Interactive Transcript
0:00
We'll open it up to any questions.
0:02
Yeah. So, so Brady, thank you.
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I mean, great cases, some of 'em a little bit complex,
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but let me, uh, we have a few minutes for questions
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and hopefully some answers.
0:12
So Brady, I'm gonna start with you, uh, submitted questions.
0:16
There's two, regarding the femoral tunnel screw,
0:20
is it crucial to be at 10 o'clock, two o'clock?
0:25
What about the position of the femoral screw
0:28
with ACL reconstructions?
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So you'll probably, that's a good question.
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You'll probably get different answers, uh,
0:35
from different orthopedic surgeons.
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So depending on the screw position will,
0:42
um, kind of confer different stability to the knee.
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So some surgeons will say, well, I meant
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to place it a little bit lower, uh, to confer, uh,
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more rotational stability
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of the knee while other surgeons say, I want
0:55
to put it slightly higher to confer more, uh,
0:58
translational stability.
1:00
And that is where you start to di dive in the discussion of,
1:03
uh, whether we should be doing double bundle reconstructions
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to more, um, anatomically approximate the true, um,
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an anatomy of the ACL.
1:14
So, um, I think for just one bundle, I think the 10
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or two o'clock position, uh, in that Corona plane is,
1:20
is a relatively good position.
1:23
Good. There's another question here.
1:25
I don't quite understand it.
1:26
I'm gonna read it to you the way it appears here,
1:29
and maybe you can clarify it.
1:32
It says this, if there
1:34
are any infection post ACL reconstruction,
1:39
when there is an indication to re operate.
1:41
Uh, so I think, is there any infection
1:45
in post ACL reconstructions when there is an indication
1:49
to reoperate?
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Do you un I'm not quite sure. I, I
1:52
Don't quite understand that.
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But I mean, from the infection standpoint,
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the patient should obviously be seen by their physician,
2:00
have the relevant, um, laboratory parameters, um, in hand.
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That would include things like E-S-R-C-R-P,
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which we know are non-specific, um, as well as white count.
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And if there's any concern for infection.
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Um, you know, as radiologists we're often called upon to,
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um, aspirate the joint.
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Uh, sometimes do bone biopsies,
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but more often just to aspirate the joint,
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at least get a white count
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and see if the fluid in the synovial, uh, space is,
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is a high white count.
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Um, and then of course send the, uh, the fluid,
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whatever we get for culture and sensitivity.
2:36
Okay. I have one question I wanna ask,
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just a general question.
2:40
When, what are the situations
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where they might do an ACL repair rather
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than a reconstruction?
2:48
Well, that's a really good question.
2:50
Um, sometimes they'll do it, uh, for partial tears.
2:54
Sometimes they'll do it for very high, um, uh, tears or,
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or tears near the roof of the intercon or notch.
3:02
That's, uh, you know, that's few and far between.
3:05
Um, they are doing that at some high-end, um, places, uh,
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like Steadman, uh, Hopkins and Philippon.
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Uh, our orthopedic surgeons are not doing them as much.
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Um, most of them, the vast majority are undergoing
3:18
reconstruction, so they're clearing out the native ligament.
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But for partial tears, you might see that done.
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I've even seen, um, patients undergo, uh,
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isolated single bundle reconstructions for partial tears
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as well, rather than a complete reconstruction.
3:33
Okay. Thanks for many.
3:35
Uh, I have a submitted question, uh, which is,
3:39
would it be helpful to drain the morel lala la
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lesion to speed healing?
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I don't know if many you're you're muted right now.
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Yeah, she says one second.
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She did turn the light on. Sorry. That's
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Good. I've lost the screen.
3:56
Um, the general consensus is
4:00
that these tend to recur after drainage.
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It's not just a single episode of bleeding.
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The injury that takes place also disrupts the lymphatics.
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And so there tends to be lymphatic fluid
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that just keeps collecting, uh, in the, so the
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accepted treatment for these is to do prolonged compression,
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and if that doesn't work, uh, to then try to attempt
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to sclero de the lesion, um, by putting in some kind
4:27
of irritant to try to get it to seal.
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So, uh, we don't even try hydrating these
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because of the high rate of them, uh,
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they fill right back up.
4:38
Okay. I have a, a question I wanna ask you.
4:40
I've seen several cases of Lyme disease in the knee
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and, uh, some of them have had prominent lymphadenopathy
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as well as myositis.
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Is that one of the diseases we should think of
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with lymphadenopathy? Yeah,
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I didn't mention it.
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I think the cases of Lyme disease you've seen must have been
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sent to you from outside
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because we don't see Lyme disease here in California.
5:03
Certainly the literature emphasizes the importance
5:06
of particularly pop platia edema
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as being very distinctive in Lyme and lymph nodes.
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I I just generally include infection in my differential if I
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have lymphadenopathy.
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Okay. And I'm sure what,
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what exactly is causing the infection varies
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depending upon where you're living.
5:22
I know in India it's always, they say tuberculosis.
5:24
I say cocc city or my Cause you say potato. I,
5:29
Uh, I have just a comment on one of the things
5:32
and, uh, any input from you is fine.
5:35
The, the, you were talking about ganglia intimate
5:38
with a gastrocnemius
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and, uh, I agree with you that in the cases I've seen,
5:44
it's been the medial head that more often I see
5:47
that alteration.
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So many years ago, one of our scholars began a study, uh,
5:52
never published of what the femoral attachments were
5:55
of the medial and lateral heads.
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And on the medial head there were often two attachments
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of the tendon with connective tissue in between,
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whereas the tendon
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of the lateral head was a single connection.
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So we thought maybe because of that difference in anatomy,
6:12
it might explain why the ganglion cysts are more
6:15
common on the medial side.
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I don't know if you have any comments about that or, or,
6:20
You know, I've talked to you about this over the years.
6:22
I, I personally think there are two heads
6:24
of the lateral gastroc anemias
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because I often see an attachment at the lateral condyle
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and then a separate bundle of fibers that inserts just
6:33
above the FCL that's often confused with thickening
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of the upper FCL.
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So I'm not convinced, I, I'm not convinced
6:41
that the anatomy is different
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and I don't know how many specimens they had,
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and I never saw the specimens from that
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Part. There wasn't a lot of
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specimens,
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and that's one of the reasons we didn't publish it. So I'll
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Show you, I'll show you some of the cases sometime maybe
6:52
next week of, uh, what looked to me like a,
6:56
a separate bundle coming over the FCL from
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the lateral gastroc.
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So I don't think, I don't use that as a distinction.
7:05
Okay. Uh, another, I just have a couple more things
7:08
I wanted to ask you about.
7:09
Uh, we talk about the ligamentum mucosa
7:12
and the course of the ligamentum mucosa as it courses
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through hoffa's fat pad curves up
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and attaches to the patella, often near the junction
7:22
with cartilage in the inferior pole of the patella.
7:24
And I've been impressed with all altered patella tracking.
7:28
When we do have fat pad edema,
7:30
we often have edema about the ligamentum mucosa.
7:34
Is that something you have seen at all?
7:37
Uh, you know, I've noticed it,
7:38
but quite honestly, I see so much edema around it.
7:42
Whenever there's a joint diffusion,
7:43
like I think fluid from the joint can track
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along that structure.
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So I don't think it's a very specific finding
7:50
for tracking dysfunction.
7:52
I I, you know, our practice has a lot of injuries
7:55
with big effusions and you'll often see that happening.
7:58
So it is a non-specific finding.
8:00
Yeah, we were gonna start a study on that and,
8:03
but we never got around to really, uh,
8:05
doing anything with it.
8:07
Uh, I wanna ask you another thing
8:09
because, you know, we deal with Achilles tendinosis.
8:12
It's often a bilateral process.
8:14
How often do you think quadriceps tendinosis,
8:18
if you image the other side,
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would the quadriceps tendon more likely be
8:22
normal or abnormal? What do you think?
8:24
I think tendinosis is a bilateral process. I, I really do.
8:28
But I think the activities that people do, first of all,
8:31
their protoplasm, you know, you're born with a tendency
8:34
to turn into, as you say, mucus or calcification, right?
8:38
Like, I'm definitely leaning towards the mucus side on my
8:41
life or maybe fat.
8:43
I'll keep that in mind.
8:45
Yeah, I think fatty degeneration go with that.
8:47
But, um, my,
8:48
I suspect it's bilateral when I see things they're
8:51
so often bilateral.
8:53
Yeah. And I just mentioned that
8:55
because there's a famous NFL quarterback who, uh,
8:58
had a rupture of his achilles tendon.
9:01
I won't mention his name, he's back playing again,
9:04
but I wonder what his other ankle looks like
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and what his other Achilles tendon. No,
9:08
I'm sure there are certain sports like kicking at are done
9:12
on a unilateral basis,
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but most of the tendinosis I see is it's not related
9:17
to one physical activity.
9:19
It's just sort of the way they're made aging.
9:23
Okay. We're gonna stop there.
9:25
We, we've gone to a little bit later. It's been a long day.
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I would remind everyone that we're back to the four hour,
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uh, time tomorrow.
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We're gonna head down the lower extremity
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and tomorrow we'll be spending time on the ankle and foot.
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I want to thank thank Brady and Minnie, a spectacular day.
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Terrific lectures. Uh, I learned a lot.
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I even took some notes here.
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Uh, you know, so I really appreciate, uh, the quality
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of the lectures and how complete they were.