pathology.
48:06
So here's this with collection and debris within.
48:10
You could also have, again,
48:13
all throughout echogenic contents or hyper.
48:18
So depending upon the various testing you have to
48:23
use your various transducers
48:25
and various, uh, optimization skills to be able
48:30
to evaluate the gallbladder.
48:32
It's a simple organ, it's very easy to see.
48:36
And however we together can reduce the
48:41
discrepancy in the reports in the gallbladder imaging.
48:44
Thank you so much. Thank you all for joining in.
48:48
Have any questions I'll be very happy
48:50
to do. Thank you. Thank
48:52
You so much Dr.
48:53
Singal. Yes. Uh, we will now open the floor up
48:56
to any questions our audience members may have.
48:59
It looks like we have a few in the q
49:00
and a, um, already set for you Dr.
49:04
Singal, if you're able to open that up,
49:07
I'll, I'll, okay, so what do you recommend,
49:12
uh, Ms. Sanchez asking when bladder, uh, is contracted
49:16
with gall stones even
49:17
after it does fasting, just a point of chronic inflammation
49:20
and how to exclude past?
49:22
Uh, yes, probably, uh, chronically inflamed
49:25
and thickened gall bladder may be contracted.
49:27
And how to exclude carcinoma.
49:29
We really need to do a contrast CT scan
49:31
to evaluate it further
49:33
because the adenocarcinoma gall bladder can pre,
49:37
we can present as a focal thickening
49:39
or a diffuse thickening.
49:41
So the answer would be a contrast CP scan in that case?
49:44
Correct. Thank you. Another question by Dr.
49:48
Sanja is how, what
49:50
to recommend when we see por seline RA on ultrasound.
49:54
Uh, por seline ra, like I said, it's a, the
49:59
literature says there's an increased risk of malignancy
50:02
and it's all contracted.
50:04
So of course a CT scan to evaluate
50:06
and exclude any further pathology
50:08
because when you have the shadowing from the wall,
50:11
you definitely cannot evaluate the internal contains
50:15
of the gallbladder.
50:17
So ultrasound could be limited in this scenario
50:20
to evaluate internal detail of the OSA lining
50:24
for any pathology and a contrast CT scale is needed
50:29
to solve the case.
50:31
Thank you. And uh, we've done that as well.
50:35
So next is, is there any significance
50:38
to count the gallstones in the report
50:40
or just give a size range
50:42
or just give the largest dimension?
50:45
Now this is an area where patients insist
50:48
and patients go, uh, left, right and center.
50:51
They say that so and so has already told me.
50:54
Now is there an increase or a decrease?
50:57
There's a lot of patient counseling that goes on about it.
51:00
I do try and explain that look, if I take a bag
51:03
and if I fill it with some coins
51:06
and then it would depend on which angle am I looking at
51:09
and if there's so many coins which are clustered together,
51:13
how can we really say until, unless someone pulls out
51:17
and spreads the coins on the table.
51:20
I like, I don't use the word coins,
51:22
I use a bag full of diamonds.
51:24
Like, you know, until someone spreads them you cannot.
51:27
So you'll have a difference in the reported however,
51:30
because um, more than 10 millimeter,
51:34
I think they have a surgical management criteria
51:37
and especially if they are four, five millimeter,
51:40
they could easily trickle down the CBD.
51:43
So understanding, uh, of the,
51:47
if there is a large calculus
51:48
and if it's impacted in a particular location,
51:51
it'll really irritate the muy that point
51:54
and it may lead to, uh, carcinoma neoplastic changes.
51:57
So again, that needs to be mentioned that it's an impacted
52:01
and this is the size of the calculus in
52:03
the largest field of you.
52:04
And especially when you have those gravel like which are
52:07
photo six millimeters, they're very vulnerable
52:10
to drop down into the duct
52:12
and lead to pancreatitis, lead to various other scenarios
52:16
and uh, the clinical medical emergency and other scenarios.
52:22
I do give a range.
52:24
I do give, I do not give the number more so than the sizes.
52:29
I do give few calculi because you couldn't,
52:32
unless there is a really sure that there's a single calculus
52:35
or then I would say a few calculi or a single calculi.
52:39
So I wouldn't go about the numbers,
52:41
but I would say, uh, they measure approximately
52:45
or between four to eight mm or between uh, eight to 12 mm
52:50
or between 15 to th 30.
52:53
So whatever is my uh, uh, I measure and the largest
52:58
and or the smallest and then I give a range.
53:02
I try and give some clinical picture to the clinician
53:06
and to the patient so that they have some answer,
53:09
but mainly to the clinician to understand
53:11
that if they're very small they need to watch out
53:13
for them trickling down.
53:15
If they're very large and irritating there, they could lead
53:18
to new changes because they're not moving around.
53:21
So like, you know, if some fixed
53:23
and irritating there if you need to mucosal changes.
53:27
Thank you. Thank you for your question.
53:32
Do you make a disclaimer in the report for the patients
53:34
who are difficult to reposition?
53:38
Um, technically we don't do that.
53:42
So it's up to you
53:44
or whichever way your practice is working smoothly,
53:48
whatever is the law of the land,
53:50
whatever, whatever works for you.
53:52
If there's a practice for disclaimer,
53:54
so we do it if they're difficult to reposition,
53:57
so we just uh, I mean you would just say limited scan,
54:00
you do inate patient positioning.
54:03
So this is not evaluated.
54:05
You would, uh, you would I would say yeah,
54:07
patient is non fasting so you've not evaluated that area.
54:11
You would add that and
54:13
because many times the patients would come
54:15
for a whole abdominal scan, they're coming
54:17
for a renal colleague and uh, patient is non fasting.
54:19
So the intention is not for that area,
54:22
but it's just for the kidney stone.
54:24
But you are still doing the whole abdomen
54:26
so you would make a note for that.
54:27
Right. Okay.
54:30
So where, so disclaimer. Yeah, it's up to you.
54:34
Right. So the next thanks
54:36
for the great talk, thank you so much.
54:38
Do you have any experience, uh, using point
54:40
of service pocket your scanners for evaluation of the gp?
54:44
Uh, in our country point of uh, uh, US scanners are not, uh,
54:49
authorized, uh, to be used.
54:50
So we use the, the, the big,
54:56
big state of art equipment.
54:58
We are blessed to use that. So thank you so much.
55:02
And what's the prevalence of pseu? Lithia?
55:05
I dunno, that's a term that I'm actually seeing it now.
55:08
So you mean soft calculi without any acoustic shadowing?
55:12
I I actually use that same descriptor.
55:15
I see genic archive which are mobile
55:17
but I do not see an acoustic shadowing.
55:20
So they could be in the, along the pathophysiology
55:25
but they're not casting the test acoustic shadowing.
55:27
They may resolve over a period of time
55:29
or they may lead to dense.
55:32
I use that terminology.
55:34
Could you tell the standard views
55:35
and positions of the call value evaluation?
55:37
Very information lecture.
55:39
Thank you so much for the compliment.
55:42
Standard views are longitudinal and transfer.
55:44
So for any organ in ultrasound imaging we get the
55:48
longitudinal span of the organ
55:50
and we get the appendicular scan and so, but
55:53
however it's a three dimensional scan
55:55
so people scan the organ from top to the bottom left
55:59
to right in all different sleeves.
56:01
So assuming that we've covered the 360 degree in all volume
56:05
of the tissue, thank you.
56:08
How to differentiate a small polyp
56:11
and stones in the reta mobility DY scan, as simple as that.
56:16
Polyps could not what to do if TB does not distend
56:20
after overnight fast tb.
56:22
While thickness is normal, she is not standing.
56:25
Is that for a pediatric population
56:27
or is that for an adult population?
56:29
If you're looking at raia, which should not distant
56:32
after overnight fasting, pediatric will not do.
56:36
But if an adult, if it's not dis descending
56:38
after overnight fast, probably due to chronic ly cystitis,
56:41
it's inflamed contrast and you may need to do a ct.
56:46
Lets exclude any other pathology
56:49
to patients with fatty liver.
56:51
Uh uh, this patient with fatty liver.
56:54
A contributing factor for gallstones
56:55
or that's in pathophysiology.
56:58
Remember we reading that in a pet email of 40.
57:01
So I guess obesity is a factor.
57:05
Okay, so US guided drainage of tb.
57:08
Uh, drainage of tb you mean?
57:12
I'm not into uh, doing uh, any of those interventions?
57:16
No, the contribution
57:19
of contrast enhanced ultrasound.
57:22
Okay, it's there. It's developing, it's there.
57:25
We, we use it for the liver for focal hepatic lesions
57:29
to do the RFPAs
57:30
and to um, uh,
57:33
follow the lesions in CC post RFA
57:37
but uh, we use it for gall battle.
57:39
We haven't done that as at our you.
57:43
Thank you for the wonderful presentation.
57:45
Could you please do you know istic neck
57:48
of the gallbladder causing pain?
57:51
Oh you mean like aion of the gall bladder? I can wait.
57:55
Interesting to see a case. Thank you so much though.
58:00
Okay. How to frame sludge in a mouse in corporate, again,
58:03
mobility dynamic scan.
58:04
So sludge would move.
58:06
Uh, that's uh, I had a case where there was a sludge,
58:08
which I thought was a mouse and then uh, uh, it moved
58:12
or it all moved in the dynamic imaging.
58:15
Thank you for your question.
58:17
Do you give a diagnosis that says
58:19
considered entrusted stone versus partially calcified polyp?
58:24
I wouldn't give that. That's like, uh, uh,
58:30
I would give, if I think it's a polyp, I would give a polyp.
58:32
I would give a uh, echogenic.
58:35
I would no shadowing soft
58:37
or I would give cal lab, which for doing
58:42
how can I differentiate oline from sludge.
58:46
Oline is in the wall
58:47
and sludge is in the inside the contain.
58:50
They're both different things.
58:54
Hence ma'am, it was very informative.
58:56
I gained a lot of knowledge. Thank you Dr. Bhe.
58:59
And uh, I read somewhere normal
59:04
CND thickness is four.
59:05
Uh, you mean common bile duct?
59:07
Uh, I have four to six.
59:11
Mm like depends upon the age long ary
59:14
for GB and complication.
59:17
Uh, not no of that. Thank you so much.
59:21
Thank you for all your questions.
59:25
One more has just popped in how to differentiate
59:27
to mucosal and sludge.
59:29
So basically sludge is just a collection of the bile, right?
59:32
But over a period of time they say the sludge ites
59:35
and it becomes like a mucus collection.
59:38
So an ultrasound basis you cannot really say in MUE is when
59:42
you actually operate
59:44
and you pull it out that it's just a mucus, it's not bile,
59:47
that's when it's to pathologically on the table le
59:50
but on its can be impacted
59:52
and over a period of time it can become like
59:54
a MUE or dis standard.
59:55
That, thank you. That was your question answered.
59:59
Thank you so much. All done.
60:02
Alright. Thank you so much everyone for,
60:04
for participating today, uh, in our noon conference.
60:07
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60:10
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60:15
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60:18
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60:21
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60:23
deliver a lecture entitled Introduction
60:25
to Arthritis part two.
60:27
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60:30
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60:34
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