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Training Collections
Library Memberships
On-demand course library with video lectures, expert case reviews, and more
Fellowship Certificate™ Programs
Practice-focused training programs designed to help you gain experience in a specific subspecialty area.
Ultimate Learning Pass
Unlock access to our full Course Library and all self-paced Fellowships.
Continuing Medical Education (State CME)
Complete all of your state CME requirements in one convenient place.
Noon Conference (Free)
Get access to free live lectures, every week, from top radiologists.
Case of the Week (Free)
Get a free weekly case delivered right to your inbox.
Case Crunch: Rapid Case Review (Free)
Register for free live board reviews.
Dr. Resnick's MSK Conference
Learn directly from the MSK Master himself.
Lower Extremities MRI Conference
Musculoskeletal Imaging
PET Imaging
Pediatric Imaging
For Training Programs
Supplement your training program with case-based learning for residents, registrars, fellows, and more.
For Private Practices
Upskill in high growth, advanced imaging areas.
Compliance
NewTrack, fulfill, and report on all your radiologists' credentialing and licensing requirements.
Emergency Call Prep
Prepare trainees to be on call for the emergency department with this specialized training series.
1 topic, 2 min.
45 topics, 2 hr. 39 min.
Introduction to Pancreas Imaging
2 m.Anatomy of the Pancreas
3 m.MRI Protocol (Pancreas)
6 m.Embryology (Pancreas)
4 m.Annular Pancreas Summary
2 m.Annular Pancreas on MRI
3 m.Ectopic Pancreas
3 m.Broad Classification of Pancreatic Lesions
2 m.Adenocarcinoma: Surgical Perspective
10 m.Resectable Pancreatic Head Tumor
7 m.Nonresectable Pancreatic Tumor with Perineural Invasion
8 m.Nonresectable Pancreatic Head Tumor with Liver Metastases
5 m.The Whipple Procedure (Pancreas)
1 m.Post Whipple Procedure on MRI
6 m.Differentiating Between Pancreatitis and Adenocarcinoma
4 m.Mass or Pancreatitis: Chronic Pancreatitis
5 m.Mass or Pancreatitis: Proven Chronic Pancreatitis
5 m.Groove Pancreatitis Summary
3 m.Groove Pancreatitis or Adenocarcinoma: Adenocarcinoma
4 m.Autoimmune Pancreatitis Type I Vs. Type II
4 m.Mass, Pancreatitis, or Cancer: Autoimmune Pancreatitis
7 m.IPMN Summary
8 m.Main Duct IPMN
4 m.Mixed IPMN
4 m.Malignanttransformation of main duct IPMN
3 m.Obstructive Chronic Pancreatitis
5 m.Malignant Sidebranch IPMN
3 m.Spontaneously Ruptured IPMN
3 m.Pancreatic Cystic Tumor Summary
4 m.Serous vs. Mucinous vs. SPEN Tumors
2 m.Serous Tumor, Side Branch IPMN
3 m.Sidebranch IPMN/Mucinous Tumor mimicking Serous Tumor
4 m.Classic Serous Tumor in Pancreatic Head
2 m.Mucinous Tumor (Pancreas)
3 m.Malignant Transformation of Mucinous Tumor
5 m.Classic SPN (SPEN)
3 m.NET Summary (Pancreas)
2 m.NET (Pancreas)
3 m.Cystic Necrosis of the NET vs. SPEN
4 m.Non-functional Malignant NET
5 m.Metastasis (Pancreas)
1 m.Pancreatic Metastasis
4 m.Metastasis to Pancreatic tail, RCC
6 m.Schwannoma (Pancreas)
3 m.Intrapancreatic Splenule
4 m.0:03
with a pancreatic mass, which
0:05
we have to characterize now.
0:07
And I will start with T1 fat-suppressed,
0:11
non-contrast-enhanced images here.
0:14
And we see this expansive large mass in
0:16
the pancreatic tail, which shows multiple
0:19
areas of heterogeneous T1 hyperintensities
0:23
along with the periphery of this lesion.
0:26
So that gives us an idea that possibly
0:27
this lesion actually has some internal
0:30
hemorrhages or hypertense content.
0:34
And if we correlate this with T2-weighted images,
0:38
we find that this, this entire thing which was
0:41
looking T1 hyperintense actually is T2 hyper
0:44
intense, so that further reinforces our thought
0:48
that it is possibly hemorrhagic content here.
0:51
But this tumor looks like
0:52
necrotic at multiple locations.
0:54
It is very big espine cell involving the
0:58
pancreatic tail in a young female patient.
1:01
So that is a daughter tumor.
1:04
That means it is SPN.
1:07
So, let us see how it behaves
1:08
in the arterial phase.
1:10
On the arterial phase, we can see
1:12
most of the tumor is not enhancing,
1:14
only the periphery is enhancing.
1:18
And then, if we go to the venous
1:21
phase, we can see it better, the non
1:24
enhancing part and enhancing part.
1:27
But if we compare it with the, the one we
1:29
started with the arterial phase, if we see the
1:32
enhancement pattern on the arterial phase, the
1:34
periphery of this lesion is enhancing like a rim.
1:37
Like, there is a claw formed here, and
1:40
almost everything along with the periphery is
1:41
enhancing, but the center is not enhancing.
1:43
It's mostly necrotic, mostly it
1:45
is replaced by hemorrhagic tissue.
1:48
The point is, given the age, given
1:50
the presentation along with the tail,
1:53
given the gender, it favors it as SPN.
1:56
But sometimes the same appearance can
1:57
be seen with neuroendocrine tumors.
1:59
Those are non-functional.
2:00
They can grow up to this extent and
2:02
they can undergo necrosis as well.
2:04
But usually that necrosis is not
2:05
that striking as in this case is.
2:09
So, in the case of neuroendocrine tumor,
2:11
when it is necrotic, you will still see
2:12
some of the enhancing soft tissue, and that
2:14
will enhance more during the arterial phase.
2:17
And then, non-functional neuroendocrine
2:19
tumor, still you can have tumor markers.
2:21
And patient age group can be
2:22
different, patient can be male as well.
2:24
But in this particular patient, given this
2:26
patient's presentation and the appearance of
2:28
the tumor, uh, we gave the diagnosis of SPN, and
2:32
it was proven on the postoperative pathology.
Interactive Transcript
0:03
with a pancreatic mass, which
0:05
we have to characterize now.
0:07
And I will start with T1 fat-suppressed,
0:11
non-contrast-enhanced images here.
0:14
And we see this expansive large mass in
0:16
the pancreatic tail, which shows multiple
0:19
areas of heterogeneous T1 hyperintensities
0:23
along with the periphery of this lesion.
0:26
So that gives us an idea that possibly
0:27
this lesion actually has some internal
0:30
hemorrhages or hypertense content.
0:34
And if we correlate this with T2-weighted images,
0:38
we find that this, this entire thing which was
0:41
looking T1 hyperintense actually is T2 hyper
0:44
intense, so that further reinforces our thought
0:48
that it is possibly hemorrhagic content here.
0:51
But this tumor looks like
0:52
necrotic at multiple locations.
0:54
It is very big espine cell involving the
0:58
pancreatic tail in a young female patient.
1:01
So that is a daughter tumor.
1:04
That means it is SPN.
1:07
So, let us see how it behaves
1:08
in the arterial phase.
1:10
On the arterial phase, we can see
1:12
most of the tumor is not enhancing,
1:14
only the periphery is enhancing.
1:18
And then, if we go to the venous
1:21
phase, we can see it better, the non
1:24
enhancing part and enhancing part.
1:27
But if we compare it with the, the one we
1:29
started with the arterial phase, if we see the
1:32
enhancement pattern on the arterial phase, the
1:34
periphery of this lesion is enhancing like a rim.
1:37
Like, there is a claw formed here, and
1:40
almost everything along with the periphery is
1:41
enhancing, but the center is not enhancing.
1:43
It's mostly necrotic, mostly it
1:45
is replaced by hemorrhagic tissue.
1:48
The point is, given the age, given
1:50
the presentation along with the tail,
1:53
given the gender, it favors it as SPN.
1:56
But sometimes the same appearance can
1:57
be seen with neuroendocrine tumors.
1:59
Those are non-functional.
2:00
They can grow up to this extent and
2:02
they can undergo necrosis as well.
2:04
But usually that necrosis is not
2:05
that striking as in this case is.
2:09
So, in the case of neuroendocrine tumor,
2:11
when it is necrotic, you will still see
2:12
some of the enhancing soft tissue, and that
2:14
will enhance more during the arterial phase.
2:17
And then, non-functional neuroendocrine
2:19
tumor, still you can have tumor markers.
2:21
And patient age group can be
2:22
different, patient can be male as well.
2:24
But in this particular patient, given this
2:26
patient's presentation and the appearance of
2:28
the tumor, uh, we gave the diagnosis of SPN, and
2:32
it was proven on the postoperative pathology.
Report
Faculty
Neeraj Lalwani, MD, FSAR, DABR
Professor and Chief of Abdominal Radiology
Montefiore Medical Center, New York
Tags
Pancreas
Neoplastic
MRI
Body
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