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Training Collections
Library Memberships
On-demand course library with video lectures, expert case reviews, and more
Fellowship Certificate™ Programs
Practice-focused training programs designed to help you gain experience in a specific subspecialty area.
Ultimate Learning Pass
Unlock access to our full Course Library and all self-paced Fellowships.
Continuing Medical Education (State CME)
Complete all of your state CME requirements in one convenient place.
Noon Conference (Free)
Get access to free live lectures, every week, from top radiologists.
Case of the Week (Free)
Get a free weekly case delivered right to your inbox.
Case Crunch: Rapid Case Review (Free)
Register for free live board reviews.
Dr. Resnick's MSK Conference
Learn directly from the MSK Master himself.
Lower Extremities MRI Conference
Musculoskeletal Imaging
PET Imaging
Pediatric Imaging
For Training Programs
Supplement your training program with case-based learning for residents, registrars, fellows, and more.
For Private Practices
Upskill in high growth, advanced imaging areas.
Compliance
NewTrack, fulfill, and report on all your radiologists' credentialing and licensing requirements.
Emergency Call Prep
Prepare trainees to be on call for the emergency department with this specialized training series.
3 topics, 25 min.
19 topics, 1 hr. 21 min.
Sturge Weber Syndrome
6 m.Sturge Weber Syndrome: Ribbon-like Cortical Calcification
3 m.Sturge Weber Syndrome: Progression of Disease
4 m.Glaucoma in the Setting of Sturge Weber Syndrome
6 m.Sturge Weber Calcifications Mistaken for Blood Products
4 m.Bilateral Occipital Sturge Weber Syndrome
4 m.Von Hippel-Lindau Disease (VHL) Nerve Conduction Study (NCS)
7 m.Von Hippel-Lindau Disease (VHL), Recurrent Non-Cystic Hemangioblastomas
5 m.McCune Albright Syndrome
7 m.CLOVES Syndrome
5 m.Joubert Syndrome
4 m.Dandy-walker Malformation
4 m.Chiari I
4 m.Chiari II
5 m.Chiari III
5 m.Ehlers-Danlos
7 m.Marfan Syndrome
2 m.Horner's Syndrome with Carotid Dissection
6 m.Horner's Syndrome
5 m.3 topics, 14 min.
11 topics, 44 min.
0:01
So here's a patient that presents with
0:02
a mass involving their oral tongue.
0:05
And when we look at the non-contrast T1-
0:07
weighted image, we really don't see much.
0:09
Now, maybe with the subtle leap of faith, you
0:12
can suggest that some of the fat here involving
0:15
the left half of the oral tongue is obliterated
0:17
on the right, but it's sort of a tough call.
0:20
But anyway, so it's essentially
0:21
intermediate signal on T1.
0:23
On T2-weighted images,
0:24
we can see that it's very, very bright.
0:26
And when we give contrast,
0:28
there's no enhancement whatsoever.
0:29
So this tells us that we're dealing with
0:32
a cystic mass involving the oral cavity,
0:36
specifically involving the oral tongue.
0:39
When we look at the diffusion-weighted sequences, we
0:43
can see that it's high signal on the DWI sequences.
0:47
And when we look at the ADC map, we
0:49
can see that there is increased signal
0:51
on ADC, probably due to shine-through.
0:54
So what I'm showing here right now is a cystic
0:57
lesion involving the floor of the mouth.
0:59
The patient does not have any history of
1:01
fever, and it's been there for a long time.
1:04
So this is a congenital lesion.
1:06
It's a congenital vascular lesion,
1:09
and this is a lymphatic malformation.
1:12
Now the other differential diagnosis that has
1:14
to be considered when you see something like
1:16
this on the T2 sequences, the non-contrast
1:20
T1 and the contrast-enhanced T1
1:22
sequences is the possibility of an epidermoid.
1:26
So how do we differentiate between a
1:28
lymphatic malformation and an epidermoid?
1:32
Where we look at the diffusion sequences.
1:34
So this is high signal on DWI, but the fact that
1:37
the ADC value is high signal, that suggests
1:41
a lymphatic malformation.
1:43
If the ADC value was decreased, then this would
1:46
suggest that we're dealing with an epidermoid.
1:49
And when we look at the dynamic sequences, we
1:52
can see that there's no enhancement whatsoever.
1:55
And this confirms that this cystic lesion
1:58
does not have an appreciable vascular supply.
2:01
So before, when I talked about the four
2:03
main components, which were arterial,
2:05
venous, lymphatic, and capillary,
2:08
we can see there's no enhancement.
2:10
So this really is a lymphatic
2:12
malformation involving the oral tongue.
Interactive Transcript
0:01
So here's a patient that presents with
0:02
a mass involving their oral tongue.
0:05
And when we look at the non-contrast T1-
0:07
weighted image, we really don't see much.
0:09
Now, maybe with the subtle leap of faith, you
0:12
can suggest that some of the fat here involving
0:15
the left half of the oral tongue is obliterated
0:17
on the right, but it's sort of a tough call.
0:20
But anyway, so it's essentially
0:21
intermediate signal on T1.
0:23
On T2-weighted images,
0:24
we can see that it's very, very bright.
0:26
And when we give contrast,
0:28
there's no enhancement whatsoever.
0:29
So this tells us that we're dealing with
0:32
a cystic mass involving the oral cavity,
0:36
specifically involving the oral tongue.
0:39
When we look at the diffusion-weighted sequences, we
0:43
can see that it's high signal on the DWI sequences.
0:47
And when we look at the ADC map, we
0:49
can see that there is increased signal
0:51
on ADC, probably due to shine-through.
0:54
So what I'm showing here right now is a cystic
0:57
lesion involving the floor of the mouth.
0:59
The patient does not have any history of
1:01
fever, and it's been there for a long time.
1:04
So this is a congenital lesion.
1:06
It's a congenital vascular lesion,
1:09
and this is a lymphatic malformation.
1:12
Now the other differential diagnosis that has
1:14
to be considered when you see something like
1:16
this on the T2 sequences, the non-contrast
1:20
T1 and the contrast-enhanced T1
1:22
sequences is the possibility of an epidermoid.
1:26
So how do we differentiate between a
1:28
lymphatic malformation and an epidermoid?
1:32
Where we look at the diffusion sequences.
1:34
So this is high signal on DWI, but the fact that
1:37
the ADC value is high signal, that suggests
1:41
a lymphatic malformation.
1:43
If the ADC value was decreased, then this would
1:46
suggest that we're dealing with an epidermoid.
1:49
And when we look at the dynamic sequences, we
1:52
can see that there's no enhancement whatsoever.
1:55
And this confirms that this cystic lesion
1:58
does not have an appreciable vascular supply.
2:01
So before, when I talked about the four
2:03
main components, which were arterial,
2:05
venous, lymphatic, and capillary,
2:08
we can see there's no enhancement.
2:10
So this really is a lymphatic
2:12
malformation involving the oral tongue.
Report
Description
Faculty
Mahan Mathur, MD
Associate Professor, Division of Body Imaging; Vice Chair of Education, Dept of Radiology and Biomedical Imaging
Yale School of Medicine
Tags
Retroperitoneum
MRI
Gastrointestinal (GI)
Body
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