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Wk 5, Case 1 - Review

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0:00

So the vast majority of you, uh,

0:03

identified the various fractures on this cervical spine case.

0:08

Obviously at, at Hopkins, like with most institutions,

0:11

things start with a CT scan of the cervical spine with patients who have trauma,

0:16

this patient, um, how they fall.

0:22

And, uh,

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what you notice on the sagittal reconstructed image is

0:27

that there is widening of the interspinous space here

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between, uh, 2, 3, 4, 5, 6, and seven.

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And then as we go off midline,

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we can see the plane of the fracture through the superior facet of C

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seven.

0:48

We see that this facet on the left side is

0:53

perched. It's, um, one on top of the other,

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but as we go off to the contralateral side, the right side,

1:03

it's, um, actually jumped.

1:06

So now the superior facet is behind that of the inferior facet

1:11

of C six as opposed to being kind of perched there.

1:16

The, the, it doesn't really make a difference with the terminology here,

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it's just a matter of degree. Um, what you're not seeing,

1:25

which is a little unusual,

1:27

is offset anteriorly in the spinal column.

1:32

Um,

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this is an unstable fracture and it may be that in the collar that the

1:37

patient is in, the patient is, um,

1:41

stable with in the collar, but unstable, uh,

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as soon as you take 'em outta the collar. And so, uh, for this reason,

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um, the patient will remain in the collar and this neurosurgeons will assess the

1:53

patient on the axial scans.

1:57

We're at a little bit of a peculiar, uh, plane here, um,

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for the frac, uh, for, for the angulation.

2:07

But nonetheless, as you go, um, through here,

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you can see the fracture on the left side. This is the lamina of the facet,

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and then into the facet here.

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And on the contralateral side,

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you actually have a portion of the pedicle that's involved, um,

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as well as the, um, the facet.

2:30

And here again,

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it's a little bit of obliquely through the pedicle on the contralateral side as

2:35

well. So, um,

2:41

the Corona, I'm not sure helped us very much

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for, you know, identifying the disease and the fractures.

2:56

So in, in this setting, um, two things usually happen.

3:00

One thing is that we usually recommend an MRID for ligamentous injury, uh,

3:05

to determine whether there is additional injury, uh, to the bones,

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for example, that may not be seen on the, uh, on the CT scan.

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And if the fracture does enter the frame and verium,

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which this one apparently does not, we would also recommend,

3:24

excuse me, we'd recommend the CTA and look for vertebral artery dissection.

3:29

So this patient did go onto,

3:33

go onto an MRI again. So, um,

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most of the time the main focus is on the stir image. As you know,

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the stir has the fat suppress that allows us to see the amount of edema that's

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in these soft tissues, as well as looking at the ligamentum,

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uh, ligamentous injury. So here's our distance between, um,

3:56

C six and C seven,

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and you can see the bright single intensity and the interspinous ligament. And,

4:02

um, what we're also seeing is here in the ligament and flamm,

4:07

you've got some bright signal intensity. It's, it's discontinuous at this,

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uh, C seven level. Um,

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normally we would see this darker line continuously here,

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and here we have a little gap in that dark line indicating that there is

4:23

some abnormality, uh, identified there. Um,

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sometimes what you'll also see is this bright signal intensity in the nplate.

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That's often a stress injury or bone edema associated with a fracture that may

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or may not be evident on the CT scan. The problem is, of course,

4:44

that while patients in this age group, um,

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also have DJD and the DJD Nplate disease may be bright on

4:52

the T two A scan when it's MODIC type one.

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So you look at the T one way image and, you know, look and see whether there's,

4:59

um, similar findings on T one,

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the dark one T one corresponding with the bright on stir,

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which would be the modic type one changes. So sometimes we, we end up hedging,

5:10

you know, I'm not really sure. Um, here you can see,

5:15

you know, some brighter signal intensity. The superior nplate,

5:18

I think this is of T 1, 2, 3, 4, 5, 6, 7 T one. Yeah.

5:22

So with the adjacent injury,

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you'd probably be concerned that there may be additional nplate, uh,

5:28

stress injury. Um, what you're not seeing is, uh,

5:32

cord signal abnormality, which is a good thing in this, in,

5:37

in this case. And on that sagittal,

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you wanna make sure that things haven't moved because there

5:45

is certainly a propensity for moving. Uh, this is the not,

5:50

this is not the non stir T two,

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and maybe this shows in this particular case,

5:57

The, um,

5:58

defect in the ligament and flam right here a little bit more

6:03

clearly than the, um, stir, for example.

6:07

And then you have your facet joint injury over here and here.

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So, uh, pretty routine, uh, case of how we would handle a patient with,

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uh, such a fracture with demonstration of both the, uh,

6:23

fracture on the CT as well as the mr and the result in injury to the ligaments.

6:29

Um, some people make, uh, comments about the supra spinous ligaments,

6:33

which are the ligaments that connect the is processes in this case. They,

6:37

there also seems to be bright signal intensity involving them.

6:40

And then all this is just sort of soft tissue muscular injury on the, uh,

6:44

on the stir. So, um,

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case number one, cervical spine fracture jump facet C seven,

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uh, C 67.

6:57

Any questions about the cervical spine case

7:04

on these T two weighted images, you will, um,

7:07

you should look at the vertebral artery in the wall,

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the vertebral artery to identify whether there's any evidence of dissection.

7:13

We don't do routinely for trauma axial

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T one weighted fat SAT scans,

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which are the best for looking for dissection. Um,

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but that's not part of our cervical spine protocol.

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If they specifically ask for rollout dissection,

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we will add a fats SAT T one to look for bud products in the wall

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of the vessels, be it the carotids or the Burts.

7:42

How do you tell if the spine is unstable? So we're still using the,

7:45

the le level of the, um, the columns,

7:50

um, methodology. Um,

7:52

I think that you had in your lecture with

7:57

Francis, uh, he went through a little bit more sophisticated, uh,

8:01

analysis of how you tell whether a spine is unstable. Uh,

8:05

there's all kinds of systems. Uh, I'm still old school too calm,

8:11

unstable. And, um, in this situation with, um,

8:16

not having the posterior ligament, the, the, uh, the, um,

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with only having the single column involvement by the columns,

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it would be a stable, um, injury.

8:33

But bilateral facet fractures and bilateral pedicle fractures is

8:38

unstable. So in this case,

8:40

the single column involvement I I is not as helpful

8:45

in this, in this specific case.

Report

Patient History
Fall with concern for cervical spine trauma.

Findings:
CT Cervical spine:

Acute mildly distracted right pedicle fracture of C7 with jumped/dislocated C6 inferior articulating facet with regard to the C7 superior articulating facet. The comminuted fracture likely extends to the right transverse foramen, although there is some artifact in this region. Acute comminuted mildly distracted fracture through the left C7 pedicle extending into the articular pillar with comminution and mild displacement of the superior articulating facet fracture component more than inferior articulating facet component. There is no significant associated spondylolisthesis at C6-C7. There is widening of the C6-C7 interspinous interval and mild anterior offset of the spinolaminar line on the right at C6-C7.

Irregularity of the anterior superior T1 endplate, with no definite acute fracture lucency identified, although evaluation is limited due to post observation and motion artifact.

No prevertebral edema.

Degenerative changes: Posterior osteophytic spurring and disc bulging contributing to varying degrees of mild to moderate canal stenosis, most prominent at C5-C6, where it is moderate. Multilevel uncovertebral and facet hypertrophy contributing to marked right and moderate left foraminal stenosis at C3-C4, marked right and mild left foraminal stenosis at C4-C5, and marked bilateral foraminal stenosis at C5-C6.

Normal thyroid, submandibular glands and parotid glands.

Partially imaged groundglass opacities in the upper lungs.

Additional findings: Multifocal calcific atherosclerosis.

Impressions
Acute bilateral C7 pedicle fractures, with comminution and extension into the left C7 articular pillar and left C6-C7 and C7-T1 facet joints. There is comminuted extension through the right transverse foramen at C7. Jumped right C6-C7 facets without significant spondylolisthesis.

Anterior superior endplate deformity of T1 may simply reflect osteophytic change, but is poorly evaluated due to artifact and mild compression fractures not excluded.

Findings
MR:

Redemonstrated acute fractures of the bilateral C7 pedicles with jumped right C6-C7 facet joint and fracture of the articular pillar on the left with subluxation.

Extensive T2/STIR hyperintensity within the posterior paraspinal soft tissues and along the interspinous regions most prominent at the C6-C7 level and to a lesser degree at C5-C6 and C7-T1. Minimal linear T2 hyperintensity in the prevertebral region.

Attenuated appearance of the posterior longitudinal ligament at the C6-C7 level with corresponding increased C6-C7 interspinous interval (for example seen on image 9-10 of series 3). Trace amount of fluid signal just dorsal to the posterior longitudinal ligament at this level may represent trace extra-axial blood product (seen on image 9 of series 5 closed).

T1 hypointensity along the anterior superior T1 vertebral body with
corresponding T2/STIR hyperintensity, likely representing bony contusion. No other evidence of acute vertebral body fracture or height loss in the other cervical vertebrae. There is chronic appearing inferior endplate deformity and sclerosis at the C5 level with osteophytic spurring.

The cervical cord is mildly indented along its ventral surface at multiple levels related to degenerative spondylotic changes, but no abnormally increased T2 signal is identified throughout the cervical spinal cord. No frank spinal cord compression is noted although there is degenerative spinal canal narrowing.

C2-C3: No spinal canal narrowing or right foraminal stenosis. Hypertrophic facet arthropathy is present with mild left foraminal narrowing..

C3-C4: Mild spinal canal narrowing related to circumferential disc bulging and ligamentum flavum thickening. Mild bilateral neuroforaminal stenosis due to uncovertebral and facet hypertrophy.


C4-C5: Mild to moderate spinal canal narrowing due to circumferential disc bulging with small central protrusion. Moderate right and mild left neuroforaminal stenosis due to uncovertebral and facet hypertrophy, more on the right.

C5-C6: Moderate to severe spinal canal narrowing due to circumferential disc bulge with broad-based central protrusion, with endplate osteophytic spurring that is better seen on prior CT. Severe bilateral neuroforaminal stenoses due to uncovertebral
hypertrophy and extension of disc bulging.


C6-C7: Circumferential disc bulging without significant central spinal canal narrowing. Mild right and moderate left neuroforaminal stenosis due to uncovertebral hypertrophy.


C7-T1: No significant neural foraminal stenosis or spinal canal narrowing.

Tiny T1 hyperintense foci along the undersurface of the right tentorial leaflet (images 12 and 13 on series 4), likely represent tiny incidental lipomas.

Impressions
1. Redemonstrated acute fractures of the bilateral C7 pedicles with fracture on the left extending into the articular pillar, characterized to better advantage on recent performed CT and likely without significant change. Stable jumped right C6-C7 facet joint.
2. Attenuated appearance of the posterior longitudinal ligament at C6-C7 with corresponding widening of the C6-C7 interspinous interval raises concern for ligamentous injury. Trace fluid signal just dorsal to the posterior longitudinal ligament at this level may represent a trace amount of extra-axial blood product.
3. T1 hypointensity along the anterosuperior T1 vertebral body with corresponding T2/STIR hyperintensity, likely representing bony contusion. No other evidence of acute vertebral body fracture or height loss.
4. No abnormally elevated signal within the cervical cord to suggest cord injury.
5. Extensive signal intensity within the posterior paraspinal soft tissues and interspinous regions most prominent at the C6-C7 level, likely reflecting interspinous ligament is injury.
6. Multilevel degenerative changes in the cervical spine with spinal canal and foraminal stenosis. There is moderate to severe canal stenosis at the C5-C6 level related to disc and endplate spurring.

Case Discussion

Faculty

David M Yousem, MD, MBA

Professor of Radiology, Vice Chairman and Associate Dean

Johns Hopkins University

Joshua P Nickerson, MD

Associate Professor of Neuroradiology

Oregon Health & Science University

Francis Deng, MD

Assistant Professor of Radiology and Radiological Science

Johns Hopkins University School of Medicine

Tags

Trauma

Neuroradiology

MRI

CT

Brain